Zobrazeno 1 - 10
of 17
pro vyhledávání: '"Becky M. Sebastian"'
Autor:
Sanjoy Rowchowdhury, Sangeeta R. Kashyap, John P. Kirwan, Laura E. Nagy, Megan R. McMullen, Becky M. Sebastian, Ariel E. Feldstein, Philip R. Schauer, Antoinette D. Hillian
Publikováno v:
Journal of Biological Chemistry. 288:22565-22575
Complement activation is implicated in the development of obesity and insulin resistance, and loss of signaling by the anaphylatoxin C3a prevents obesity-induced insulin resistance in mice. Here we have identified C1q in the classical pathway as requ
Autor:
Hui Tang, Laura E. Nagy, Antoinette D. Hillian, Xiaocong Chen, Becky M. Sebastian, Donald W. Jacobsen, Armend Axhemi
Publikováno v:
Alcoholism: Clinical and Experimental Research. 36:214-222
Background: Adipose tissue is an important target for ethanol action. One important effect of ethanol is to reduce the secretion of adiponectin from adipocytes; this decrease is associated with lowered circulating adiponectin in rodent models of chro
Autor:
Hui Tang, Donald W. Jacobsen, Becky M. Sebastian, Laura E. Nagy, Armend Axhemi, Megan Mr McMullen, Xiaocong Chen
Publikováno v:
Hepatology. 49:1554-1562
Adiponectin, an adipokine primarily secreted by adipose tissue, comprises about 0.01% of total serum protein (1). It circulates in three oligomeric forms: high molecular weight (HMW), middle molecular weight (MMW) and low molecular weight (LMW). Adip
Autor:
Brian T. Pratt, James C. Alexander, Xiaocong Chen, Laura E. Nagy, Ilya Bederman, Li Kang, Stephen F. Previs, Becky M. Sebastian
Publikováno v:
Journal of Biological Chemistry. 282:28465-28473
Chronic ethanol consumption disrupts whole-body lipid metabolism. Here we tested the hypothesis that regulation of triglyceride homeostasis in adipose tissue is vulnerable to long-term ethanol exposure. After chronic ethanol feeding, total body fat c
Autor:
Stephen F. Previs, Becky M. Sebastian, Laura E. Nagy, Li Kang, Brian T. Pratt, Michele T. Pritchard
Publikováno v:
Alcoholism: Clinical and Experimental Research. 31:1581-1588
Background: Chronic ethanol consumption disrupts glucose homeostasis and is associated with the development of insulin resistance. While adipose tissue and skeletal muscle are the two major organs utilizing glucose in response to insulin, the relativ
Autor:
Hui, Tang, Becky M, Sebastian, Armend, Axhemi, Xiaocong, Chen, Antoinette D, Hillian, Donald W, Jacobsen, Laura E, Nagy
Publikováno v:
Alcoholism, clinical and experimental research. 36(2)
Adipose tissue is an important target for ethanol action. One important effect of ethanol is to reduce the secretion of adiponectin from adipocytes; this decrease is associated with lowered circulating adiponectin in rodent models of chronic ethanol
Autor:
Sanjoy Roychowdhury, Hui Tang, Gregory L. Stahl, Becky M. Sebastian, Antoinette D. Hillian, Laura E. Nagy, Kazue Takahashi, Ariel E. Feldstein
Chronic, heavy alcohol exposure results in inflammation in adipose tissue, insulin resistance, and liver injury. Here we have identified a CYP2E1/Bid/C1q-dependent pathway that is activated in response to chronic ethanol and is required for the devel
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c11ca068cbd6f6a0ad913734c3a333db
https://europepmc.org/articles/PMC3195631/
https://europepmc.org/articles/PMC3195631/
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 447
Chronic ethanol consumption dysregulates glucose and lipid homeostasis, is associated with insulin resistance, and alters serum levels of adipokines including adiponectin and tumor necrosis factor-alpha. However, the mechanisms involved in these chro
Publikováno v:
Alcohol ISBN: 9781588299062
Chronic ethanol consumption dysregulates glucose and lipid homeostasis, is associated with insulin resistance, and alters serum levels of adipokines including adiponectin and tumor necrosis factor-alpha. However, the mechanisms involved in these chro
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d23b089ee5b8d387ed7419559a17331c
https://doi.org/10.1007/978-1-59745-242-7_23
https://doi.org/10.1007/978-1-59745-242-7_23
Autor:
Li, Kang, Xiaocong, Chen, Becky M, Sebastian, Brian T, Pratt, Ilya R, Bederman, James C, Alexander, Stephen F, Previs, Laura E, Nagy
Publikováno v:
The Journal of biological chemistry. 282(39)
Chronic ethanol consumption disrupts whole-body lipid metabolism. Here we tested the hypothesis that regulation of triglyceride homeostasis in adipose tissue is vulnerable to long-term ethanol exposure. After chronic ethanol feeding, total body fat c