Zobrazeno 1 - 10
of 25
pro vyhledávání: '"Becky A. de la Houssaye"'
Autor:
Brittany V Martin-Murphy, Qiang You, Hong Wang, Becky A De La Houssaye, Timothy P Reilly, Jacob E Friedman, Cynthia Ju
Publikováno v:
PLoS ONE, Vol 9, Iss 1, p e80949 (2014)
Current estimates suggest that over one-third of the adult population has metabolic syndrome and three-fourths of the obese population has non-alcoholic fatty liver disease (NAFLD). Inflammation in metabolic tissues has emerged as a universal feature
Externí odkaz:
https://doaj.org/article/23e8d529b1eb47849fc628a09d998622
Autor:
Margaret J R Heerwagen, Michael S Stewart, Becky A de la Houssaye, Rachel C Janssen, Jacob E Friedman
Publikováno v:
PLoS ONE, Vol 8, Iss 6, p e67791 (2013)
Maternal and pediatric obesity has risen dramatically over recent years, and is a known predictor of adverse long-term metabolic outcomes in offspring. However, which particular aspects of obese pregnancy promote such outcomes is less clear. While ma
Externí odkaz:
https://doaj.org/article/039655526bf74d1084b4c45490a6bf5a
Autor:
Charles E. Robertson, Angelo D'Alessandro, Moshe Levi, Jacob E. Friedman, Alba Alfonso-Garcia, Xiaoxin X. Wang, Karen R. Jonscher, Aimee L. Anderson, Evgenia Dobrinskikh, Daniel N. Frank, Rachel C. Janssen, Taylor K. Soderborg, David J. Orlicky, Becky A. de la Houssaye, Alexander Fast, Linda K. Johnson, Eric O. Potma, Karim C. El Kasmi, Julie A. Reisz
Publikováno v:
Hepatology Communications
Friedman, JE; Dobrinskikh, E; Alfonso-Garcia, A; Fast, A; Janssen, RC; Soderborg, TK; et al.(2018). Pyrroloquinoline quinone prevents developmental programming of microbial dysbiosis and macrophage polarization to attenuate liver fibrosis in offspring of obese mice.. Hepatology communications, 2(3), 313-328. doi: 10.1002/hep4.1139. UC Irvine: Retrieved from: http://www.escholarship.org/uc/item/7pv1r620
Friedman, JE; Dobrinskikh, E; Alfonso-Garcia, A; Fast, A; Janssen, RC; Soderborg, TK; et al.(2018). Pyrroloquinoline quinone prevents developmental programming of microbial dysbiosis and macrophage polarization to attenuate liver fibrosis in offspring of obese mice.. Hepatology communications, 2(3), 313-328. doi: 10.1002/hep4.1139. UC Irvine: Retrieved from: http://www.escholarship.org/uc/item/7pv1r620
Increasingly, evidence suggests that exposure to maternal obesity creates an inflammatory environment in utero, exerting long-lasting postnatal signatures on the juvenile innate immune system and microbiome that may predispose offspring to developmen
Autor:
Allison L.B. Shapiro, Dana Dabelea, Becky A. de la Houssaye, Zachary W. Patinkin, Lauren A. Vanderlinden, Jacob E. Friedman, Rachel C. Janssen, Peter R. Baker
Publikováno v:
Scientific Reports, Vol 7, Iss 1, Pp 1-10 (2017)
Scientific Reports
Scientific Reports
The intrauterine period is a critical time wherein developmental exposure can influence risk for chronic disease including childhood obesity. Using umbilical cord-derived mesenchymal stem cells (uMSC) from offspring born to normal-weight and obese mo
Autor:
Ardythe L. Morrow, Nancy F. Krebs, Bridget E. Young, Sheela R. Geraghty, Barbara S. Davidson, Laura Pyle, Zachary W. Patinkin, Becky A. de la Houssaye
Publikováno v:
Maternal and Child Health Journal. 21:1367-1376
Objective Obesity in adults is associated with inflammation and oxidative stress. Whether or not this phenotype is reflected in human milk (HM) composition, or may impact infant growth remains unknown. We investigated whether HM from overweight/obese
Autor:
Linda A. Barbour, Becky A. de la Houssaye, Michael C. Rudolph, Angela C Tomczik, Charles E. Robertson, Stephanie A. Santorico, Dominick J. Lemas, Diana Ir, Teri L. Hernandez, Nancy F. Krebs, Tiffany L. Weir, Jacob E. Friedman, Peter R. Baker, Daniel N. Frank, Zachary W. Patinkin, Taylor K. Soderborg, Bridget E. Young
Publikováno v:
The American Journal of Clinical Nutrition. 103:1291-1300
BACKGROUND Increased maternal body mass index (BMI) is a robust risk factor for later pediatric obesity. Accumulating evidence suggests that human milk (HM) may attenuate the transfer of obesity from mother to offspring, potentially through its effec
Autor:
Becky A. de la Houssaye, Jacob E. Friedman, Rachael E. Van Pelt, William T. Donahoo, Linda A. Barbour, Catherine Chartier-Logan, Melanie S. Reece, Rachel C. Janssen, Regina M. Reynolds, Linda J. Daniels, Molly A. Anderson, Teri L. Hernandez, Margaret J. R. Heerwagen
Publikováno v:
Diabetes Care
OBJECTIVE Diet therapy in gestational diabetes mellitus (GDM) has focused on carbohydrate restriction but is poorly substantiated. In this pilot randomized clinical trial, we challenged the conventional low-carbohydrate/higher-fat (LC/CONV) diet, hyp
Autor:
Paul S. MacLean, David M. Presby, Julie A. Houck, Ginger C. Johnson, Taylor K. Soderborg, Patricia G. Webb, Becky A. de la Houssaye, Matthew R. Jackman, Michael C. Rudolph, Jacob E. Friedman, Ivana V. Yang
Publikováno v:
Diabetes. 67(4)
Adipose tissue expansion progresses rapidly during postnatal life, influenced by both prenatal maternal factors and postnatal developmental cues. The ratio of omega-6 (n-6) relative to n-3 polyunsaturated fatty acids (PUFAs) is believed to regulate p
Autor:
Michael Woontner, Dana Dabelea, Jacob E. Friedman, Peter R. Baker, Zachary W. Patinkin, Becky A. de la Houssaye, Allison L.B. Shapiro, Lauren A. Vanderlinden, Kristen E. Boyle
Publikováno v:
JCI insight. 2(21)
Maternal obesity is a global health problem that increases offspring obesity risk. The metabolic pathways underlying early developmental programming in human infants at risk for obesity remain poorly understood, largely due to barriers in fetal/infan
Autor:
Karen R. Jonscher, Yuhuan Luo, Garrett Florey, Xiaoxin X. Wang, Moshe Levi, Jacob E. Friedman, Margaret J. R. Heerwagen, Becky A. de la Houssaye, Ellen Wiitala, Brian C. DeFelice, Alba Alfonso-Garcia, Raleigh Jonscher, Michael S. Stewart, Eric O. Potma, Oliver Fiehn, Rachel C. Janssen
Publikováno v:
FASEB journal : official publication of the Federation of American Societies for Experimental Biology. 31(4)
Nonalcoholic fatty liver disease (NAFLD) is widespread in adults and children. Early exposure to maternal obesity or Western-style diet (WD) increases steatosis and oxidative stress in fetal liver and is associated with lifetime disease risk in the o