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of 5
pro vyhledávání: '"Beatriz Russell"'
Autor:
Saumitri Bhattacharyya, Juraj Kavecansky, Samir Acharya, Beatriz Russell, April Sandy, Erin M. Perchiniak, Joanna Groden, Patrick M. Grierson, Jeremy Keirsey
Publikováno v:
Cancer Research. 71:561-571
Cells deficient in the recQ-like helicase BLM are characterized by chromosome changes that suggest the disruption of normal mechanisms needed to resolve recombination intermediates and to maintain chromosome stability. Human BLM and topoisomerase II
Autor:
Dawn Marie Goudelock, Yolanda Sanchez, Kecheng Jiang, Beatriz Russell, Melissa Maxfield, Elizabeth Pereira
Publikováno v:
Journal of Biological Chemistry. 278:25207-25217
Checkpoints are biochemical pathways that provide the cell with mechanisms to detect DNA damage and respond by arresting the cell cycle to allow DNA repair. The conserved checkpoint kinase Chk1 regulates mitotic progression in response to DNA damage
Autor:
John J. Turchi, Kate Lillard-Wetherell, Juraj Kavecansky, Jeremy Keirsey, Kambiz Tahmaseb, Joanna Groden, Saumitri Bhattacharyya, Beatriz Russell
The BLM helicase associates with the telomere structural proteins TRF1 and TRF2 in immortalized cells using the alternative lengthening of telomere (ALT) pathways. This work focuses on identifying protein partners of BLM in cells using ALT. Mass spec
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9eba85cad1a31c5c0b7927df7df28de3
https://europepmc.org/articles/PMC2685679/
https://europepmc.org/articles/PMC2685679/
Autor:
Joanna Groden, Beatriz Russell
Publikováno v:
Genetics of Colorectal Cancer ISBN: 9780387095677
Bloom syndrome (BS) was first described by Dr. David Bloom in 1954 after he observed a small number of patients of Ashkenazi Jewish origin with erythematous lesions of the face and small stature (Bloom 1954). Approximately 10 years later, the chromos
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9ee058a6559ebde1342a26887d07aeb5
https://doi.org/10.1007/978-0-387-09568-4_13
https://doi.org/10.1007/978-0-387-09568-4_13
Publikováno v:
The Journal of biological chemistry. 278(32)
Checkpoints are biochemical pathways that provide cells a mechanism to detect DNA damage and respond by arresting the cell cycle to allow DNA repair. The conserved checkpoint kinase, Chk1, regulates mitotic progression in response to DNA damage by bl