Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Beata Tryniszewska"'
Autor:
Júlia Kurkó, András Vida, Tímea Ocskó, Beata Tryniszewska, Tibor A Rauch, Tibor T Glant, Zoltán Szekanecz, Katalin Mikecz
Publikováno v:
PLoS ONE, Vol 9, Iss 11, p e111815 (2014)
Myeloid-derived suppressor cells (MDSCs) are innate immune cells capable of suppressing T-cell responses. We previously reported the presence of MDSCs with a granulocytic phenotype in the synovial fluid (SF) of mice with proteoglycan (PG)-induced art
Externí odkaz:
https://doaj.org/article/220eedaecf3a4d2cb43cb713ab9d9e19
Autor:
Timea Besenyei, Andras Kadar, Beata Tryniszewska, Julia Kurko, Tibor A. Rauch, Tibor T. Glant, Katalin Mikecz, Zoltan Szekanecz
Publikováno v:
Clinical and Developmental Immunology, Vol 2012 (2012)
Rheumatoid arthritis (RA) is a polygenic autoimmune disease primarily affecting the synovial joints. Numerous animal models show similarities to RA in humans; some of them not only mimic the clinical phenotypes but also demonstrate the involvement of
Externí odkaz:
https://doaj.org/article/ea70ac6441d9482888cdaff6d7a961fd
Autor:
Beata Tryniszewska, James O. Meyer, Lixia Yue, Andreas D. Kistler, Jochen Reiser, Hao Yu, Dolly Mehta, Stuart E. Dryer, Mohd Hafeez Faridi
Publikováno v:
Journal of the American Society of Nephrology. 27:3308-3319
Gain-of-function mutations of classic transient receptor potential channel 6 (TRPC6) were identified in familial FSGS, and increased expression of wild-type TRPC6 in glomeruli is observed in several human acquired proteinuric diseases. Synaptopodin,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::75f42fea06ac943831f47a6bbad10507
https://doi.org/10.1681/asn.2015080896
https://doi.org/10.1681/asn.2015080896
Autor:
Tibor A. Rauch, Timea Besenyei, Robert S. Katz, János Gál, Beata Tryniszewska, Katalin Mikecz, András Kádár, Györgyike Soós, Júlia Kurkó, Zoltán Szekanecz, Tibor T. Glant, Joel A. Block, Gyula Hoffmann
Publikováno v:
Arthritis & Rheumatism. 65:1725-1735
Objective To identify epigenetic factors that are implicated in the pathogenesis of rheumatoid arthritis (RA), and to explore the therapeutic potential of the targeted inhibition of these factors. Methods Polymerase chain reaction (PCR) arrays were u
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::473c3749c2613e7909a460d5818e89ae
https://doi.org/10.1002/art.37986
https://doi.org/10.1002/art.37986
Autor:
Robin M. Voigt, Martha Hotz Vitaterna, Beata Tryniszewska, Ranjan Kc, Hee Jeong Im, Qing-Jun Meng, Fred W. Turek, Ali Keshavarzian, Xin Li, Christopher B. Forsyth, Keith C. Summa
Publikováno v:
Scientific Reports
A variety of environmental factors contribute to progressive development of osteoarthritis (OA). Environmental factors that upset circadian rhythms have been linked to various diseases. Our recent work establishes chronic environmental circadian disr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ca6def6ab84bffe31ade063b8d41232f
http://europepmc.org/articles/PMC4653622
http://europepmc.org/articles/PMC4653622
Autor:
Beata Tryniszewska, Tibor A. Rauch, András Vida, Tímea Ocskó, Katalin Mikecz, Tibor T. Glant, Júlia Kurkó, Zoltán Szekanecz
Publikováno v:
PLoS ONE, Vol 9, Iss 11, p e111815 (2014)
PLoS ONE
PLoS ONE
Background Myeloid-derived suppressor cells (MDSCs) are innate immune cells capable of suppressing T-cell responses. We previously reported the presence of MDSCs with a granulocytic phenotype in the synovial fluid (SF) of mice with proteoglycan (PG)-
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b77600bb258f87e208ffbceaa85e7685
http://europepmc.org/articles/PMC4219784?pdf=render
http://europepmc.org/articles/PMC4219784?pdf=render
Autor:
Tibor T. Glant, Beata Tryniszewska, András Kádár, Timea Besenyei, Katalin Mikecz, Júlia Kurkó, Tibor A. Rauch, Zoltán Szekanecz
Publikováno v:
Clinical and Developmental Immunology, Vol 2012 (2012)
Clinical and Developmental Immunology
Clinical and Developmental Immunology
Rheumatoid arthritis (RA) is a polygenic autoimmune disease primarily affecting the synovial joints. Numerous animal models show similarities to RA in humans; some of them not only mimic the clinical phenotypes but also demonstrate the involvement of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e279cf8b7e06729cc56f2458ccd4cdbe
https://doaj.org/article/ea70ac6441d9482888cdaff6d7a961fd
https://doaj.org/article/ea70ac6441d9482888cdaff6d7a961fd
Autor:
Beata Tryniszewska, Zsuzsa Gyorfy, Marianna Radacs, Gabor Hutas, Vincent C. Hascall, Katalin Mikecz, Gyorgy Nagyeri, Tibor T. Glant, Sheida Ghassemi-Nejad, Katalin Olasz
TSG-6 (TNF-α-stimulated gene/protein 6), a hyaluronan (HA)-binding protein, has been implicated in the negative regulation of inflammatory tissue destruction. However, little is known about the tissue/cell-specific expression of TSG-6 in inflammator
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b04f391fe6fbcb62aaa51cb29174f48f
Autor:
Beata Tryniszewska, Michael D. Tharp, Melinda Simon, Tibor T. Glant, Andrew B. Nesterovitch, Zsuzsa Gyorfy, Tibor A. Rauch, Sewon Kang, Nada Elbuluk, Ellen C. Moore, Gary J. Fisher, Katalin Mikecz, Mark D. Hoffman
Publikováno v:
The American journal of pathology. 178(4)
We have found a B2 repeat insertion in the gene encoding protein tyrosine phosphatase nonreceptor type 6 (PTPN6) in a mouse that developed a skin disorder with clinical and histopathological features resembling those seen in human neutrophilic dermat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3b1361f0ac2e876d935b388036e97b80
https://pubmed.ncbi.nlm.nih.gov/21406173
https://pubmed.ncbi.nlm.nih.gov/21406173
Publikováno v:
The Journal of Immunology. 186:167.21-167.21
Joint damage in rheumatoid arthritis (RA) is largely mediated by synovial fluid (SF) neutrophils. In proteoglycan (PG)-induced arthritis (PGIA), an autoimmune mouse model of RA, SF cells show high expression of the myeloid lineage markers CD11b and G
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::dce30028fc18105de3bb9ff421817257
https://doi.org/10.4049/jimmunol.186.supp.167.21
https://doi.org/10.4049/jimmunol.186.supp.167.21