Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Batbold Boldbaatar"'
Autor:
Surbhi Jain, Sitong Chen, Kung-Chao Chang, Yih-Jyh Lin, Chi-Tan Hu, Batbold Boldbaatar, James P Hamilton, Selena Y Lin, Ting-Tsung Chang, Shun-Hua Chen, Wei Song, Stephen J Meltzer, Timothy M Block, Ying-Hsiu Su
Publikováno v:
PLoS ONE, Vol 7, Iss 4, p e35789 (2012)
Hypermethylation of the glutathione S-transferase π 1 (GSTP1) gene promoter region has been reported to be a potential biomarker to distinguish hepatocellular carcinoma (HCC) from other liver diseases. However, reports regarding how specific a marke
Externí odkaz:
https://doaj.org/article/8157d69ee3964b4d8dbef3aed7b088c5
Publikováno v:
Amyloid : the international journal of experimental and clinical investigation : the official journal of the International Society of Amyloidosis. 26(sup1)
Autor:
George J. Murphy, John L. Berk, Andrew A. Wilson, Vaishali Sanchorawala, Lawreen H. Connors, Richard M. Giadone, Jessica D. Rosarda, Marianne F. James, Prithvi Reddy Akepati, Arianne C Thomas, R. Luke Wiseman, Batbold Boldbaatar
Hereditary transthyretin amyloidosis (ATTR amyloidosis) is an autosomal dominant protein-folding disorder caused by over 100 distinct mutations in the transthyretin (TTR) gene. In ATTR amyloidosis, protein secreted from the liver aggregates and forms
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::06a3440f00d13d6de8c2f20a15a3a9e4
https://europepmc.org/articles/PMC6319917/
https://europepmc.org/articles/PMC6319917/
Publikováno v:
Amyloid. 26:83-84
Wild-type transthyretin amyloidosis (ATTRwt) is a sporadic protein misfolding disease with no specific biomarkers or treatment [1]. The disease most often features cardiomyopathy and heart failure ...
Autor:
Lijia Xie, Ying Hsiu Su, Chi Tan Hu, Timothy M. Block, Stephen J. Meltzer, James P. Hamilton, Selena Y. Lin, Shun Hua Chen, Surbhi Jain, Batbold Boldbaatar, Wei Song
Publikováno v:
Hepatology Research. 45:1110-1123
Aim Aberrant methylation of the promoter, P2, and the first exon, E1, regions of the tumor suppressor gene RASSF1A, have been associated with hepatocellular carcinoma (HCC), albeit with poor specificity. This study analyzed the methylation profiles o
Autor:
Shanpeng Li, Marc Iglarz, Richard M. Wilson, Flora Sam, Batbold Boldbaatar, María Valero-Muñoz
Publikováno v:
Circulation. Heart failure. 9(11)
Background— Despite the increasing prevalence of heart failure with preserved ejection fraction (HFpEF) in humans, there remains no evidence-based therapies for HFpEF. Endothelin-1 (ET-1) antagonists are a possibility because elevated ET-1 levels a
Autor:
Ying Hsiu Su, Yih Jyh Lin, James P. Hamilton, Shun Hua Chen, Surbhi Jain, Wei Song, Ting-Tsung Chang, Kung Chao Chang, Timothy M. Block, Chi Tan Hu, Batbold Boldbaatar, Stephen J. Meltzer, Selena Y. Lin, Sitong Chen
Publikováno v:
PLoS ONE
PLoS ONE, Vol 7, Iss 4, p e35789 (2012)
PLoS ONE, Vol 7, Iss 4, p e35789 (2012)
Hypermethylation of the glutathione S-transferase π 1 (GSTP1) gene promoter region has been reported to be a potential biomarker to distinguish hepatocellular carcinoma (HCC) from other liver diseases. However, reports regarding how specific a marke
Autor:
Chi-Tan Hu, Timothy M. Block, Ying-Hsiu Su, Sitong Chen, Ran Yan, Selena Y. Lin, Batbold Boldbaatar, Wei Song, Haitao Guo, Surbhi Jain
Publikováno v:
Cancer Research. 74:1373-1373
Hepatitis B virus (HBV) is a hepatotropic virus causing hepatitis, cirrhosis and hepatocellular carcinoma. In patients, HBV DNA circulates in blood as virion DNA and exists in hepatocytes both as nuclear form (episomal cccDNA and integrated DNA) and
Publikováno v:
Cancer Research. 74:876-876
HBV related liver disease ranges from hepatitis, cirrhosis, and hepatocellular carcinoma (HCC), which has a 5-year survival rate of 14% because it is difficult to diagnose at its curative stages. Chronic hepatitis B virus (HBV) infections is the majo
Autor:
Valero-Munoz, Maria, Shanpeng Li, Wilson, Richard M., Batbold Boldbaatar, Iglarz, Marc, Sam, Flora
Publikováno v:
Circulation: Heart Failure; Nov2016, Vol. 9 Issue 11, p1-9, 9p, 1 Chart, 5 Graphs