Accumulation of Prion Protein in the Brain That Is Not Associated with Transmissible Disease

Autor: Piccardo, Pedro, Manson, Jean C., King, Declan, Ghetti, Bernardino, Barron, Rona M.

Publikováno v: Proceedings of the National Academy of Sciences of the United States of America, 2007 Mar . 104(11), 4712-4717.

Externí odkaz: https://www.jstor.org/stable/25426898

A gene-targeted mouse model of P102L Gerstmann-Sträussler-Scheinker syndrome

Autor: Barron, Rona M, Manson, Jean C

Publikováno v: In Clinics in Laboratory Medicine 2003 23(1):161-173

Increased susceptibility of transgenic mice expressing human PrP to experimental sheep BSE is not due to increased agent titre in sheep brain tissue

Autor: Plinston, Christopher, Hart, Patricia, Hunter, Nora, Manson, Jean C, Barron, Rona M

Publikováno v: Plinston, C, Hart, P, Hunter, N, Manson, J C & Barron, R M 2014, ' Increased susceptibility of transgenic mice expressing human PrP to experimental sheep BSE is not due to increased agent titre in sheep brain tissue ', Journal of General Virology, vol. 95, no. 8, pp. 1855-1859 . https://doi.org/10.1099/vir.0.065730-0

Bovine Spongiform Encephalopathy (BSE) in cattle and variant Creutzfeldt Jacob disease (vCJD) in humans have previously been shown to be caused by the same strain of transmissible spongiform encephalopathy (TSE) agent. It is hypothesised that the age

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od______3094::fe737993c7f896d12c231c6762c5a1ac
https://www.pure.ed.ac.uk/ws/files/16494032/Plinston_JGV_2014.pdf

Mechanism of PrP-amyloid formation in mice without transmissible spongiform encephalopathy

Autor: Jeffrey, Martin, McGovern, Gillian, Chambers, Emily V., King, Declan, González, Lorenzo, Manson, Jean C., Ghetti, Bernardino, Piccardo, Pedro, Barron, Rona M.

Publikováno v: Jeffrey, M, McGovern, G, Chambers, E V, King, D, Gonzalez, L, Manson, J C, Ghetti, B, Piccardo, P & Barron, R M 2012, ' Mechanism of PrP-amyloid formation in mice without transmissible spongiform encephalopathy ', Brain Pathology, vol. 22, no. 1, pp. 58-66 . https://doi.org/10.1111/j.1750-3639.2011.00508.x

Gerstmann-Straussler-Scheinker (GSS) P102L disease is a familial form of a transmissible spongiform encephalopathy (TSE) that can present with or without vacuolation of neuropil. Inefficient disease transmission into 101LL transgenic mice was previou

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::40173268c464cdd6e7b9a7ae83c4e796
https://www.pure.ed.ac.uk/ws/files/8619472/Mechanism_of_PrP_amyloid_formation_in_mice_without_transmissible_spongiform_encephalopathy.pdf

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