Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Barbara Bruser"'
Publikováno v:
Biochemistry and Cell Biology. 68:810-813
A low rate of catabolism of cortisol by lymphocytes correlates with high sensitivity of the cells to the steroid and causes them to die at a greater rate than control samples. Since lymphocytes of patients with chronic lymphocytic leukemia respond to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::264d14f95a3239e601c4a202e683eef2
https://doi.org/10.1139/o90-118
https://doi.org/10.1139/o90-118
Autor:
Ami Klein, Barbara Bruser
Publikováno v:
Life sciences. 50(12)
In the present work we undertook to ascertain whether butylated hydroxytoluene (BHT), which is used in food as an antioxidant, is capable of either inhibiting human lymphocyte stimulation or acting synergistically with cortisol and prednisolone to th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9d790f0ce4cf3bfcc7a2ea442f23c457
https://pubmed.ncbi.nlm.nih.gov/1532042
https://pubmed.ncbi.nlm.nih.gov/1532042
Autor:
Ami Klein, Paul Gordon, A. Malkin, Mark A. Wainberg, Sharon Ramcharitar, Louis Mercure, Barbara Bruser
Publikováno v:
AIDS (London, England). 4(9)
Elevations in the levels of unsaturated fatty acids (FAs) in membrane lipids lead to an increase in cell membrane fluidity and may also be involved in cell fusion and death through the loss of normal membrane function and integrity. Since the infecti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9876b948d4ab7b15b662cc4c14668026
https://pubmed.ncbi.nlm.nih.gov/1979225
https://pubmed.ncbi.nlm.nih.gov/1979225
Publikováno v:
Biochemistry and Cell Biology. 65:409-413
We have shown previously that cortisol-sensitive lymphocytes (thymocytes) have a much lower capacity than cortisol-resistant cells to catabolize cortisol. In the present study, we attempt to demonstrate that inhibition of cortisol catabolism may make
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::96f21099e2a32eb63221688396f5e51f
https://doi.org/10.1139/o87-052
https://doi.org/10.1139/o87-052
Publikováno v:
Tumor Biology. 10:149-152
We have shown previously that sera of cancer patients (CPS) possess ethanol-extractable substances which can inhibit the catabolism of cortisol by lymphocytes (CCL). In the present study an attempt was made to purify the inhibitory material by gel fi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fefa558b90a55868896286a610eefc63
https://doi.org/10.1159/000217611
https://doi.org/10.1159/000217611
Publikováno v:
Journal of Endocrinology. 112:259-264
We have observed previously that the rate of cortisol catabolism by lymphocytes (CCL) was indicative of the vulnerability of these cells to cortisol. We attempted to ascertain whether cortisol-sensitive lymphocytes (e.g. thymocytes) metabolize cortis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fb7f09903f41a6fb1815529ef3a44717
https://doi.org/10.1677/joe.0.1120259
https://doi.org/10.1677/joe.0.1120259
Publikováno v:
Metabolism. 38:278-281
We have shown previously that cortisol-sensitive lymphocytes (thymocytes) have a much lower capacity than cortisolresistant cells to catabolize cortisol and that linoleic acid inhibits the catabolism of cortisol by lymphocytes and modulates the sensi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0acae39d251adffc7904e87bd777d78d
https://doi.org/10.1016/0026-0495(89)90088-7
https://doi.org/10.1016/0026-0495(89)90088-7
Publikováno v:
Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine. 9(5)
We have shown previously that cortisol-sensitive lymphocytes (thymocytes) have a much lower capacity than cortisol-resistant cells to catabolize cortisol. We have also shown that sera of cancer patients (CPS) possess ethanol-extractable substance(s)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::41a1e7ba8904f970cf11bd2b4d93b10b
https://pubmed.ncbi.nlm.nih.gov/3194673
https://pubmed.ncbi.nlm.nih.gov/3194673