Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Barbara A. Stoos"'
Autor:
Barbara A. Stoos, Jeffrey L. Garvin
Publikováno v:
Clinical and Experimental Pharmacology and Physiology. 24:591-594
SUMMARY 1. In vivo studies have demonstrated that nitric oxide (NO) induces natriuresis. Nitric oxide-induced natriuresis occurs independently of changes in renal perfusion pressure, indicating that it is the result of a tubular effect of NO. 2. In s
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d32d1fda303fb8c6121bbaafff21c406
https://doi.org/10.1111/j.1440-1681.1997.tb02097.x
https://doi.org/10.1111/j.1440-1681.1997.tb02097.x
Publikováno v:
Journal of the American Society of Nephrology. 6:89-94
Indirect evidence suggests that nitric oxide inhibits sodium reabsorption by the collecting duct; however, direct evidence is lacking. It was hypothesized that endothelium-derived nitric oxide inhibits sodium flux in the cortical collecting duct by b
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9c89d2f372ddfeb571b96742daa86c86
https://doi.org/10.1681/asn.v6189
https://doi.org/10.1681/asn.v6189
Publikováno v:
Journal of the American Society of Nephrology. 4:1855-1860
Previously, it has been shown that the addition of bradykinin (Bk) to M-1 cortical collecting duct cells in the presence of endothelial cells decreased short-circuit current (Isc), a measure of net active transport. This effect is presumably due to t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::82d7ac2046110612b0c87fffade32b69
https://doi.org/10.1681/asn.v4111855
https://doi.org/10.1681/asn.v4111855
Publikováno v:
American Journal of Physiology-Renal Physiology. 263:F1-F6
Previous data suggest that atrial natriuretic factor (ANF) and bradykinin (BK) interact to increase Na+ and water excretion. We propose that this interaction is due to a synergistic action that inhibits Na+ absorption in the distal nephron. We examin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bcc3a87c5005a4851c9c6fec0c4abb77
https://doi.org/10.1152/ajprenal.1992.263.1.f1
https://doi.org/10.1152/ajprenal.1992.263.1.f1
Autor:
Nancy J. Hong, Dianne M. Boesch, Jeffrey L. Garvin, Barbara A. Stoos, Pablo A. Ortiz, Craig F. Plato
Publikováno v:
Hypertension (Dallas, Tex. : 1979). 41(3 Pt 2)
l -Arginine inhibits thick ascending limb (THAL) NaCl absorption by activating endothelial NO synthase (eNOS) and increasing NO production. Inhibition of renal NO production combined with a high-salt diet produces hypertension, and the THAL has been
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cec995dd0e2982598131c0061ce2c691
https://pubmed.ncbi.nlm.nih.gov/12623979
https://pubmed.ncbi.nlm.nih.gov/12623979
Publikováno v:
Hypertension (Dallas, Tex. : 1979). 34(3)
Abstract —The factor responsible for salt sensitivity of blood pressure in Dahl rats is unclear but presumably resides in the kidney. We tested the hypotheses that (1) thick ascending limbs of Dahl salt-sensitive rats (DS) absorb more NaCl than tho
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7ee044cf2b32335e354ae72246ee7156
https://pubmed.ncbi.nlm.nih.gov/10489402
https://pubmed.ncbi.nlm.nih.gov/10489402
Autor:
Jeffrey L. Garvin, Barbara A. Stoos
Publikováno v:
Biochemical pharmacology. 53(5)
Guanosine 3',5' cyclic monophosphate (cGMP) acts as a second messenger in the inner medullary collecting duct (IMCD) where it inhibits sodium transport; therefore, it is important to investigate processes that regulate intracellular cGMP levels. We h
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d6f42e1fa12b9022fc9cf9c5963993ed
https://pubmed.ncbi.nlm.nih.gov/9113081
https://pubmed.ncbi.nlm.nih.gov/9113081
Autor:
Jeffrey L. Garvin, Barbara A. Stoos
Publikováno v:
The American journal of physiology. 271(6 Pt 2)
Guanosine 3',5'-cyclic monophosphate (cGMP) is an important second messenger that regulates transport in the nephron. We propose that the transport mechanisms that remove cGMP from the cell are different in the luminal and basolateral membranes of th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e0d51639639e8aa6815fcf5598a71017
https://pubmed.ncbi.nlm.nih.gov/8997369
https://pubmed.ncbi.nlm.nih.gov/8997369
Publikováno v:
Hypertension (Dallas, Tex. : 1979). 27(3 Pt 2)
Abstract Nitric oxide has a diuretic effect in vivo. We have shown that nitric oxide inhibits antidiuretic hormone–stimulated osmotic water permeability in the collecting duct; however, the mechanism by which this occurs is unknown. We hypothesized
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::13393fed21921a123f94e28e7836214a
https://pubmed.ncbi.nlm.nih.gov/8613224
https://pubmed.ncbi.nlm.nih.gov/8613224
Publikováno v:
The Journal of clinical investigation. 89(3)
Stimulation of the release of endothelium-derived relaxing factor (EDRF) in the kidney has been shown to result in natriuresis without affecting glomerular filtration rate. This may be due to EDRF directly regulating solute transport in the cortical
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1dfeb2f8fc5e2cecce0b2f03543a6335
https://pubmed.ncbi.nlm.nih.gov/1311716
https://pubmed.ncbi.nlm.nih.gov/1311716