Zobrazeno 1 - 10
of 60
pro vyhledávání: '"Bah, Isatou"'
Autor:
Bah, Isatou, Alkhateeb, Tuqa, Kumbhare, Ajinkya, Youssef, Dima, Yao, Zhi Q., Hawkin, Gregory A., McCall, Charles E., El Gazzar, Mohamed *
Publikováno v:
In Molecular Immunology July 2020 123:97-105
Autor:
Alkhateeb, Tuqa, Kumbhare, Ajinkya, Bah, Isatou, Youssef, Dima, Yao, Zhi Q., McCall, Charles E., El Gazzar, Mohamed
Publikováno v:
In Molecular Immunology August 2019 112:72-81
Publikováno v:
In Cellular Immunology October 2018 332:32-38
Autor:
Bah, Isatou1 (AUTHOR), Alkhateeb, Tuqa1 (AUTHOR), Youssef, Dima1 (AUTHOR), Yao, Zhi Q.1 (AUTHOR), McCall, Charles E.2 (AUTHOR), El Gazzar, Mohamed1 (AUTHOR)
Publikováno v:
Journal of Innate Immunity. 2022, Vol. 14 Issue 2, p112-123. 12p.
Publikováno v:
In Cellular Immunology August 2022 378
Akademický článek
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Publikováno v:
Appalachian Student Research Forum.
Myeloid-derived suppressor cells (MDSC) expand during sepsis, suppress both innate and adaptive immunity, and promote chronic immunosuppression, which characterizes the late/chronic phase of sepsis. We previously reported that the transcription facto
Externí odkaz:
https://dc.etsu.edu/asrf/2019/schedule/187
Autor:
Alkhateeb, Tuqa, Bah, Isatou, Kumbhare, Ajinkya, Youssef, Dima, Yao, Zhi Q., McCall, Charles E., Gazzar, Mohamed E.
Publikováno v:
ETSU Faculty Works.
Myeloid-derived suppressor cells (MDSCs) expand during mouse and human sepsis, but the mechanism responsible for this is unclear. We previously reported that nuclear transport of S100A9 protein programs Gr1CD11b myeloid precursors into MDSCs in septi
Externí odkaz:
https://dc.etsu.edu/etsu-works-2/215
Publikováno v:
ETSU Faculty Works.
Myeloid-derived suppressor cells (MDSCs) prolong sepsis by promoting immunosuppression. We reported that sepsis MDSC development requires long non-coding RNA Hotairm1 interactions with S100A9. Using a mouse model that simulates the immunobiology of s
Externí odkaz:
https://dc.etsu.edu/etsu-works-2/210
Autor:
Bah, Isatou, Alkhateeb, Tuqa, Youssef, Dima, Yao, Zhi Q., McCall, Charles E., El Gazzar, Mohamed
Publikováno v:
ETSU Faculty Works.
Sepsis-induced myeloid-derived suppressor cells (MDSCs) increase mortality risk. We previously identified that long non-coding RNA Hotairm1 supports myeloid precursor shifts to Gr1+CD11b+ MDSCs during mouse sepsis. A major unanswered question is what
Externí odkaz:
https://dc.etsu.edu/etsu-works/9708
https://dc.etsu.edu/cgi/viewcontent.cgi?article=10974&context=etsu-works
https://dc.etsu.edu/cgi/viewcontent.cgi?article=10974&context=etsu-works