Zobrazeno 1 - 10
of 170
pro vyhledávání: '"BURDICK, M. D."'
Autor:
Keane, M. P., Douglas Arenberg, Lynch Iii, J. P., Whyte, R. I., Iannettoni, M. D., Burdick, M. D., Wilke, C. A., Morris, S. B., Glass, M. C., Digiovine, B., Kunkel, S. L., Strieter, R. M.
Publikováno v:
Scopus-Elsevier
Idiopathic pulmonary fibrosis (IPF) is a chronic and often fatal disorder. Fibroplasia and deposition of extracellular matrix are dependent, in part, on angiogenesis. We postulated that an imbalance exists in the expression of angiogenic (IL-8) vs an
Autor:
Tempero, R. M., Hollingsworth, M. A., Burdick, M. D., Angela Finch, Taylor, S. M., Vogen, S. M., Morgan, E. L., Sanderson, S. D.
Publikováno v:
Scopus-Elsevier
A conformationally biased decapeptide agonist of human C5a anaphylatoxin (YSFKPMPLaR) was used as a molecular adjuvant in stimulating Ab responses against peptide epitopes derived from human MUC1 glycoprotein and the human mu and kappa opioid recepto
Publikováno v:
Burdick, M D; Murray, L A; Keane, M P; Xue, Y Y; Zisman, D A; Belperio, J A; et al.(2005). CXCL11 attenuates bleomycin-induced pulmonary fibrosis via inhibition of vascular remodeling. American Journal of Respiratory and Critical Care Medicine, 171(3), 261-268. UCLA: Retrieved from: http://www.escholarship.org/uc/item/60q3f2g1
Aberrant vascular remodeling is a central hallmark for the development and progression of idiopathic pulmonary fibrosis. The mechanisms underlying the pathophysiologic alterations, however, are poorly understood. A recent phase 11 trial of interferon
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od_______325::a87233734c0b430fd63df5391c691089
http://www.escholarship.org/uc/item/60q3f2g1
http://www.escholarship.org/uc/item/60q3f2g1
Autor:
Phillips, R J, Burdick, M D, Hong, K, Lutz, M A, Murray, L A, Xue, Y Y, Belperio, J A, Keane, M P, Strieter, R M
Publikováno v:
Phillips, R J; Burdick, M D; Hong, K; Lutz, M A; Murray, L A; Xue, Y Y; et al.(2004). Circulating fibrocytes traffic to the lungs in response to CXCL12 and mediate fibrosis. Journal of Clinical Investigation, 114(3), 438-446. UCLA: Retrieved from: http://www.escholarship.org/uc/item/666511sh
Previous reports have identified a circulating pool of CD45(+) collagen I+ CXCR4(+) (CD45(+)Col I(+)CXCR4(+)) cells, termed fibrocytes, that traffic to areas of fibrosis. No studies have demonstrated that these cells actually contribute to fibrosis,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od_______325::0312218aa8d1c98cc3c834491e55856e
http://www.escholarship.org/uc/item/666511sh
http://www.escholarship.org/uc/item/666511sh
Publikováno v:
Addison, C L; Belperio, J A; Burdick, M D; & Strieter, R M. (2004). Overexpression of the duffy antigen receptor for chemokines (DARC) by NSCLC tumor cells results in increased tumor necrosis. BMC Cancer, 4, 1-15. UCLA: Retrieved from: http://www.escholarship.org/uc/item/1md362zw
Background: The Duffy antigen receptor for chemokines (DARC) is known to be a promiscuous chemokine receptor that binds a variety of CXC and CC chemokines in the absence of any detectable signal transduction events. Within the CXC group of chemokines
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od_______325::5b28f218ff4d59a259d288ed0524bfb4
http://www.escholarship.org/uc/item/1md362zw
http://www.escholarship.org/uc/item/1md362zw
Publikováno v:
Phillips, R J; Burdick, M D; Lutz, M; Belperio, J A; Keane, M P; & Strieter, R M. (2003). The stromal derived factor-1/CXCL12-CXC chemokine receptor 4 biological axis in non-small cell lung cancer metastases. American Journal of Respiratory and Critical Care Medicine, 167(12), 1676-1686. UC Davis: Retrieved from: http://www.escholarship.org/uc/item/7tc7r69k
Non-small cell lung cancer is characterized by a specific metastatic pattern. The mechanism for organ-specific metastasis is poorly understood, although evidence has suggested that the chemokine stromal derived factor-1 (CXCL12) and its cognate recep
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od_______325::26bb5eb2fca957e4f743d1749e398f86
http://www.escholarship.org/uc/item/7tc7r69k
http://www.escholarship.org/uc/item/7tc7r69k
Autor:
Koch, A. E., Kunkel, S. L., Pearce, W. H., Shah, M. R., Parikh, D., Evanoff, H. L., Haines, G. K., Burdick, M. D., Strieter, R. M.
Inflammatory leukocytes play a central role in the pathogenesis of human atherosclerotic disease, from early atherogenesis to the late stages of atherosclerosis, such as aneurysm formation. We have shown previously that human abdominal aortic aneurys
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::a89e5f5f35fc5d0e351bd73fd657f55b
https://europepmc.org/articles/PMC1886921/
https://europepmc.org/articles/PMC1886921/
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