Zobrazeno 1 - 10
of 3 317
pro vyhledávání: '"BER, Base excision repair"'
Akademický článek
Tento výsledek nelze pro nepřihlášené uživatele zobrazit.
K zobrazení výsledku je třeba se přihlásit.
K zobrazení výsledku je třeba se přihlásit.
Autor:
Chankova, Stephka G.1, Yurina, Nadezhda P.2 nadezhdayurina@hotmail.com, Todorova, Teodora I.1, Parvanova, Petya N.1
Publikováno v:
BioRisk: Biodiversity & Ecosystem Risk Assessment. 2023, Issue 20, p7-22. 16p.
Publikováno v:
In DNA Repair June 2020 90
Publikováno v:
Biofutur
Biofutur, 2006, 25 (271), pp.35-39
HAL
Biofutur, Elsevier-Cachan : Lavoisier, 2006, 25 (271), pp.35-39
Biofutur, 2006, 25 (271), pp.35-39
HAL
Biofutur, Elsevier-Cachan : Lavoisier, 2006, 25 (271), pp.35-39
International audience; La majorité des lésions de l'ADN résulte des réactions cellulaires endogènes : c'est le concept « d'ennemi de l'intérieur ». La voie de réparation par excision de bases (BER) est critique dans la lutte contre ces lés
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::96119f68efa6d3f6bd1ae1d7bf269a8b
https://hal.science/hal-00387927
https://hal.science/hal-00387927
Genotype resistance to oxidative stress, induced by various physical/chemical stimuli has been the focus of scientists for the last decades, with several aspects – ecological (the formation of the genetic elite of population), agricultural and medi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e82321da8e9d4173eca2d36b9be4604c
https://zenodo.org/record/7944012
https://zenodo.org/record/7944012
Autor:
Raimoglou, Damla a, Cimci, Murat a, Citak, Elif b, Unal, Selin b, Malikova, Narmina b, Durmaz, Eser a, Guven, Mehmet b, ⁎
Publikováno v:
In Human Gene December 2024 42
Autor:
Hao Wang, Lei Wang, Keyi Jia, Yu Liu, Sha Zhao, Wei Li, Caicun Zhou, Minlin Jiang, Bin Chen, Yayi He
Publikováno v:
Acta Pharmaceutica Sinica. B
Acta Pharmaceutica Sinica B, Vol 11, Iss 10, Pp 2983-2994 (2021)
Acta Pharmaceutica Sinica B, Vol 11, Iss 10, Pp 2983-2994 (2021)
Genomic instability remains an enabling feature of cancer and promotes malignant transformation. Alterations of DNA damage response (DDR) pathways allow genomic instability, generate neoantigens, upregulate the expression of programmed death ligand 1
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bbe73202674a833d134ec811bcbf7332
https://doi.org/10.1016/j.apsb.2021.01.003
https://doi.org/10.1016/j.apsb.2021.01.003
Autor:
Huiting Wu, Kangdong Liu, Ziming Dong, Yanyan Zhu, Jimin Zhao, Lili Zhu, Yongwei Ding, Baoyin Yuan, Jing Lu, Yangyang Zhang, Yan Qiao, Tingting Niu, Jiace Qin, Xinhuan Chen, Ge Jin
Publikováno v:
Neoplasia: An International Journal for Oncology Research, Vol 23, Iss 7, Pp 663-675 (2021)
Neoplasia (New York, N.Y.)
Neoplasia (New York, N.Y.)
Esophageal mucosa undergoes mild, moderate, severe dysplasia, and other precancerous lesions and eventually develops into carcinoma in situ, and understanding the developmental progress of esophageal precancerous lesions is beneficial to prevent them
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::52a95db3d69f8101c03db349d149368a
http://www.sciencedirect.com/science/article/pii/S1476558621000269
http://www.sciencedirect.com/science/article/pii/S1476558621000269
Autor:
Yanxia Wang, Wei Wang, Shiyu Chen, Longhui Zhong, Rong Wang, Sijie Ding, Hong Yang, Xiaoqing Luo, Yueyun Ma, Shunli Peng
Publikováno v:
Neoplasia (New York, N.Y.)
Neoplasia: An International Journal for Oncology Research, Vol 22, Iss 9, Pp 365-375 (2020)
Neoplasia: An International Journal for Oncology Research, Vol 22, Iss 9, Pp 365-375 (2020)
The addition of bevacizumab to chemotherapy has prolonged overall and progression-free survival rates for metastatic colorectal cancer (mCRC). However, KRAS-mutant (KRAS-mut) CRC, lacking an ideal targeted agent, represents an inferior-response subgr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8afea475b3889b1fb4e7acc1b320187d
https://doi.org/10.1016/j.neo.2020.06.001
https://doi.org/10.1016/j.neo.2020.06.001
Autor:
Spiros Vlahopoulos, Zsolt Radak, Attila Bacsi, Bing Zhu, Leopoldo Aguilera-Aguirre, Allan R. Brasier, Xueqing Ba, Ruoxi Wang, Wenjing Hao, Tapas K. Hazra, Lang Pan, Istvan Boldogh, Tianyang Qi
Publikováno v:
Redox Biology, Vol 18, Iss, Pp 43-53 (2018)
Redox Biology
Redox Biology
8-Oxoguanine DNA glycosylase 1 (OGG1) initiates the base excision repair pathway by removing one of the most abundant DNA lesions, 8-oxo-7,8-dihydroguanine (8-oxoG). Recent data showed that 8-oxoG not only is a pro-mutagenic genomic base lesion, but
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fa78aa331af47be4e5a92c2e745491dc
http://www.sciencedirect.com/science/article/pii/S2213231718303562
http://www.sciencedirect.com/science/article/pii/S2213231718303562