Zobrazeno 1 - 10
of 26
pro vyhledávání: '"B. H. Lorell"'
Autor:
O. H. Frazier, Edmund H. Sonnenblick, Michael R. Bristow, Leslie A. Leinwand, A. J. Moss, Jay N. Cohn, Leslie Reinlib, B. H. Lorell, Richard A. Walsh, Stephen C. Mockrin, Wilson S. Colucci, Kenneth R. Chien
Publikováno v:
Circulation. 95:766-770
The SEP identified priorities to support in future basic and clinical research and pointed out directions likely to result in advances against heart failure. The list is not intended to be all-encompassing and does not address, for example, exciting
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::084f2259fea6013c7feeb3c44bd72328
https://doi.org/10.1161/01.cir.95.4.766
https://doi.org/10.1161/01.cir.95.4.766
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 265:H834-H842
The purpose of the present study was to evaluate the feasibility of partitioning myocardial O2 consumption (VO2) into mechanical and nonmechanical components in the whole heart preparation using a negative inotrope, 2,3-butanedione monoxime (BDM), wh
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::04128fc06c9652840f5a52d429a4189d
https://doi.org/10.1152/ajpheart.1993.265.3.h834
https://doi.org/10.1152/ajpheart.1993.265.3.h834
Publikováno v:
Circulation Research. 73:550-558
The recovery of systolic and diastolic function during unstable angina may be modified by the repetition of brief episodes of ischemia and by the presence of left ventricular hypertrophy (LVH). We studied the effects of six consecutive 5-minute cycle
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1ba7fd723092f345bbe292dfa2d2057d
https://doi.org/10.1161/01.res.73.3.550
https://doi.org/10.1161/01.res.73.3.550
Publikováno v:
Circulation Research. 73:184-192
Left ventricular hypertrophy (LVH) is associated with reinduction of the fetal program of gene expression. It is unclear whether this pattern of cardiac gene expression changes with the development of left ventricular decompensation and failure. To a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e063a1111a83ef87d3d4cc7cba210e19
https://doi.org/10.1161/01.res.73.1.184
https://doi.org/10.1161/01.res.73.1.184
Publikováno v:
Circulation Research. 70:931-943
Hearts with compensatory pressure-overload hypertrophy show an increased intracardiac activation of angiotensin II that may contribute to ischemic diastolic dysfunction. We studied whether pressure-overload hypertrophy in response to aortic banding w
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::adcf8adbc237f70719491a2c867d4144
https://doi.org/10.1161/01.res.70.5.931
https://doi.org/10.1161/01.res.70.5.931
Autor:
Frederick J. Schoen, Ellen O. Weinberg, Mark J. Cunningham, B H Lorell, G A Peeters, Carl S. Apstein, William H. Barry
Publikováno v:
Circulation Research. 67:415-425
In studies that attempt to measure intracellular calcium [( Ca2+]i) in the intact heart with the calcium indicator indo 1-AM, a fundamental assumption is that the signals report changes in myocyte [Ca2+]i. We studied isolated perfused rabbit hearts l
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ab8c2040ec9ca46e03044113dcb20db3
https://doi.org/10.1161/01.res.67.2.415
https://doi.org/10.1161/01.res.67.2.415
Publikováno v:
Circulation Research. 66:406-415
Myocardial hypertrophy can result in increased sensitivity toward the development of mechanical dysfunction during hypoxia. Alterations in glycolytic metabolism may contribute to this. We studied the response to 15 minutes of hypoxia in hypertrophied
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5eda13258b6d821c40f2b5a742b1cc97
https://doi.org/10.1161/01.res.66.2.406
https://doi.org/10.1161/01.res.66.2.406
Publikováno v:
European Heart Journal. 11:54-64
The evaluation of patients with left ventricular hypertrophy and the clinical syndrome of congestive heart failure requires the ability to distinguish between the etiologies of abnormal systolic contractile function and abnormalities of diastolic rel
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5787ec01d2b6c27242f96710bb2fd5df
https://doi.org/10.1093/eurheartj/11.suppl_g.54
https://doi.org/10.1093/eurheartj/11.suppl_g.54
Autor:
A H, Kadish, A E, Buxton, H L, Kennedy, B P, Knight, J W, Mason, C D, Schuger, C M, Tracy, A W, Boone, M, Elnicki, J W, Hirshfeld, B H, Lorell, G P, Rodgers, H H, Weitz
Publikováno v:
Journal of the American College of Cardiology. 38(7)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::b38f830fef5aaa3276d5da4b1ee779e8
https://pubmed.ncbi.nlm.nih.gov/11738321
https://pubmed.ncbi.nlm.nih.gov/11738321
Publikováno v:
Pflugers Archiv : European journal of physiology. 439(3)
Previous studies have demonstrated that cardiac function changes with development of pressure overload-induced hypertrophy. The present study was undertaken to discover the basis for the changes in sarcoplasmic reticulum (SR) functions: uptake, (as r
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9a1a9d6bed41cc8a31488f352320d357
https://pubmed.ncbi.nlm.nih.gov/10650986
https://pubmed.ncbi.nlm.nih.gov/10650986