Zobrazeno 1 - 10
of 13
pro vyhledávání: '"B. A. Hover"'
Publikováno v:
Diabetes. 24:476-488
Publikováno v:
Endocrinology. 97:698-708
Iodoacetate and iodoacetamide were compared as to their capacity to block islet glycolysis and interfere with glucose inhibition of glucagon release and glucose stimulation of insulin release. Glycolysis was measured in isolated rat islet by the rate
Publikováno v:
Diabetes. 24:476-488
Metabolic interactions between glucose and amino acids were studied with isolated rat islets using glucose utilization and lactate formation as indicators. Certain amino acids (8–10 mM) are capable of greatly stimulating lactate formation from 5 mM
Publikováno v:
Diabetes. 24:369-372
The efficacy of alpha and beta-D-glucose in causing insulin release and suppressing glucagon release from the isolated perfused rat pancreas was tested. In order to allow simultaneous assessment of glucose effect on both alpha and beta-cells, the pan
Publikováno v:
Diabetes. 24:489-496
Autor:
Anthony S. Pagliara, James A. Ferrendelli, Franz M. Matschinsky, C. S. Pace, B A Hover, A D Williams
Publikováno v:
Journal of Biological Chemistry. 251:6053-6061
The glucose responsiveness of alpha- and beta-cells of normal as well as untreated and insulin-treated streptozotocin diabetic rats was tested in the extracorporeal perfusion system. Also assessed was the possible in vitro effect of added insulin on
Publikováno v:
Journal of Clinical Investigation. 55:244-255
The hyperglucagonemia that occurs in vivo in animals made diabetic with alloxan or streptozotocin is not suppressed by high glucose but is suppressed by exogenous insulin. These observations together with other studies suggested that insulin-dependen
Publikováno v:
Diabetes. 24:369-372
It has been suggested that the hyperglucagonemia observed in diabetic animals and man may be due to an impairment of glucose uptake and metabolism by the alpha-cells resulting in a decreased production of ATP. To test this hypothesis glucose, ATP, gl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::500c0beb4e33e42469717aaf7f486579
https://europepmc.org/articles/PMC333287/
https://europepmc.org/articles/PMC333287/
Publikováno v:
Endocrinology. 97(3)
Iodoacetate and iodoacetamide were compared as to their capacity to block islet glycolysis and interfere with glucose inhibition of glucagon release and glucose stimulation of insulin release. Glycolysis was measured in isolated rat islet by the rate