Zobrazeno 1 - 8
of 8
pro vyhledávání: '"B M, Clancy"'
Publikováno v:
The Journal of biological chemistry. 267(6)
Several studies have demonstrated that the intrinsic catalytic activity of cell surface glucose transporters is highly regulated in 3T3-L1 adipocytes expressing GLUT1 (erythrocyte/brain) and GLUT4 (adipocyte/skeletal muscle) glucose transporter isofo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::9b94d3893b070d3cecfc48d7004d7baf
https://pubmed.ncbi.nlm.nih.gov/1310982
https://pubmed.ncbi.nlm.nih.gov/1310982
Autor:
A, Pessino, D N, Hebert, C W, Woon, S A, Harrison, B M, Clancy, J M, Buxton, A, Carruthers, M P, Czech
Publikováno v:
The Journal of biological chemistry. 266(30)
In this study we tested the hypothesis that functional erythrocyte-type glucose transporters (GLUT1) exist as oligomeric complexes by expressing chimeric transporter proteins in Chinese hamster ovary cells harboring endogenous GLUT1 transporters. The
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::2078575355a80e22d8fd1841fe019995
https://pubmed.ncbi.nlm.nih.gov/1939082
https://pubmed.ncbi.nlm.nih.gov/1939082
Publikováno v:
The Journal of biological chemistry. 266(29)
Previous studies suggest that regulation of hexose uptake in Chinese hamster ovary fibroblasts can occur by alterations in glucose transporter intrinsic activity without changes in cell surface transporter number (Harrison, S. A., Buxton, J. M., Helg
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::979da0dc4fd13671dceefa5b04926e29
https://pubmed.ncbi.nlm.nih.gov/1918056
https://pubmed.ncbi.nlm.nih.gov/1918056
Publikováno v:
The Journal of biological chemistry. 266(16)
In this study, we tested the hypothesis that hexose transport regulation may involve proteins with relatively rapid turnover rates. 3T3-L1 adipocytes, which exhibit 10-fold increases in hexose transport rates within 30 min of the addition of 100 nM i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::146e648d013d8ea4c8af3ee99199617c
https://pubmed.ncbi.nlm.nih.gov/2037570
https://pubmed.ncbi.nlm.nih.gov/2037570
Publikováno v:
The Journal of biological chemistry. 265(33)
Complementary DNA encoding a HepG2 cell-facilitated glucose transporter (GLUT1) was subcloned into a metal-inducible, mammalian expression vector, pLEN. Mouse 3T3-L1 fibroblasts transfected with this new construct, pLENGT, exhibited zinc-inducible ex
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::0fef7ce1c6498809f6649c21c85091e8
https://pubmed.ncbi.nlm.nih.gov/2173694
https://pubmed.ncbi.nlm.nih.gov/2173694
Autor:
B M, Clancy, M P, Czech
Publikováno v:
The Journal of biological chemistry. 265(21)
Exposure of 3T3-L1 adipocytes to 100 ng/ml of cholera toxin or 1 mM dibutyryl cyclic AMP caused a marked stimulation of deoxyglucose transport. A maximal increase of 10- to 15-fold was observed after 12-24 h of exposure, while 100 nM insulin elicited
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::1bc3c442473c12cea4b68b642d7a50f2
https://pubmed.ncbi.nlm.nih.gov/2165064
https://pubmed.ncbi.nlm.nih.gov/2165064
Autor:
B M, Clancy, S, Maayani
Publikováno v:
The Journal of pharmacology and experimental therapeutics. 233(3)
The 5-HT2 receptor in isolated rabbit thoracic aorta was characterized by examining the relationships between structure and activity of nine tryptamine analogs. All assays were conducted after blockade of the alpha adrenergic receptor and inactivatio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::7f89004373d1f8830ad6ec7fd6294782
https://pubmed.ncbi.nlm.nih.gov/4009488
https://pubmed.ncbi.nlm.nih.gov/4009488
Publikováno v:
The Journal of pharmacology and experimental therapeutics. 242(1)
The kinetics of agonist and antagonist interactions with the 5-hydroxytryptamine2 receptor were studied in the isolated rabbit aorta by following the antagonist-induced decrease in the steady-state response to an agonist. A model describing the compe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::eaa6bf2696417d841d3cab6ec0e3e63e
https://pubmed.ncbi.nlm.nih.gov/3612519
https://pubmed.ncbi.nlm.nih.gov/3612519