Zobrazeno 1 - 10
of 162
pro vyhledávání: '"B E, Sobel"'
Publikováno v:
Journal of Lipid Research, Vol 26, Iss 8, Pp 955-963 (1985)
To assess the role of beta-VLDL in diet-induced atherogenesis, the in vivo metabolism and aortic accumulation of 125I-labeled beta-VLDL were investigated in cholesterol-fed rabbits and chow-fed controls. 125I-labeled HDL and 125I-labeled albumin were
Externí odkaz:
https://doaj.org/article/29bfc01a189244d1aa91b72224e1418e
Publikováno v:
Diabetes. 47:290-293
Autor:
B E Sobel
Publikováno v:
Circulation. 90:2147-2152
Publikováno v:
Diabetes. 43:104-109
Autor:
Joseph Loscalzo, Lawrence H. Cohn, Jagat Narula, Adam Timmis, Shahbudin H. Rahimtoola, Galen S. Wagner, Eduardo Marbán, Hugo Ector, David G. Iosseliani, Endre Bodnar, Allen J. Taylor, Henry Krum, Kathleen S. Stone, William C. Roberts, Chim C. Lang, W. Remme, Michael Lesch, Douglas P. Zipes, Kim A. Eagle, B E Sobel, Sanjeev Saksena, Spencer B. King, Arthur J. Moss, Cindy L. Grines, F. Van de Werf, Alan S. Pearlman, E. E. Van Der Wall, Nanette K. Wenger, Karl Swedberg, Anthony N. DeMaria, Christopher J. White, Hans Michael Piper, Jane E. Freedman, F. Alfonso, Eldon R. Smith, Robert W. Emery, Michael R. Rosen, Jeffrey S. Borer, Robert Roberts, Bilgin Timuralp, Rolf Ekroth, A. Camm, Ra-id Abdulla, Patrizio Lancellotti
Publikováno v:
Anadolu Kardiyoloji Dergisi/The Anatolian Journal of Cardiology.
Publikováno v:
Diabetes. 42:1-7
Publikováno v:
Diabetes. 41:890-895
Long-term blockade of nitric oxide synthesis in rats modulates coronary capillary network remodeling
Autor:
D, Goto, S, Fujii, A K, Zaman, I, Sakuma, M, Gao, T, Koyama, J, Mitchell, J, Woodcock-Mitchell, B E, Sobel, A, Kitabatake
Publikováno v:
Angiogenesis. 3(2)
Long-term blockade of nitric oxide synthesis with N(omega)-nitro-L-arginine methyl ester (L-NAME) induces cardiac perivascular fibrosis in rats. Its relationship to expression of angiogenic growth factors and capillary network remodeling is not under
Publikováno v:
Nutrition, metabolism, and cardiovascular diseases : NMCD. 12(6)
Concentrations in blood of plasminogen activator inhibitor type 1 (PAI-1) and circulating free (non-esterified) fatty acids (FFA) are increased in diabetes and may accelerate atherosclerosis. We have shown that FFA increase expression of PAI-1 by act
Publikováno v:
Journal of thrombosis and thrombolysis. 12(2)
Optimal anti-thrombotic therapy for acute coronary syndromes (ACS) should suppress pro-thrombotic activity at the site of plaque rupture. We sought to determine whether platelet reactivity is increased in blood in the immediate vicinity of a ruptured