Zobrazeno 1 - 10
of 22
pro vyhledávání: '"B D, Roberts"'
Publikováno v:
Catalysis of Organic Reactions
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::24f0870212ffc9fb69d0b97a0e42fe99
https://doi.org/10.1201/9781003067184-46
https://doi.org/10.1201/9781003067184-46
Publikováno v:
The Journal of Immunology. 150:625-634
Using the CD4+ model of chronic HIV-1 infection, OM-10.1, we investigated the influence of TNF-alpha regulatory networks on induced viral expression. Previously, OM-10.1 cultures were characterized to respond to exogenous TNF-alpha, as nearly 100% of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::15e8fb35d44267ceb9ae69535c62e0e5
https://doi.org/10.4049/jimmunol.150.2.625
https://doi.org/10.4049/jimmunol.150.2.625
Publikováno v:
Journal of virology. 74(1)
Antigens derived from host cells are detectable in the envelope of human immunodeficiency virus type 1 (HIV-1) and result in a distinctive viral phenotype reflecting that of the host cell. An immunomagnetic capture assay targeting discriminatory host
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::633f34dd3e55a2a598f173ebef668aec
https://pubmed.ncbi.nlm.nih.gov/10590100
https://pubmed.ncbi.nlm.nih.gov/10590100
Publikováno v:
Blood. 89(5)
Controversy exists concerning the preferential infection and replication of human immunodeficiency virus-1 (HIV-1) within naive (CD45RA+) and memory (CD45RO+) subsets of CD4+ lymphocytes. To explore the susceptibility of these subsets to HIV-1 infect
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::1abb4791d16e70d235274599c39e578b
https://pubmed.ncbi.nlm.nih.gov/9057646
https://pubmed.ncbi.nlm.nih.gov/9057646
Publikováno v:
Archives of virology. 142(6)
Because of an inherent dependence on host cell second and third messenger signaling pathways for activation of HIV-1 expression, a potential exists for a relationship between the induction of latent HIV-1 and cell-cycle-related events. To investigate
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::60a2b2752a9c3b625977a9c9d5cbf925
https://pubmed.ncbi.nlm.nih.gov/9229000
https://pubmed.ncbi.nlm.nih.gov/9229000
Autor:
B D, Roberts
Publikováno v:
Journal of insurance medicine (New York, N.Y.). 26(1)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::ac15f9cbca727c5471e28c3274524b9e
https://pubmed.ncbi.nlm.nih.gov/10147098
https://pubmed.ncbi.nlm.nih.gov/10147098
Autor:
B D, Roberts
Publikováno v:
Journal of insurance medicine (New York, N.Y.). 26(1)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::16d322f716dacfc7922b91848565a9c0
https://pubmed.ncbi.nlm.nih.gov/10147063
https://pubmed.ncbi.nlm.nih.gov/10147063
Publikováno v:
AIDS (London, England). 7(7)
To examine the inter-relationship between HIV-1 infection and the cell surface receptors for tumor necrosis factor (TNF)-alpha, an immunoregulatory cytokine that can enhance HIV-1 replication.Infected promyelocytic and promonocytic cells were examine
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::a8147c096fbbe1e2ee0ab6c3e1717634
https://pubmed.ncbi.nlm.nih.gov/8395188
https://pubmed.ncbi.nlm.nih.gov/8395188
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 150(2)
Using the CD4+ model of chronic HIV-1 infection, OM-10.1, we investigated the influence of TNF-alpha regulatory networks on induced viral expression. Previously, OM-10.1 cultures were characterized to respond to exogenous TNF-alpha, as nearly 100% of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::33a6d26c0745162ead1ec9a2ed6f3988
https://pubmed.ncbi.nlm.nih.gov/8380428
https://pubmed.ncbi.nlm.nih.gov/8380428
Autor:
B. D. Roberts, A. Clearfield
Publikováno v:
Inorganic Chemistry. 27:3237-3240
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4b6d605de4045d17b0b75cc9771513b0
https://doi.org/10.1021/ic00291a040
https://doi.org/10.1021/ic00291a040