Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Bérengère Valtat"'
Autor:
Lisa M. Nicholas, Bérengère Valtat, Anya Medina, Lotta Andersson, Mia Abels, Inês G. Mollet, Deepak Jain, Lena Eliasson, Nils Wierup, Malin Fex, Hindrik Mulder
Publikováno v:
Molecular Metabolism, Vol 6, Iss 7, Pp 651-663 (2017)
Objective: Insulin release from pancreatic β-cells is controlled by plasma glucose levels via mitochondrial fuel metabolism. Therefore, insulin secretion is critically dependent on mitochondrial DNA (mtDNA) and the genes it encodes. Mitochondrial tr
Externí odkaz:
https://doaj.org/article/7c8420984bd24f1c94b700a9d5958e76
Autor:
Jiangming Sun, Yang De Marinis, Peter Osmark, Pratibha Singh, Annika Bagge, Bérengère Valtat, Petter Vikman, Peter Spégel, Hindrik Mulder
Publikováno v:
PLoS ONE, Vol 11, Iss 10, p e0164962 (2016)
RNA editing is a post-transcriptional alteration of RNA sequences that, via insertions, deletions or base substitutions, can affect protein structure as well as RNA and protein expression. Recently, it has been suggested that RNA editing may be more
Externí odkaz:
https://doaj.org/article/06f86d308cd74c9d86fcfc09592bffd8
Autor:
Lotta E Andersson, Bérengère Valtat, Annika Bagge, Vladimir V Sharoyko, David G Nicholls, Philippe Ravassard, Raphael Scharfmann, Peter Spégel, Hindrik Mulder
Publikováno v:
PLoS ONE, Vol 10, Iss 3, p e0120879 (2015)
Studies on beta cell metabolism are often conducted in rodent beta cell lines due to the lack of stable human beta cell lines. Recently, a human cell line, EndoC-βH1, was generated. Here we investigate stimulus-secretion coupling in this cell line,
Externí odkaz:
https://doaj.org/article/1dce2a75662a436da2a7511d4e83b1f0
Autor:
Bertrand Blondeau, Iman Sahly, Emmanuelle Massouridès, Amrit Singh-Estivalet, Bérengère Valtat, Delphine Dorchene, Frédéric Jaisser, Bernadette Bréant, Francois Tronche
Publikováno v:
PLoS ONE, Vol 7, Iss 2, p e30210 (2012)
Conditional gene deletion in specific cell populations has helped the understanding of pancreas development. Using this approach, we have shown that deleting the glucocorticoid receptor (GR) gene in pancreatic precursor cells leads to a doubled beta-
Externí odkaz:
https://doaj.org/article/5539b34367ce41bcb9b2446660cb8495
Autor:
Inês G. Mollet, Malin Fex, Lisa M. Nicholas, Bérengère Valtat, Nils Wierup, Lena Eliasson, Mia Abels, Deepak Jain, Hindrik Mulder, Lotta Andersson, Anya Medina
Publikováno v:
Molecular Metabolism
Molecular Metabolism, Vol 6, Iss 7, Pp 651-663 (2017)
Molecular Metabolism, Vol 6, Iss 7, Pp 651-663 (2017)
Objective Insulin release from pancreatic β-cells is controlled by plasma glucose levels via mitochondrial fuel metabolism. Therefore, insulin secretion is critically dependent on mitochondrial DNA (mtDNA) and the genes it encodes. Mitochondrial tra
Autor:
Bérengère Valtat, Hindrik Mulder, Jiangming Sun, Pratibha Singh, Yang De Marinis, Peter Osmark, Petter Vikman, Peter Spégel, Annika Bagge
Publikováno v:
PLoS ONE
PLoS ONE, Vol 11, Iss 10, p e0164962 (2016)
PLoS ONE, Vol 11, Iss 10, p e0164962 (2016)
RNA editing is a post-transcriptional alteration of RNA sequences that, via insertions, deletions or base substitutions, can affect protein structure as well as RNA and protein expression. Recently, it has been suggested that RNA editing may be more
Autor:
Vladimir V. Sharoyko, Raphael Scharfmann, David G. Nicholls, Annika Bagge, Peter Spégel, Philippe Ravassard, Lotta Andersson, Bérengère Valtat, Hindrik Mulder
Publikováno v:
PLoS ONE
PLoS ONE, 2015, pp.e0120879. ⟨10.1371/journal.pone.0120879.s004⟩
PLoS ONE; 10(3), no e0120879 (2015)
PLoS ONE, Public Library of Science, 2015, pp.e0120879. ⟨10.1371/journal.pone.0120879.s004⟩
PLoS ONE, Vol 10, Iss 3, p e0120879 (2015)
PLoS ONE, Public Library of Science, 2015, pp.e0120879. 〈10.1371/journal.pone.0120879.s004〉
PLoS ONE, 2015, pp.e0120879. ⟨10.1371/journal.pone.0120879.s004⟩
PLoS ONE; 10(3), no e0120879 (2015)
PLoS ONE, Public Library of Science, 2015, pp.e0120879. ⟨10.1371/journal.pone.0120879.s004⟩
PLoS ONE, Vol 10, Iss 3, p e0120879 (2015)
PLoS ONE, Public Library of Science, 2015, pp.e0120879. 〈10.1371/journal.pone.0120879.s004〉
International audience; AIMS/HYPOTHESIS:Studies on beta cell metabolism are often conducted in rodent beta cell lines due to the lack of stable human beta cell lines. Recently, a human cell line, EndoC-βH1, was generated. Here we investigate stimulu
Autor:
Jean-Pierre Riveline, Pascal Ferré, Ping Zhang, Bérengère Valtat, Nicolas Venteclef, Jean-François Gautier, B. Blondeau, Mathieu Armanet, A. Besseiche, Bernadette Breant, Amrit Singh-Estivalet, Daniel P. Kelly, François Tronche
Publikováno v:
Diabetes
Diabetes, American Diabetes Association, 2013, 62 (4), pp.1206-1216. ⟨10.2337/db12-0314⟩
Diabetes, 2013, 62 (4), pp.1206-1216. ⟨10.2337/db12-0314⟩
Diabetes, American Diabetes Association, 2013, 62 (4), pp.1206-1216. ⟨10.2337/db12-0314⟩
Diabetes, 2013, 62 (4), pp.1206-1216. ⟨10.2337/db12-0314⟩
International audience; Adult beta-cell dysfunction, a hallmark of type 2 diabetes, can be programmed by adverse fetal environment. We have shown that fetal glucocorticoids (GCs) participate in this programming through inhibition of beta-cell develop
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0a679ea9efec5b2e4d7cd8c3c5e830f3
https://hal.archives-ouvertes.fr/hal-01543309
https://hal.archives-ouvertes.fr/hal-01543309
Autor:
Bernadette Breant, A. Bailly, Bérengère Valtat, J. Callebert, Mathieu Armanet, B. Blondeau, Jean-Pierre Riveline, Emmanuelle Massouridès, J.F. Gautier, Arndt Benecke, Amrit Singh-Estivalet, François Tronche
Publikováno v:
Diabetes & Metabolism. 38:A20
Introduction Notre objectif est de comprendre les mecanismes qui regulent le developpement et la fonction des cellules β pour apprehender les places respectives des anomalies de masse et de fonction β dans l’etiologie du diabete de type 2. Nos tr
Autor:
Bérengère Valtat, Annie Durand, Mathieu Armanet, E. Ramond, Jennifer Rieusset, Jean-Pierre Riveline, Bernadette Breant, B. Blondeau
Publikováno v:
Diabetes & Metabolism. 38:A69
Rationnel Notre equipe s’interesse aux regulateurs de la fonction beta pancreatique. Nos travaux precedents ont montre que la voie des glucocorticoides (GC) inhibe le developpement et la fonction des cellules beta. Plus specifiquement, nous avons m