Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Azusa Enomoto"'
Publikováno v:
Biological and Pharmaceutical Bulletin. 27:1388-1391
Although a blockade or lack of N-type Ca(2+) channels has been reported to suppress neuronal pathological processes in several animal models of pain and ischemic brain injury, information is still limited regarding the neuroprotective effects of a du
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b58d2b9825395096b0d6dd5cb79e3a70
https://doi.org/10.1248/bpb.27.1388
https://doi.org/10.1248/bpb.27.1388
Autor:
Tomoyuki Konda, Hajime Koganei, Tetsuya Ogawa, Kosaku Nitta, Shizuka Aritomi, Kazumi Niinuma, Azusa Enomoto
Publikováno v:
American journal of nephrology. 30(2)
Background: The L/N-type calcium channel blocker (CCB) cilnidipine has been demonstrated to suppress sympathetic nerve activity. In the present study, we investigated the effects of cilnidipine on the renin-angiotensin-aldosterone system (RAAS) in SH
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::eb186b381bd4d7ec777f1b89a6ef18b7
https://pubmed.ncbi.nlm.nih.gov/19325231
https://pubmed.ncbi.nlm.nih.gov/19325231
Publikováno v:
Biologicalpharmaceutical bulletin. 29(5)
The sympathetic nerve activity plays an important role on the renal function through the vasoactive system and the renin-angiotensin system. Although interest in the renal protective effects of anti-sympathetic agents has been increased, there are no
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d6417dab2b5b17dce4924b4913c611c6
https://pubmed.ncbi.nlm.nih.gov/16651722
https://pubmed.ncbi.nlm.nih.gov/16651722
Publikováno v:
Nephron. Physiology. 101(1)
Background/Aims: The L/N-type calcium channel blocker (CCB) cilnidipine has been demonstrated to suppress progressive renal disease in a variety of experimental models, but the characteristic effects of N-type calcium channel blocking action on renal
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a1bd0161af28305858d895bdbad6cc2c
https://pubmed.ncbi.nlm.nih.gov/15886499
https://pubmed.ncbi.nlm.nih.gov/15886499