Zobrazeno 1 - 10
of 32
pro vyhledávání: '"Ayten Kandilci"'
Autor:
Emrah Özçelik, Ahmet Kalaycı, Büşra Çelik, Açelya Avcı, Hasan Akyol, İrfan Baki Kılıç, Türkan Güzel, Metin Çetin, Merve Tuzlakoğlu Öztürk, Zihni Onur Çalışkaner, Melike Tombaz, Dilan Yoleri, Özlen Konu, Ayten Kandilci
Publikováno v:
PLoS ONE, Vol 17, Iss 10, p e0275476 (2022)
DEK has a short isoform (DEK isoform-2; DEK2) that lacks amino acid residues between 49-82. The full-length DEK (DEK isoform-1; DEK1) is ubiquitously expressed and plays a role in different cellular processes but whether DEK2 is involved in these pro
Externí odkaz:
https://doaj.org/article/12d8b3f3b29b44a38c024ce11afae005
Autor:
Zihni Onur Çalışkaner, Türkan Çakar, Emrah Özçelik, Ahmet Özdilek, Annette S Kim, Öner Doğan, Amma Bosompem, Gerard Grosveld, Bülent Saka, Ayten Kandilci
Publikováno v:
PLoS ONE, Vol 12, Iss 5, p e0178025 (2017)
Overexpression of DEK oncogene is associated with increased proliferation of carcinoma cells and it is observed in several solid tumors due to the amplification of the 6p22.3 chromosomal region where DEK locates. Although the same chromosomal amplifi
Externí odkaz:
https://doaj.org/article/95856b20c4854a819afceeeeebc01fda
Autor:
Ayten Kandilci, Jacqueline Surtel, Laura Janke, Geoffrey Neale, Sabrina Terranova, Gerard C Grosveld
Publikováno v:
PLoS ONE, Vol 8, Iss 4, p e61706 (2013)
The MN1 oncogene is deregulated in human acute myeloid leukemia and its overexpression induces proliferation and represses myeloid differentiation of primitive human and mouse hematopoietic cells, leading to myeloid leukemia in mouse models. To delin
Externí odkaz:
https://doaj.org/article/f07a0178cade44ecab044bbf59c1da74
Autor:
İrfan Baki Kılıç, Açelya Yaşar, İrem Yalım Camcı, Türkan Güzel, Ayşegül Karahasan, Tamer Yağcı, Naci Çine, Ayten Kandilci
DEK locates in the nucleus of the cells or the cytoplasmic granules of neutrophils and plays different roles in cellular processes including NETosis, a suicide mechanism of neutrophils. Here we showed that the interaction of rDEK with CXCR2 leads to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9834a1b294bb6d0671a3fee3ab79ba4a
https://doi.org/10.22541/au.168067984.41873361/v1
https://doi.org/10.22541/au.168067984.41873361/v1
Supplementary Table 3 from Overexpression of N-Myc Rapidly Causes Acute Myeloid Leukemia in Mice
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::081e87875b3478a874c50c17135a8670
https://doi.org/10.1158/0008-5472.22371438
https://doi.org/10.1158/0008-5472.22371438
Supplementary Table 2 from Overexpression of N-Myc Rapidly Causes Acute Myeloid Leukemia in Mice
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::79370731c291f679aea38b96d464b57f
https://doi.org/10.1158/0008-5472.22371441
https://doi.org/10.1158/0008-5472.22371441
Supplementary Figure 1 from Overexpression of N-Myc Rapidly Causes Acute Myeloid Leukemia in Mice
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e728ce14b029b20c5c36081952050e1a
https://doi.org/10.1158/0008-5472.22371450.v1
https://doi.org/10.1158/0008-5472.22371450.v1
Supplementary Figure 2 from Overexpression of N-Myc Rapidly Causes Acute Myeloid Leukemia in Mice
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::572f13ee58128bf33233b6af37a48361
https://doi.org/10.1158/0008-5472.22371447.v1
https://doi.org/10.1158/0008-5472.22371447.v1
Supplementary Table 1 from Overexpression of N-Myc Rapidly Causes Acute Myeloid Leukemia in Mice
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::27e7c84ef20ecadc3cbfe09e026adf7c
https://doi.org/10.1158/0008-5472.22371444.v1
https://doi.org/10.1158/0008-5472.22371444.v1
Supplementary Table 4 from Overexpression of N-Myc Rapidly Causes Acute Myeloid Leukemia in Mice
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ae917544ed7c0bf3b4d2ea8ef8fc92cf
https://doi.org/10.1158/0008-5472.22371435.v1
https://doi.org/10.1158/0008-5472.22371435.v1