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Host-encoded antivirulence defenses: host physiologies teach pathogens to play nice.

Autor: Ayres JS; Molecular and Systems Physiology Lab, The Salk Institute for Biological Studies, 10010 N. Torrey Pines Road, La Jolla, CA 92037, USA; Gene Expression Lab, The Salk Institute for Biological Studies, 10010 N. Torrey Pines Road, La Jolla, CA 92037, USA; NOMIS Center for Immunobiology and Microbial Pathogenesis, Salk Institute for Biological Studies, La Jolla, CA, USA; Howard Hughes Medical Institute, The Salk Institute for Biological Studies, 10010 N. Torrey Pines Road, La Jolla, CA 92037, USA. Electronic address: jayres@salk.edu.

Publikováno v: Current opinion in immunology [Curr Opin Immunol] 2024 Oct 08; Vol. 91, pp. 102472. Date of Electronic Publication: 2024 Oct 08.

CD4+ T cells regulate sickness-induced anorexia and fat wasting during a chronic parasitic infection.

Autor: Redford SE; Molecular and Systems Physiology Lab, Salk Institute for Biological Studies, 10010 N. Torrey Pines Road, La Jolla, CA 92037, USA; Division of Biological Sciences, University of California, San Diego, La Jolla, CA 92037, USA; NOMIS Center for Immunobiology and Microbial Pathogenesis, Salk Institute for Biological Studies, La Jolla, CA, USA; Gene Expression Lab, Salk Institute for Biological Studies, 10010 N. Torrey Pines Road, La Jolla, CA 92037, USA., Varanasi SK; NOMIS Center for Immunobiology and Microbial Pathogenesis, Salk Institute for Biological Studies, La Jolla, CA, USA., Sanchez KK; Molecular and Systems Physiology Lab, Salk Institute for Biological Studies, 10010 N. Torrey Pines Road, La Jolla, CA 92037, USA; NOMIS Center for Immunobiology and Microbial Pathogenesis, Salk Institute for Biological Studies, La Jolla, CA, USA; Gene Expression Lab, Salk Institute for Biological Studies, 10010 N. Torrey Pines Road, La Jolla, CA 92037, USA., Thorup NR; Molecular and Systems Physiology Lab, Salk Institute for Biological Studies, 10010 N. Torrey Pines Road, La Jolla, CA 92037, USA; NOMIS Center for Immunobiology and Microbial Pathogenesis, Salk Institute for Biological Studies, La Jolla, CA, USA; Gene Expression Lab, Salk Institute for Biological Studies, 10010 N. Torrey Pines Road, La Jolla, CA 92037, USA., Ayres JS; Molecular and Systems Physiology Lab, Salk Institute for Biological Studies, 10010 N. Torrey Pines Road, La Jolla, CA 92037, USA; NOMIS Center for Immunobiology and Microbial Pathogenesis, Salk Institute for Biological Studies, La Jolla, CA, USA; Gene Expression Lab, Salk Institute for Biological Studies, 10010 N. Torrey Pines Road, La Jolla, CA 92037, USA. Electronic address: jayres@salk.edu.

Publikováno v: Cell reports [Cell Rep] 2023 Aug 29; Vol. 42 (8), pp. 112814. Date of Electronic Publication: 2023 Jul 24.

Cachexia: A systemic consequence of progressive, unresolved disease.

Autor: Ferrer M; Cold Spring Harbor Laboratory, Cold Spring Harbor, NY 11724, USA; MRC Cancer Unit, University of Cambridge, Hutchison Research Centre, Cambridge Biomedical Campus, Cambridge CB2 0XZ, UK., Anthony TG; Department of Nutritional Sciences, Rutgers School of Environmental and Biological Sciences, The State University of New Jersey, New Brunswick, NJ 08901, USA., Ayres JS; Salk Institute for Biological Studies, La Jolla, CA 92037, USA., Biffi G; University of Cambridge, Cancer Research UK Cambridge Institute, Li Ka Shing Centre, Cambridge CB2 0RE, UK., Brown JC; Pennington Biomedical Research Center, Louisiana State University System, Baton Rouge, LA 70808, USA., Caan BJ; Kaiser Permanente Northern California Division of Research, Oakland, CA 94612, USA., Cespedes Feliciano EM; Kaiser Permanente Northern California Division of Research, Oakland, CA 94612, USA., Coll AP; Wellcome Trust-MRC Institute of Metabolic Science and MRC Metabolic Diseases Unit, University of Cambridge, Cambridge CB2 0QQ, UK., Dunne RF; University of Rochester Medical Center, University of Rochester, Rochester, NY 14642, USA., Goncalves MD; Division of Endocrinology, Department of Medicine, Weill Cornell Medicine, New York, NY 10021, USA., Grethlein J; Ruprecht Karl University of Heidelberg, Heidelberg 69117, Germany., Heymsfield SB; Pennington Biomedical Research Center, Louisiana State University System, Baton Rouge, LA 70808, USA., Hui S; Harvard T.H. Chan School of Public Health, Harvard University, Boston, MA 02115, USA., Jamal-Hanjani M; Department of Medical Oncology, University College London Hospitals, London WC1E 6DD, UK; Cancer Research UK Lung Cancer Centre of Excellence and Cancer Metastasis Laboratory, University College London Cancer Institute, London WC1E 6DD, UK., Lam JM; Cancer Research UK Lung Cancer Centre of Excellence and Cancer Metastasis Laboratory, University College London Cancer Institute, London WC1E 6DD, UK., Lewis DY; The Beatson Institute for Cancer Research, Cancer Research UK, Glasgow G61 1BD, UK., McCandlish D; Cold Spring Harbor Laboratory, Cold Spring Harbor, NY 11724, USA., Mustian KM; University of Rochester Medical Center, University of Rochester, Rochester, NY 14642, USA., O'Rahilly S; Wellcome Trust-MRC Institute of Metabolic Science and MRC Metabolic Diseases Unit, University of Cambridge, Cambridge CB2 0QQ, UK., Perrimon N; Department of Genetics, Blavatnik Institute, Howard Hughes Medical Institute, Harvard Medical School, Boston, MA 02115, USA., White EP; Rutgers Cancer Institute of New Jersey, Department of Molecular Biology and Biochemistry, Rutgers University, The State University of New Jersey, New Brunswick, NJ 08901, USA; Ludwig Princeton Branch, Ludwig Institute for Cancer Research, Princeton University, Princeton, NJ 08544, USA., Janowitz T; Cold Spring Harbor Laboratory, Cold Spring Harbor, NY 11724, USA; Northwell Health Cancer Institute, Northwell Health, New Hyde Park, NY 11042, USA. Electronic address: janowitz@cshl.edu.

Publikováno v: Cell [Cell] 2023 Apr 27; Vol. 186 (9), pp. 1824-1845.

Chronic Leptin Deficiency Improves Tolerance of Physiological Damage and Host-Pathogen Cooperation during Yersinia pseudotuberculosis Infection.

Autor: Sanchez KK; Molecular and Systems Physiology Lab, Gene Expression Lab, Nomis Center for Immunobiology and Microbial Pathogenesis, Salk Institute for Biological Studiesgrid.250671.7, La Jolla, California, USA., Troha K; Molecular and Systems Physiology Lab, Gene Expression Lab, Nomis Center for Immunobiology and Microbial Pathogenesis, Salk Institute for Biological Studiesgrid.250671.7, La Jolla, California, USA., Mu A; Molecular and Systems Physiology Lab, Gene Expression Lab, Nomis Center for Immunobiology and Microbial Pathogenesis, Salk Institute for Biological Studiesgrid.250671.7, La Jolla, California, USA., Redford SE; Molecular and Systems Physiology Lab, Gene Expression Lab, Nomis Center for Immunobiology and Microbial Pathogenesis, Salk Institute for Biological Studiesgrid.250671.7, La Jolla, California, USA.; Division of Biological Sciences, University of California, San Diego, La Jolla, California, USA., McCarville JL; Molecular and Systems Physiology Lab, Gene Expression Lab, Nomis Center for Immunobiology and Microbial Pathogenesis, Salk Institute for Biological Studiesgrid.250671.7, La Jolla, California, USA., Insenga A; Molecular and Systems Physiology Lab, Gene Expression Lab, Nomis Center for Immunobiology and Microbial Pathogenesis, Salk Institute for Biological Studiesgrid.250671.7, La Jolla, California, USA., Stengel S; Molecular and Systems Physiology Lab, Gene Expression Lab, Nomis Center for Immunobiology and Microbial Pathogenesis, Salk Institute for Biological Studiesgrid.250671.7, La Jolla, California, USA., Lee YM; Molecular and Systems Physiology Lab, Gene Expression Lab, Nomis Center for Immunobiology and Microbial Pathogenesis, Salk Institute for Biological Studiesgrid.250671.7, La Jolla, California, USA., Ayres JS; Molecular and Systems Physiology Lab, Gene Expression Lab, Nomis Center for Immunobiology and Microbial Pathogenesis, Salk Institute for Biological Studiesgrid.250671.7, La Jolla, California, USA.

Publikováno v: Infection and immunity [Infect Immun] 2022 Sep 15; Vol. 90 (9), pp. e0024222. Date of Electronic Publication: 2022 Aug 04.