Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Aylin Domaniku"'
Autor:
Samet Agca, Aylin Domaniku‐Waraich, Sevval Nur Bilgic, Melis Sucuoglu, Meric Dag, Sukru Anil Dogan, Serkan Kir
Publikováno v:
Journal of Cachexia, Sarcopenia and Muscle, Vol 15, Iss 5, Pp 1898-1914 (2024)
Abstract Background Tumour‐induced skeletal muscle wasting in the context of cancer cachexia is a condition with profound implications for patient survival. The loss of muscle mass is a significant clinical obstacle and is linked to reduced toleran
Externí odkaz:
https://doaj.org/article/d0200d5c1178411a990b0436bd6dd43a
Autor:
Bahar Zehra Camurdanoglu Weber, Samet Agca, Aylin Domaniku, Sevval Nur Bilgic, Dilsad H. Arabaci, Serkan Kir
Publikováno v:
Journal of Cachexia, Sarcopenia and Muscle, Vol 13, Iss 3, Pp 1582-1594 (2022)
Abstract Background Lung cancer is the primary cause of cancer deaths worldwide. Activation of epidermal growth factor receptor (EGFR) leads to lung cancer progression and poor prognosis while involuntary weight loss remains a major problem. Tumour
Externí odkaz:
https://doaj.org/article/0c4c3930c4bf4348aec010f619d43e47
Publikováno v:
Proceedings, Vol 2, Iss 25, p 1590 (2018)
Genetic alterations affect carcinogenesis, and recent studies demonstrated that long non-coding RNAs (lncRNAs) have critical roles during this process. Hox transcript antisense intergenic RNA (HOTAIR) is a lncRNA molecule that affects proliferation,
Externí odkaz:
https://doaj.org/article/44f394b71d97411790f48cfbe465d017
SummaryProgressive weakness and muscle loss are associated with multiple chronic conditions including muscular dystrophy and cancer. Cancer-associated cachexia, characterized by dramatic weight loss and fatigue, leads to reduced quality of life and p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::46932c6cac41e38d24d23cc634bd8761
https://doi.org/10.1101/2023.01.26.525658
https://doi.org/10.1101/2023.01.26.525658
Autor:
Sevval Nur Bilgic, Aylin Domaniku, Batu Toledo, Samet Agca, Bahar Z. C. Weber, Dilsad H. Arabaci, Zeynep Ozornek, Pascale Lause, Jean-Paul Thissen, Audrey Loumaye, Serkan Kir
SummarySkeletal muscle atrophy is a hallmark of the cachexia syndrome that is associated with poor survival and reduced quality of life in cancer patients1. Muscle atrophy involves excessive protein catabolism and loss of muscle mass and strength2. A
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c1bb65d1df7b203e6888336a9e2b7ae6
https://doi.org/10.1101/2023.01.23.525138
https://doi.org/10.1101/2023.01.23.525138
Autor:
Sevval Nur Bilgic, Aylin Domaniku, Batu Toledo, Samet Agca, Bahar Z. C. Weber, Dilsad H. Arabaci, Zeynep Ozornek, Pascale Lause, Jean-Paul Thissen, Audrey Loumaye, Serkan Kir
Publikováno v:
Nature, Vol. 617, no. 7962, p. 827-834 (2023)
Skeletal muscle atrophy is a hallmark of the cachexia syndrome that is associated with poor survival and reduced quality of life in patients with cancer. Muscle atrophy involves excessive protein catabolism and loss of muscle mass and strength. An ef
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c94866902808b779fc04a2d13ba973dc
https://hdl.handle.net/2078.1/276654
https://hdl.handle.net/2078.1/276654
Autor:
Jennie Olofsson, Peter Lönnerberg, Caroline Ingre, Elena Rodriguez-Vieitez, Inci Sevval Aksoylu, Caroline Mijnsbergen, Manuela Lehmann, Jan H. Veldink, Inti von Gohren Antequera, Albert C. Ludolph, Anna Szczepińska, Stefan Wouters, Mathias Uhlén, Ulf Kläppe, Eleonora Aronica, Hermieneke Vergunst-Bosch, Anna Månberg, Julia Remnestål, Sebastian A. Lewandowski, Lwaki Ebarasi, Peter Nilsson, Aylin Domaniku, Robert A. Harris, Nathan G. Skene, Jasper J. Anink, Joke De Vocht, Eva Hedlund, Annemarie Hübers, Koen Poesen, Philip Van Damme, Marta Trusohamn, Jonathan D. Gilthorpe, Maxim De Schaepdryver, Folkert Sanders
Publikováno v:
Nature medicine, 27(4), 640-646. Nature Publishing Group
Nat Med
Nature medicine 27(4), 640-646 (2021). doi:10.1038/s41591-021-01295-9
Nat Med
Nature medicine 27(4), 640-646 (2021). doi:10.1038/s41591-021-01295-9
Apart from well-defined factors in neuronal cells1, only a few reports consider that the variability of sporadic amyotrophic lateral sclerosis (ALS) progression can depend on less-defined contributions from glia2,3 and blood vessels4. In this study w
Autor:
Eva Hedlund, Caroline Ingre, Jan H. Veldink, Jonathan D. Gilthorpe, Caroline Mijnsbergen, Inti von Gohren Antequera, Annemarie Hübers, Eleonora Aronica, Maxim De Schaepdryver, Aylin Domaniku, Ulf Kläppe, Sebastian A. Lewandowski, Lwaki Ebarasi, Jennie Olofsson, Jasper J. Anink, Koen Poesen, Julia Remnestål, Robert A. Harris, Folkert Sanders, Stefan Wouters, Peter Nilsson, Elena Rodriguez-Vieitez, Inci Sevval Aksoylu, Manuela Lehmann, Nathan G. Skene, Albert C. Ludolph, Anna Månberg, Joke De Vocht, Anna Szczepińska, Mathias Uhlén, Philip Van Damme, Marta Trusohamn, Peter Lönnerberg, Hermieneke Vergunst-Bosch
Publikováno v:
Nature medicine 27(7), 1308-1308 (2021). doi:10.1038/s41591-021-01414-6
In the version of this article initially published, the label along the right margin of the top row in Fig. 2d (SO1DG93A) was incorrect. The correct label is ‘SOD1G93A’. The error has been corrected in the HTML and PDF versions of the article.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bb869fc574bc3c3fd27b25b90be5d0ac
https://pub.dzne.de/record/155543
https://pub.dzne.de/record/155543