Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Ayako Fukami"'
Autor:
Masaki Kondo, Tatsuhito Himeno, Jiro Kato, Shin Tsunekawa, Jiro Nakamura, Keiko Naruse, Tetsuji Okawa, Eita Uenishi, Yusuke Seino, Ayako Fukami, Yutaka Oiso, Hideki Kamiya, Yoji Hamada, Taiga Shibata, Atsushi Fujiya
Publikováno v:
Journal of Diabetes and its Complications. 28:588-595
Aims/hypothesis Although the initial healing stage involves a re-epithelialization in humans, diabetic foot ulceration (DFU) has been investigated using rodent models with wounds on the thigh skin, in which a wound contraction is initiated. In this s
Autor:
Yusuke Seino, Tatsuhito Himeno, Jiro Kato, Eita Uenishi, Hideki Kamiya, Shin Tsunekawa, Atsushi Fujiya, Jiro Nakamura, Tetsuji Okawa, Ayako Fukami, Hiroshi Nagasaki, Yutaka Oiso, Yoji Hamada
Publikováno v:
Obesity. 22:371-379
Objective The S100 calcium binding protein B (S100B) implicated in brain inflammation acts via the receptor of advanced glycation end products (RAGE) and is also secreted from adipocytes. We investigated the role of S100B in the interaction between a
Autor:
Daniel J. Drucker, Yoshiko Takagishi, Michiyo Yamamoto, Eriko Sakamoto-Miura, Shin Tsunekawa, Ayako Fukami, Yusuke Seino, Yutaka Seino, Yoshiharu Murata, Chisato Sugiyama, Yoshitaka Hayashi, Nobuaki Ozaki, Safina Ali, Tatsuhito Himeno, Yutaka Oiso
Publikováno v:
Diabetes
Glucagon and glucagon-like peptide-1 (GLP-1) are produced in pancreatic a-cells and enteroendocrine L-cells, respectively, in a tissue-specific manner from the same precursor, proglucagon, that is encoded by glucagon gene (Gcg), and play critical rol
Autor:
Kota Ishikawa, Norio Harada, Yusuke Seino, Yukiyasu Toyoda, Naohiro Mizutani, Jiro Nakamura, Eriko Sakamoto, Shin Tsunekawa, Ayako Fukami, Nobuya Inagaki, Hidetada Ogata, Eita Uenishi, Ichitomo Miwa, Hideki Kamiya, Yoji Hamada, Nobuaki Ozaki, Hiroyuki Sato, Yutaka Oiso
Publikováno v:
Journal of Diabetes Investigation. 3:432-440
Aims/Introduction: Excessive intake of sucrose can cause severe health issues, such as diabetes mellitus. In animal studies, consumption of a high-sucrose diet (SUC) has been shown to cause obesity, insulin resistance and glucose intolerance. However
Autor:
Katsumi Shibata, Jun Sato, Yoshitaka Hayashi, Hiroki Miyahira, Yutaka Oiso, Yoshiko Takagishi, Michiyo Yamamoto, Chika Watanabe, Ayako Fukami, Tsutomu Fukuwatari, Nobuaki Ozaki, Yoshiharu Murata, Yusuke Seino
Publikováno v:
Diabetes
Glucagon is believed to be one of the most important peptides for upregulating blood glucose levels. However, homozygous glucagon–green fluorescent protein (gfp) knock-in mice (Gcggfp/gfp: GCGKO) are normoglycemic despite the absence of proglucagon
Autor:
Atsushi, Fujiya, Hiroshi, Nagasaki, Yusuke, Seino, Tetsuji, Okawa, Jiro, Kato, Ayako, Fukami, Tatsuhito, Himeno, Eita, Uenishi, Shin, Tsunekawa, Hideki, Kamiya, Jiro, Nakamura, Yutaka, Oiso, Yoji, Hamada
Publikováno v:
Obesity (Silver Spring, Md.). 22(2)
The S100 calcium binding protein B (S100B) implicated in brain inflammation acts via the receptor of advanced glycation end products (RAGE) and is also secreted from adipocytes. We investigated the role of S100B in the interaction between adipocytes
Autor:
Nobuaki Ozaki, Yoshitaka Hayashi, Hidetada Ogata, Kota Ishikawa, Keita Kinoshita, Yutaka Oiso, Hiroshi Arima, Yusuke Takagi, Ayako Fukami, Ryuya Maekawa, Shin Tsunekawa, Yusuke Seino, Atsushi Iida, Takako Izumoto, Daisuke Yabe, Shinobu Shimizu, Yoji Hamada
Publikováno v:
Diabetologia
Aims/hypothesis The action of incretin hormones including glucose-dependent insulinotropic polypeptide (GIP) and glucagon-like peptide-1 (GLP-1) is potentiated in animal models defective in glucagon action. It has been reported that such animal model
Autor:
Jiro Kato1, Hideki Kamiya1,2 hkamiya@aichi-med-u.ac.jp, Tatsuhito Himeno1, Taiga Shibata1, Masaki Kondo1, Tetsuji Okawa1, Atsushi Fujiya1, Ayako Fukami1, Eita Uenishi1, Yusuke Seino1,3, Shin Tsunekawa1, Yoji Hamada1,3, Keiko Naruse4, Yutaka Oiso1, Jiro Nakamura1
Publikováno v:
Journal of Diabetes & its Complications. 2014, Vol. 28 Issue 5, p588-595. 8p.