Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Aurora Rodríguez-Martínez"'
Autor:
Juan Carlos Balandrán, José Dávila-Velderrain, Antonio Sandoval-Cabrera, Gabriela Zamora-Herrera, Vanessa Terán-Cerqueda, Lilia Adela García-Stivalet, José Alejandro Limón-Flores, Erick Armenta-Castro, Aurora Rodríguez-Martínez, Bertha Alicia Leon-Chavez, Verónica Vallejo-Ruiz, Duane C. Hassane, Sonia Mayra Pérez-Tapia, Vianney Ortiz-Navarrete, Monica L. Guzman, Rosana Pelayo
Publikováno v:
Frontiers in Immunology, Vol 12 (2021)
B-cell acute lymphoblastic leukemia (B-ALL) results from the expansion of malignant lymphoid precursors within the bone marrow (BM), where hematopoietic niches and microenvironmental signals provide leukemia-initiating cells (LICs) the conditions to
Externí odkaz:
https://doaj.org/article/6caa116391044fefa37ea44363f1c043
Autor:
Jussara Rios de los Rios, Jennifer Enciso, Armando Vilchis-Ordoñez, Ricardo Vázquez-Ramírez, Dalia Ramirez-Ramirez, Juan Carlos Balandrán, Aurora Rodríguez-Martínez, Martha Ruiz-Tachiquín, Ericka Pompa-Mera, Luis Mendoza, Gustavo Pedraza-Alva, Hector Mayani, Muller Fabbri, Rosana Pelayo
Publikováno v:
Journal of Leukocyte Biology. 112:31-45
Leukemogenesis is proposed to result from the continuous interplay between inducive bone marrow (BM) microenvironments and malignant precursor cells. Recent findings point toward an abnormal production of proinflammatory mediators within the BM from
Autor:
Sonia Mayra Pérez-Tapia, Erick Armenta-Castro, Vianney Ortiz-Navarrete, Jose Davila-Velderrain, Lilia Adela García-Stivalet, Aurora Rodríguez-Martínez, Rosana Pelayo, Vanessa Terán-Cerqueda, Monica L. Guzman, Antonio Sandoval-Cabrera, Duane C. Hassane, Bertha Alicia León-Chávez, Verónica Vallejo-Ruiz, Gabriela Zamora-Herrera, José Alejandro Limón-Flores, Juan Carlos Balandrán
Publikováno v:
Frontiers in Immunology, Vol 12 (2021)
Frontiers in Immunology
Frontiers in Immunology
B-cell acute lymphoblastic leukemia (B-ALL) results from the expansion of malignant lymphoid precursors within the bone marrow (BM), where hematopoietic niches and microenvironmental signals provide leukemia-initiating cells (LICs) the conditions to