Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Auria Godard"'
Autor:
Sara El Hoss, Sylvie Cochet, Auria Godard, Hongxia Yan, Michaël Dussiot, Giacomo Frati, Bénédicte Boutonnat-Faucher, Sandrine Laurance, Olivier Renaud, Laure Joseph, Annarita Miccio, Valentine Brousse, Mohandas Narla, Wassim El Nemer
Publikováno v:
Haematologica, Vol 106, Iss 10 (2020)
While ineffective erythropoiesis has long been recognized as a key contributor to anemia in thalassemia, its role in anemia of sickle cell disease (SCD) has not been critically explored. Using in vitro and in vivo derived human erythroblasts we asses
Externí odkaz:
https://doaj.org/article/aaaa847b38394956a803db1d63a540dd
Publikováno v:
Current Opinion in Hematology
Current Opinion in Hematology, 2021, 28 (3), pp.171-176. ⟨10.1097/MOH.0000000000000642⟩
Current Opinion in Hematology, Lippincott, Williams & Wilkins, 2021, 28 (3), pp.171-176. ⟨10.1097/MOH.0000000000000642⟩
Current Opinion in Hematology, 2021, 28 (3), pp.171-176. ⟨10.1097/MOH.0000000000000642⟩
Current Opinion in Hematology, Lippincott, Williams & Wilkins, 2021, 28 (3), pp.171-176. ⟨10.1097/MOH.0000000000000642⟩
Purpose of review Sickle cell disease (SCD) is a hemolytic anemia caused by a point mutation in the β globin gene leading to the expression of an abnormal hemoglobin (HbS) that polymerizes under hypoxic conditions driving red cell sickling. Circulat
Autor:
Benedicte Boutonnat-Faucher, Sylvie Cochet, Sara El Hoss, Mickael Dussiot, Annarita Miccio, Sandrine Laurance, Hongxia Yan, Olivier Renaud, Wassim El Nemer, Laure Joseph, Narla Mohandas, Valentine Brousse, Giacomo Frati, Auria Godard
Publikováno v:
Transfusion Clinique et Biologique. 28:S47
Autor:
Hongxia Yan, Mohandas Narla, Sylvie Cochet, Sandrine Laurance, Giacomo Frati, Olivier Renaud, Laure Joseph, Annarita Miccio, Auria Godard, Benedicte Boutonnat-Faucher, Wassim El Nemer, Valentine Brousse, Michael Dussiot, Sara El Hoss
Publikováno v:
Blood. 136:14-15
Sickle cell disease (SCD) is an autosomal hereditary recessive disorder caused by a point mutation in the β globin gene resulting in a Glu-to-Val substitution at the 6th position of the β globin protein. The resulting abnormal hemoglobin (HbS) poly