Zobrazeno 1 - 10
of 35
pro vyhledávání: '"Aurélie Hautefort"'
Autor:
Andrea Peloso, Stéphanie Lacotte, Quentin Gex, Florence Slits, Beat Moeckli, Graziano Oldani, Matthieu Tihy, Aurélie Hautefort, Brenda Kwak, Laura Rubbia-Brandt, Christian Toso
Publikováno v:
PLoS ONE, Vol 18, Iss 12 (2023)
Externí odkaz:
https://doaj.org/article/afcfd49c6704415a8412e784975d0771
Publikováno v:
Vascular Biology, Vol 1, Iss 1, Pp H117-H124 (2019)
Gap junctions are essential for intercellular crosstalk in blood and lymphatic vasculature. These clusters of intercellular channels ensure direct communication among endothelial cells and between endothelial and smooth muscle cells, and the synchron
Externí odkaz:
https://doaj.org/article/7dd45f0ef8b94dbf8e26dbc91f591f07
Publikováno v:
International Journal of Molecular Sciences, Vol 22, Iss 11, p 5734 (2021)
This review highlights current knowledge on the expression and function of connexins and pannexins, transmembrane channel proteins that play an important role in intercellular communication, in both the developing and mature lymphatic vasculature. A
Externí odkaz:
https://doaj.org/article/2193825301ca4cc0bace4ec443c3ba5e
Autor:
Jean-François Denis, Mannekomba R. Diagbouga, Filippo Molica, Aurélie Hautefort, Tanja Linnerz, Masakatsu Watanabe, Sylvain Lemeille, Julien Y. Bertrand, Brenda R. Kwak
Publikováno v:
Frontiers in Physiology, Vol 10 (2019)
Shear stress, a blood flow-induced frictional force, is essential in the control of endothelial cell (EC) homeostasis. High laminar shear stress (HLSS), as observed in straight parts of arteries, assures a quiescent non-activated endothelium through
Externí odkaz:
https://doaj.org/article/d4e39aa6517b4912b2f208c2ef0119b3
Autor:
V. Elyasigomari, SJ Wort, Frédéric Perros, Bingling Xu, Kian Fan Chung, Sharon Mumby, W Xu, Aurélie Hautefort, Grégoire Manaud, Christopher K.M. Hui, Marc Humbert, Ian M. Adcock
Publikováno v:
Pulmonary Circulation
Pulmonary Circulation, Vol 11 (2021)
Pulmonary Circulation, Vol 11 (2021)
Pulmonary arterial hypertension describes a group of diseases characterised by raised pulmonary vascular resistance, resulting from vascular remodelling in the pre-capillary resistance arterioles. Left untreated, patients die from right heart failure
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::47d45fc5a84c0f35266df6fdcc1bd4fa
http://hdl.handle.net/10044/1/85866
http://hdl.handle.net/10044/1/85866
Autor:
Séverine Remy, Juliette Bignard, Audrey Courboulin, Gérald Simonneau, Maria-Rosa Ghigna, Marc Humbert, Ignacio Anegon, Mélanie Lambert, Fabrice Antigny, Monica Florio, Esther J. Nossent, Banghua Sun, Harm Jan Bogaard, Elie Fadel, Aurélie Hautefort, Anton Vonk Noordegraaf, Angèle Boet, Barbara Girerd, Maria-Candida Vinhas, Grégoire Manaud, Sophie Nadaud, Frédéric Perros, Stijn E. Verleden, Olivier Claude, Sébastien J. Dumas, Florent Soubrier, Peter Dorfmüller, Benoit Ranchoux, Florence Lecerf, Olaf Mercier, David Montani, Katrien Grünberg
Publikováno v:
American Journal of Respiratory Cell and Molecular Biology
American Journal of Respiratory Cell and Molecular Biology, 2020, Epub ahead of print. ⟨10.1165/rcmb.2019-0015OC⟩
American Journal of Respiratory Cell and Molecular Biology, 63(1), 118-131. American Thoracic Society
American Journal of Respiratory Cell and Molecular Biology, American Thoracic Society, 2020, Epub ahead of print. ⟨10.1165/rcmb.2019-0015OC⟩
Manaud, G, Nossent, E J, Lambert, M, Ghigna, M R, Boet, A, Vinhas, M C, Ranchoux, B, Dumas, S J, Courboulin, A, Girerd, B, Soubrier, F, Bignard, J, Claude, O, Lecerf, F, Hautefort, A, Florio, M, Sun, B, Nadaud, S, Verleden, S E, Remy, S, Anegon, I, Bogaard, H J, Mercier, O, Fadel, E, Simonneau, G, Noordegraaf, A V, Grunberg, K, Humbert, M, Montani, D, Dorfmuller, P, Antigny, F & Perros, F 2020, ' Comparison of human and experimental pulmonary veno-occlusive disease ', American Journal of Respiratory Cell and Molecular Biology, vol. 63, no. 1, pp. 118-131 . https://doi.org/10.1165/rcmb.2019-0015OC
American journal of respiratory cell and molecular biology
American Journal of Respiratory Cell and Molecular Biology, 2020, Epub ahead of print. ⟨10.1165/rcmb.2019-0015OC⟩
American Journal of Respiratory Cell and Molecular Biology, 63(1), 118-131. American Thoracic Society
American Journal of Respiratory Cell and Molecular Biology, American Thoracic Society, 2020, Epub ahead of print. ⟨10.1165/rcmb.2019-0015OC⟩
Manaud, G, Nossent, E J, Lambert, M, Ghigna, M R, Boet, A, Vinhas, M C, Ranchoux, B, Dumas, S J, Courboulin, A, Girerd, B, Soubrier, F, Bignard, J, Claude, O, Lecerf, F, Hautefort, A, Florio, M, Sun, B, Nadaud, S, Verleden, S E, Remy, S, Anegon, I, Bogaard, H J, Mercier, O, Fadel, E, Simonneau, G, Noordegraaf, A V, Grunberg, K, Humbert, M, Montani, D, Dorfmuller, P, Antigny, F & Perros, F 2020, ' Comparison of human and experimental pulmonary veno-occlusive disease ', American Journal of Respiratory Cell and Molecular Biology, vol. 63, no. 1, pp. 118-131 . https://doi.org/10.1165/rcmb.2019-0015OC
American journal of respiratory cell and molecular biology
Pulmonary veno-occlusive disease (PVOD) occurs in humans either as a heritable form (hPVOD) due to biallelic inactivating mutations of EIF2AK4 (encoding GCN2) or as a sporadic form in older age (sPVOD). The chemotherapeutic agent mitomycin C (MMC) is
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8f6a44aa70dbdb40b0e9cfded999f5f8
https://lirias.kuleuven.be/handle/123456789/653108
https://lirias.kuleuven.be/handle/123456789/653108
Autor:
Thomas Bertero, Catherine Rucker-Martin, David Montani, Laurent Tesson, Marc Humbert, Fabrice Antigny, Frédéric Perros, Séverine Remy, Véronique Capuano, Barbara Girerd, Ignacio Anegon, Boris Manoury, Christine Péchoux, Olaf Mercier, Mélanie Lambert, Valérie Domergue, Morad K. Nakhleh, Aurélie Hautefort, Angèle Boet
Publikováno v:
Circulation Research
Circulation Research, American Heart Association, 2019, 125 (7), pp.678-695. ⟨10.1161/CIRCRESAHA.119.314793⟩
Circulation Research, 2019, 125 (7), pp.678-695. ⟨10.1161/CIRCRESAHA.119.314793⟩
Circulation Research, American Heart Association, 2019, 125 (7), pp.678-695. ⟨10.1161/CIRCRESAHA.119.314793⟩
Circulation Research, 2019, 125 (7), pp.678-695. ⟨10.1161/CIRCRESAHA.119.314793⟩
Rationale: Pulmonary arterial hypertension is a severe lethal cardiopulmonary disease. Loss of function mutations in KCNK3 (potassium channel subfamily K member 3) gene, which encodes an outward rectifier K + channel, have been identified in pulmonar
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bb2e5a85d626ceb4ed23c4529ed9583c
https://hal.archives-ouvertes.fr/hal-02346635/document
https://hal.archives-ouvertes.fr/hal-02346635/document
Autor:
Mélanie, Lambert, Véronique, Capuano, Angèle, Boet, Laurent, Tesson, Thomas, Bertero, Morad K, Nakhleh, Séverine, Remy, Ignacio, Anegon, Christine, Pechoux, Aurélie, Hautefort, Catherine, Rucker-Martin, Boris, Manoury, Valérie, Domergue, Olaf, Mercier, Barbara, Girerd, David, Montani, Frédéric, Perros, Marc, Humbert, Fabrice, Antigny
Publikováno v:
Circulation research. 125(7)
Pulmonary arterial hypertension is a severe lethal cardiopulmonary disease. Loss of function mutations inWe have demonstrated that KCNK3 dysfunction is common to heritable and nonheritable pulmonary arterial hypertension and to experimental pulmonary
Autor:
Graziano Pelli, Beat A. Imhof, Filippo Molica, Sandrine Morel, Brenda R. Kwak, Aurélie Hautefort, Pierre Fontana, Merlijn J. Meens, Yalin Emre, Eliana Scemes
Publikováno v:
Thrombosis Research, Vol. 183 (2019) pp. 56-62
Background Hemostasis is a tightly regulated physiological process to rapidly induce hemostatic plugs at sites of vascular injury. Inappropriate activation of this process may lead to thrombosis, i.e. pathological blood clot formation in uninjured ve
Autor:
Ian M. Adcock, Frédéric Perros, Aurélie Hautefort, V. Elyasigomari, John Wort, Marc Humbert, Sharon Mumby, Christopher K.M. Hui
Publikováno v:
D106. PICK YOUR POISON: GENES, DRUGS, AND PAH.