Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Ateeq R. Chaudhry"'
Autor:
Ateeq R. Chaudhry, Chelsea Thompson, James Denvir, Donald A. Primerano, Jun Fan, Travis B. Salisbury, Sean A. Piwarski
Publikováno v:
Biochem Pharmacol
The aryl hydrocarbon receptor (AHR) is a ligand-activated transcription factor. Triple negative breast cancer (TNBC) is the most aggressive breast cancer subtype. TNBC expresses AHR and AHR ligands have anti-cancer activity in TNBC. The aggressivenes
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f5858ecfba5bb8d8ee732045e74e7957
https://europepmc.org/articles/PMC7418053/
https://europepmc.org/articles/PMC7418053/
Autor:
Subha Arthur, Jun Fan, Travis B. Salisbury, Ateeq R. Chaudhry, James Denvir, Justin K. Tomblin, Donald A. Primerano
Publikováno v:
Biochemical Pharmacology. 106:94-103
The aryl hydrocarbon receptor (AHR) is a ligand-activated transcription factor that is regulated by environmental toxicants that function as AHR agonists such as 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD). L-Type Amino Acid Transporter 1 (LAT1) is a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a2cbdc910cb15e71d06915df1b564fbd
https://doi.org/10.1016/j.bcp.2016.02.020
https://doi.org/10.1016/j.bcp.2016.02.020
Autor:
Carla Cook, Nalini Santanam, Gary Z. Morris, Travis B. Salisbury, Ateeq R. Chaudhry, Justin K. Tomblin
Publikováno v:
ISRN Endocrinology
Obesity increases human cancer risk and the risk for cancer recurrence. Adipocytes secrete paracrine factors termed adipokines that stimulate signaling in cancer cells that induce proliferation. The aryl hydrocarbon receptor (AHR) is a ligand-activat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::87e8908e93b8861c701d8642947efbae
https://doi.org/10.1155/2013/104850
https://doi.org/10.1155/2013/104850
Publikováno v:
The FASEB Journal. 26
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::28bb7755d5a7678808ee6924600ba39f
https://doi.org/10.1096/fasebj.26.1_supplement.lb569
https://doi.org/10.1096/fasebj.26.1_supplement.lb569