Zobrazeno 1 - 10
of 38
pro vyhledávání: '"Asma, Quessar"'
Publikováno v:
Asian Pacific Journal of Cancer Biology, Vol 6, Iss 1, Pp 5-13 (2021)
Acute myeloïd leukemia (AML) is the most frequent form of acute leukemia among adults and the most aggressive type of leukemia, which is associated with the lowest survival rate. Patients with AML are treated with intensive chemotherapy and many fac
Externí odkaz:
https://doaj.org/article/420d75f2d9af44afb60191008a025c4c
Autor:
Kaltoum, Ait Boujmia Oum, Sellama, Nadifi, Hind, Dehbi, Yaya, Kassogue, Mouna, Lamchahab, Asma, Quessar
Publikováno v:
In Current Research in Translational Medicine January 2020 68(1):29-35
Autor:
Oum Kaltoum Ait Boujmia, Sellama Nadifi, Hind Dehbi, Yaya Kassogue, Mouna Lamchahab, Asma Quessar
Publikováno v:
Middle East Journal of Cancer, Vol 11, Iss 2, Pp 127-139 (2020)
Background: Acute myeloid leukemia (AML) is a complex disease that is linked to genetic and environmental factors. The gluthatione S-transferase (GST) is a family of enzymes that play a crucial role in the detoxification of carcinogens. These compoun
Externí odkaz:
https://doaj.org/article/5f94afa51b42499bb65ac85bc8b3bbee
Autor:
Faucher, Catherine, Adam, Catherine, Bancillon, Nelly, Bertrand, Elisabeth, Colledani, Fabienne, de Berranger, Eva, Denis, Virginie, Girard, Isabelle, Hamzy, Fati, Loukili, Noureddine, Mannone, Lionel, Mercier, Lara, Perrin, Agnes, Vasseur, Alyette, Asma, Quessar, Bompoint, Caroline, Yafour, Nabil, Yakoub-Agha, Ibrahim, Jost, Edgar
Publikováno v:
In Bulletin du Cancer January 2019 106(1) Supplement:S1-S9
Publikováno v:
Middle East Journal of Cancer, Vol 6, Iss 4, Pp 229-235 (2015)
Background: Despite the impressive results obtained with imatinib, inadequate response or resistance are observed in certain patients. It is known that imatinib is a substrate of a multidrug resistance gene (MDR1). Thus, interindividual genetic di
Externí odkaz:
https://doaj.org/article/02f105ff2e6e463ba354b297cf979b43
Publikováno v:
Middle East Journal of Cancer, Vol 4, Iss 1, Pp 1-5 (2013)
Background: According to numerous studies, FMS-like tyrosine kinase 3, internal tandem duplication, and the D835 mutation are associated with a poor prognostic clinical outcome in acute myeloid leukemia patients. Detection of the FMS-like tyrosine ki
Externí odkaz:
https://doaj.org/article/adb41716c4144752bf691d6f9b7760ca
Publikováno v:
Asian Pacific Journal of Cancer Prevention : APJCP
The human multidrug resistance MDR1 gene plays a crucial role in the absorption, transport, metabolism and elimination of harmful compounds. An impaired metabolism of these compounds related to genetic polymorphism may cause cancer such as acute myel
Publikováno v:
The Pan African Medical Journal, Vol 21, Iss 147 (2015)
La cytogénétique constitue un outil indispensable pour le diagnostic et le pronostic de la leucémie aigue myéloïde (LAM). La t(8 ;16)(p11 ;p13) est rare au cours de cette pathologie. Nous décrivons le cas d'une patiente de 22 ans, admise pour u
Externí odkaz:
https://doaj.org/article/17c20e346ee040a78a27b69bc27348a3
Publikováno v:
The Pan African Medical Journal, Vol 19, Iss 39 (2014)
Primary Plasma cell leukaemia (pPCL) is a rare plasma cell (PC) malignancy. The strict criteria for the diagnosis is an absolute PC number greater 2 X 109/L or a plasmocytosis accounting for = 20% of the differential white cell count that does not ar
Externí odkaz:
https://doaj.org/article/e80c89619cb241a4bfc5429d07730b6d
Autor:
Siham Cherkaoui, Mohamed Rachid, Asma Quessar, Fatima Bachir, Abdellah Madani, Bouchra Oukkache, Mouna Lamchahab, Nisrine Khoubila, Meryem Qachouh, Marielle Igala, Nezha Hda
Acute Myeloblastic Leukaemia (AML) is a malignant haemopathy characterized by infiltration and accumulation in the bone marrow by myeloblastic-type blast cells causing medullary insufficiency. At the global level, the search for improved AML treatmen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::b68d69ab6805531b4cb38b348a87cde1
https://doi.org/10.21203/rs.3.rs-409672/v1
https://doi.org/10.21203/rs.3.rs-409672/v1