Zobrazeno 1 - 10
of 21
pro vyhledávání: '"Ashley N. Oliveira"'
Publikováno v:
Skeletal Muscle, Vol 14, Iss 1, Pp 1-13 (2024)
Abstract Background Muscle atrophy is a common consequence of the loss of innervation and is accompanied by mitochondrial dysfunction. Mitophagy is the adaptive process through which damaged mitochondria are removed via the lysosomes, which are regul
Externí odkaz:
https://doaj.org/article/d319cf9a31ed44a9975624117faf7779
Publikováno v:
Autophagy Reports, Vol 1, Iss 1, Pp 75-78 (2022)
Since being discovered in the late 1990s, TP53/TRP53/p53 has been a focal point of research, lauded for its role as a tumor-suppressor protein. Considerable attention has also been devoted to understanding how TP53 regulates metabolic programming of
Externí odkaz:
https://doaj.org/article/b8e6c779a3d241bc823b59a8abbba236
Publikováno v:
Skeletal Muscle, Vol 12, Iss 1, Pp 1-18 (2022)
Abstract Background Aging decreases skeletal muscle mass and quality. Maintenance of healthy muscle is regulated by a balance between protein and organellar synthesis and their degradation. The autophagy-lysosome system is responsible for the selecti
Externí odkaz:
https://doaj.org/article/1dc864b717d44a95b58ec6c74ccc4d1f
Autor:
Ashley N. Oliveira, David A. Hood
Publikováno v:
Sports Medicine and Health Science, Vol 1, Iss 1, Pp 11-18 (2019)
Mitochondria are vital organelles that provide energy for muscle function. When these organelles become dysfunctional, they produce less energy as well as excessive levels of reactive oxygen species which can trigger muscle atrophy, weakness and loss
Externí odkaz:
https://doaj.org/article/723eade409fe4c5a8d6638c9f2ad240c
Autor:
Avigail T. Erlich, Liam D. Tryon, Matthew J. Crilly, Jonathan M. Memme, Zahra S. Mesbah Moosavi, Ashley N. Oliveira, Kaitlyn Beyfuss, David A. Hood
Publikováno v:
Integrative Medicine Research, Vol 5, Iss 3, Pp 187-197 (2016)
Skeletal muscle mitochondrial content and function are regulated by a number of specialized molecular pathways that remain to be fully defined. Although a number of proteins have been identified to be important for the maintenance of mitochondria in
Externí odkaz:
https://doaj.org/article/d17e2bd310dd4084832f10b56220007d
Autor:
Brandon J. Richards, Mikhaela Slavin, Ashley N. Oliveira, Victoria C. Sanfrancesco, David A. Hood
Publikováno v:
Seminars in Cell & Developmental Biology. 143:28-36
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::712be011b8d708f74863107418cabcae
https://doi.org/10.1016/j.semcdb.2022.01.002
https://doi.org/10.1016/j.semcdb.2022.01.002
Autor:
Anna Vainshtein, Mikhaela B. Slavin, Arthur J. Cheng, Jonathan M. Memme, Ashley N. Oliveira, Christopher G. R. Perry, Ali A. Abdul-Sater, Angelo N. Belcastro, Michael C. Riddell, Matthew Triolo, Tara L. Haas, Emilie Roudier, David A. Hood
Publikováno v:
Journal of Applied Physiology. 133:1381-1393
Exercise is one of the only non-pharmacologic remedies known to counteract genetic and chronic diseases by enhancing health and improving life span. Although the many benefits of regular physical activity have been recognized for some time, the intri
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b8c9c327f60786cf19707c0768e94cd7
https://doi.org/10.1152/japplphysiol.00475.2022
https://doi.org/10.1152/japplphysiol.00475.2022
Publikováno v:
American Journal of Physiology-Cell Physiology. 322:C913-C926
The adaptive plasticity of mitochondria within a skeletal muscle is regulated by signals converging on a myriad of regulatory networks that operate during conditions of increased (i.e., exercise) and decreased (inactivity, disuse) energy requirements
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e326a67f80c3b0605683321d365461ef
https://doi.org/10.1152/ajpcell.00065.2022
https://doi.org/10.1152/ajpcell.00065.2022
Autor:
David A. Hood, Ashley N. Oliveira
Publikováno v:
Sports Medicine and Health Science, Vol 1, Iss 1, Pp 11-18 (2019)
Mitochondria are vital organelles that provide energy for muscle function. When these organelles become dysfunctional, they produce less energy as well as excessive levels of reactive oxygen species which can trigger muscle atrophy, weakness and loss
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c759f50f26875f6dcd72345324d27266
https://pubmed.ncbi.nlm.nih.gov/35782464
https://pubmed.ncbi.nlm.nih.gov/35782464
Publikováno v:
Exercise and Sport Sciences Reviews. 49:67-76
Exercise stimulates the biogenesis of mitochondria in muscle. Some literature supports the use of pharmaceuticals to enhance mitochondria as a substitute for exercise. We provide evidence that exercise rejuvenates mitochondrial function, thereby augm
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e74532622575a3fc8a52993c1a336ae0
https://doi.org/10.1249/jes.0000000000000250
https://doi.org/10.1249/jes.0000000000000250