Corrigendum to "Beta cell specific cannabinoid 1 receptor deletion counteracts progression to hyperglycemia in non-obese diabetic mice" [Mol Metab 82 (2024 April) 101906].

Autor: Aseer KR; Laboratory of Clinical Investigation, National Institute on Aging, National Institutes of Health, Baltimore, MD 21224, USA; Department of Surgery, University of Maryland School of Medicine, Baltimore, MD 21201, USA., Mazucanti CH; Laboratory of Clinical Investigation, National Institute on Aging, National Institutes of Health, Baltimore, MD 21224, USA., O'Connell JF; Laboratory of Clinical Investigation, National Institute on Aging, National Institutes of Health, Baltimore, MD 21224, USA., González-Mariscal I; Inserm UMR1190 - Translational Research of Diabetes, Pôle recherche 3ème Ouest, 1, place de Verdun 59045 Lille Cedex, France., Verma A; Department of Surgery, University of Maryland School of Medicine, Baltimore, MD 21201, USA., Yao Q; Laboratory of Clinical Investigation, National Institute on Aging, National Institutes of Health, Baltimore, MD 21224, USA., Dunn C; Laboratory of Molecular Biology & Immunology, National Institute on Aging, National Institutes of Health, Baltimore, MD 21224, USA., Liu QR; Laboratory of Clinical Investigation, National Institute on Aging, National Institutes of Health, Baltimore, MD 21224, USA., Egan JM; Laboratory of Clinical Investigation, National Institute on Aging, National Institutes of Health, Baltimore, MD 21224, USA., Doyle ME; Laboratory of Clinical Investigation, National Institute on Aging, National Institutes of Health, Baltimore, MD 21224, USA. Electronic address: doyleme@nih.gov.

Publikováno v: Molecular metabolism [Mol Metab] 2024 Apr; Vol. 82, pp. 101917. Date of Electronic Publication: 2024 Mar 16.

Anti-Inflammatory and Pro-Autophagy Effects of the Cannabinoid Receptor CB2R: Possibility of Modulation in Type 1 Diabetes.

Autor: Liu QR; Laboratory of Clinical Investigation, National Institute on Aging, NIH, Baltimore, MD, United States., Aseer KR; Laboratory of Clinical Investigation, National Institute on Aging, NIH, Baltimore, MD, United States., Yao Q; Laboratory of Clinical Investigation, National Institute on Aging, NIH, Baltimore, MD, United States., Zhong X; Ben May Department for Cancer Research, The University of Chicago, Chicago, IL, United States., Ghosh P; Laboratory of Clinical Investigation, National Institute on Aging, NIH, Baltimore, MD, United States., O'Connell JF; Laboratory of Clinical Investigation, National Institute on Aging, NIH, Baltimore, MD, United States., Egan JM; Laboratory of Clinical Investigation, National Institute on Aging, NIH, Baltimore, MD, United States.

Publikováno v: Frontiers in pharmacology [Front Pharmacol] 2022 Jan 18; Vol. 12, pp. 809965. Date of Electronic Publication: 2022 Jan 18 (Print Publication: 2021).

Hepatocyte cannabinoid 1 receptor nullification alleviates toxin-induced liver damage via NF-κB signaling.

Autor: Kim Y; Laboratory of Clinical Investigation, National Institute on Aging, National Institutes of Health, Baltimore, MD, USA.; Department of Nutritional Sciences, Oklahoma State University, Stillwater, OK, USA., Gautam S; Laboratory of Clinical Investigation, National Institute on Aging, National Institutes of Health, Baltimore, MD, USA., Aseer KR; Laboratory of Clinical Investigation, National Institute on Aging, National Institutes of Health, Baltimore, MD, USA., Kim J; Laboratory of Molecular Biology and Immunology, National Institute on Aging, National Institutes of Health, Baltimore, MD, USA., Chandrasekaran P; Laboratory of Clinical Investigation, National Institute on Aging, National Institutes of Health, Baltimore, MD, USA., Mazucanti CH; Laboratory of Clinical Investigation, National Institute on Aging, National Institutes of Health, Baltimore, MD, USA., Ghosh P; Laboratory of Clinical Investigation, National Institute on Aging, National Institutes of Health, Baltimore, MD, USA., O'Connell JF; Laboratory of Clinical Investigation, National Institute on Aging, National Institutes of Health, Baltimore, MD, USA., Doyle ME; Laboratory of Clinical Investigation, National Institute on Aging, National Institutes of Health, Baltimore, MD, USA., Appleton A; Laboratory of Clinical Investigation, National Institute on Aging, National Institutes of Health, Baltimore, MD, USA., Lehrmann E; Laboratory of Genetics and Genomics, National Institute on Aging, National Institutes of Health, Baltimore, MD, USA., Liu QR; Laboratory of Clinical Investigation, National Institute on Aging, National Institutes of Health, Baltimore, MD, USA., Egan JM; Laboratory of Clinical Investigation, National Institute on Aging, National Institutes of Health, Baltimore, MD, USA. eganj@grc.nia.nih.gov.

Publikováno v: Cell death & disease [Cell Death Dis] 2020 Dec 09; Vol. 11 (12), pp. 1044. Date of Electronic Publication: 2020 Dec 09.

Loss of DJ-1 promotes browning of white adipose tissue in diet-induced obese mice.

Autor: Silvester AJ; Department of Biotechnology, Daegu University, Gyeongsan, Gyeongbuk 38453, Republic of Korea., Aseer KR; Department of Biotechnology, Daegu University, Gyeongsan, Gyeongbuk 38453, Republic of Korea., Jang HJ; School of Life Sciences, Ulsan National Institute of Science and Technology, Ulsan 44919, Republic of Korea., Ryu R; Department of Food Science and Nutrition, Center for Food and Nutritional Genomics Research, Kyungpook National University, Daegu 41566, Republic of Korea., Kwon EY; Department of Food Science and Nutrition, Center for Food and Nutritional Genomics Research, Kyungpook National University, Daegu 41566, Republic of Korea., Park JG; Advanced Bio Convergence Center, Pohang Technopark Foundation, Pohang 37668, Republic of Korea., Cho KH; Gyeongbuk Institute for Bioindustry, Andong 31984, Republic of Korea., Chaudhari HN; Department of Biotechnology, Daegu University, Gyeongsan, Gyeongbuk 38453, Republic of Korea., Choi MS; Department of Food Science and Nutrition, Center for Food and Nutritional Genomics Research, Kyungpook National University, Daegu 41566, Republic of Korea., Suh PG; School of Life Sciences, Ulsan National Institute of Science and Technology, Ulsan 44919, Republic of Korea., Yun JW; Department of Biotechnology, Daegu University, Gyeongsan, Gyeongbuk 38453, Republic of Korea. Electronic address: jwyun@daegu.ac.kr.

Publikováno v: The Journal of nutritional biochemistry [J Nutr Biochem] 2018 Nov; Vol. 61, pp. 56-67. Date of Electronic Publication: 2018 Jul 24.

Hydrothermal conversion of Magnolia liliiflora into nitrogen-doped carbon dots as an effective turn-off fluorescence sensing, multi-colour cell imaging and fluorescent ink.

Autor: Atchudan R; School of Chemical Engineering, Yeungnam University, Gyeongsan, 38541, Republic of Korea. Electronic address: atchudanr@yu.ac.kr., Edison TNJI; School of Chemical Engineering, Yeungnam University, Gyeongsan, 38541, Republic of Korea., Aseer KR; Department of Biotechnology, Daegu University, Kyungsan, Kyungbuk, 38453, Republic of Korea., Perumal S; Department of Applied Chemistry, Kyungpook National University, Daegu, 41566, Republic of Korea., Lee YR; School of Chemical Engineering, Yeungnam University, Gyeongsan, 38541, Republic of Korea. Electronic address: yrlee@yu.ac.kr.

Publikováno v: Colloids and surfaces. B, Biointerfaces [Colloids Surf B Biointerfaces] 2018 Sep 01; Vol. 169, pp. 321-328. Date of Electronic Publication: 2018 May 17.