Zobrazeno 1 - 10
of 40
pro vyhledávání: '"Arun Fotedar"'
Autor:
Arun Fotedar, Jasjit Singh Bhasin, Ashok Chakravarty, Avinash Kulkarni, Gaurav Bhalla, Fahmina Anwar, Shashidhar Rao
Publikováno v:
Journal of Family Medicine and Primary Care, Vol 7, Iss 1, Pp 77-80 (2018)
Introduction: Iron deficiency anemia represents 3rd largest disease burden, with an estimated 6.9 billion disability-adjusted life years.Iron-fortified cereals (IFIC) can contribute substantially in preventing iron deficiency anemia and maintaining a
Externí odkaz:
https://doaj.org/article/ad245a407cf94066a77a626d91e79f9b
Autor:
Michael Howell, Robert L. Margolis, Justin J Choi, Arun Fotedar, Howard Brickner, Céline DerMardirossian, Andreas Panopoulos, Daniel Miller, Rati Fotedar, Jean-Michel Saffin, Violaine D. Delorme-Walker
Publikováno v:
The Journal of Cell Biology
WISp39 associates with Hsp90, Coronin 1B, and Slingshot phosphatase to regulate Cofilin activation and Arp2/3 complex localization at the leading edge of migrating cells.
We previously identified Waf1 Cip1 stabilizing protein 39 (WISp39) as a bi
We previously identified Waf1 Cip1 stabilizing protein 39 (WISp39) as a bi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e236c8bb0c2ab3553f77346d5b0ac380
https://doi.org/10.1083/jcb.201410095
https://doi.org/10.1083/jcb.201410095
Publikováno v:
Molecular Immunology. 47:1154-1160
Endogenous mouse mammary tumor proviruses (MMTV; Mtv loci) deletes Vbeta6 expressing T cells in the thymus of Mtv-7(+) DBA/2 (H2(d)) mice through negative selection. We found that in Mtv-7(-) BALB/c (H2(d)) mice, Vbeta6 is a dominant V gene used in T
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::abf59131b7f91c01fa8bbe977ca0e68f
https://doi.org/10.1016/j.molimm.2009.11.006
https://doi.org/10.1016/j.molimm.2009.11.006
Autor:
Howard Brickner, Abdelhamid El Khissiin, Valerie Barbier, Brian J. Smith, Isabelle Salles-Passador, Thomas Jascur, Arun Fotedar, Rati Fotedar
Publikováno v:
Molecular Cell. 17:237-249
p21 WAF1/CIP1 , a cyclin-dependent kinase inhibitor and a critical regulator of cell cycle, is controlled transcriptionally by p53-dependent and -independent mechanisms and posttranslationally by the proteasome. We have identified WISp39, a tetratric
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::31fb7aad147426d9f238aacf6b35ef39
https://doi.org/10.1016/j.molcel.2004.11.049
https://doi.org/10.1016/j.molcel.2004.11.049
Autor:
Jérôme Boulaire, Alain Sarasin, Mourad Bendjennat, Rati Fotedar, Arun Fotedar, Valerie Barbier, Thomas Jascur, Howard Brickner
Publikováno v:
Cell. 114:599-610
p53-mediated increase in cyclin-dependent kinase inhibitor p21WAF1 protein is thought to be the major mediator of cell cycle arrest after DNA damage. Previously p21 protein levels have been reported to increase or to decrease after UV irradiation. We
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::32e5a428b15882293df4a188d59b4b95
https://doi.org/10.1016/j.cell.2003.08.001
https://doi.org/10.1016/j.cell.2003.08.001
Autor:
Isabelle, Salles-Passador, Anil, Munshi, Dominique, Cannella, Vincent, Pennaneach, Stephane, Koundrioukoff, Michel, Jaquinod, Eric, Forest, Vladimir, Podust, Arun, Fotedar, Rati, Fotedar, Michel, Jacquinod
Publikováno v:
Nucleic Acids Research. 31:5202-5211
Replication factor C (RF-C) complex binds to DNA primers and loads PCNA onto DNA, thereby increasing the processivity of DNA polymerases. We have previously identified a distinct region, domain B, in the large subunit of human RF-C (RF-Cp145) which b
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::973918f4a57f10dab2e24e881baae18e
https://doi.org/10.1093/nar/gkg692
https://doi.org/10.1093/nar/gkg692
Autor:
Arun Fotedar, Chandra L. Theesfeld, Martin Friedlander, Staffan Strömblad, Howard Brickner, David A. Cheresh, Edith Aguilar de Diaz
Publikováno v:
Journal of Biological Chemistry. 277:13371-13374
alpha(v)-Integrin antagonists block neovascularization in various species, whereas 20% of alpha(v)-integrin null mice are born with many normal looking blood vessels. Given that blockade of alpha(v)-integrins during angiogenesis induces p53 activity,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2eda59bfe4fab3e64cb54ee9fb61134f
https://doi.org/10.1074/jbc.c200044200
https://doi.org/10.1074/jbc.c200044200
Publikováno v:
Immunology today. 5(12)
Meiotic gene conversion in the major histocompatibility complex (MHC) appears to be involved in the generation of MHC polymorphism. Here the authors suggest that mitotic gene conversion of MHC may generate somatic variants which sensitize the immune
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4ca85aa65f41744c8b8b09996842b1ed
https://pubmed.ncbi.nlm.nih.gov/25290976
https://pubmed.ncbi.nlm.nih.gov/25290976
Autor:
Rati Fotedar, Dominique Cannella, Arun Fotedar, Patrick Fitzgerald, Denis Rousseau, Jérôme Boulaire
Publikováno v:
Oncogene. 18:4313-4325
The CDK inhibitor, p21(WAF1/Cip1) blocks cell cycle progression. In vitro, the N-terminus of p21 binds and inhibits CDK-cyclin kinase activity, whereas the C-terminus binds and inhibits PCNA (proliferating cell nuclear antigen) function. PCNA is esse
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ebbf2e0b38ae9a6eb18308452426cb9e
https://doi.org/10.1038/sj.onc.1202686
https://doi.org/10.1038/sj.onc.1202686
Autor:
Arun Fotedar, Rati Fotedar, Barbara H. Jung, Neshat Saadatmandi, Anil Munshi, Tristan Rousselle, Howard Brickner, Ludger Diederich, John C. Reed
Publikováno v:
Oncogene. 18:3652-3658
The cyclin kinase inhibitor p21WAF1/Cip1 is upregulated by the tumor suppressor p53. While p21 is central for the G-1 arrest mediated by p53, it is still unclear if p21 also functions as a downstream effector of p53 dependent apoptosis. Apoptosis ind
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3ffe7119fe969e1ab183b62dc6c512c6
https://doi.org/10.1038/sj.onc.1202693
https://doi.org/10.1038/sj.onc.1202693