Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Arshed Al-Obeidi"'
Autor:
Judith D. Goldberg, Xiaochun Li, Lori A. Leslie, Kevin A. David, Stefan K. Barta, Adam J. Olszewski, Sharyn Kurtz, Arshed Al-Obeidi, Austin I. Kim, Jordan Carter, Yun Choi, Julie E. Chang, Andrew M. Evens, Catherine Diefenbach, Adam Zayac, Jessica Caro, Suchitra Sundaram, Myung S. Kim, Andrew Matthews, Paolo Caimi, Sarah J. Nagle, Francisco J. Hernandez-Ilizaliturri
Publikováno v:
Blood
Background: Early reports suggested that cancer patients have a 1.7-fold increased risk of contracting SARS-CoV-2 and higher rates of severe events and mortality compared with general population. Patients with hematologic malignancies may have worse
Autor:
John N. Hutchinson, Alex Thompson, Jennifer D. Helble, Daniel E. Bauer, Meghan Bliss-Moreau, Ben A. Croker, Kimberly Stegmaier, Lorena Pantano, Kathleen Hanlon, Gabriela Alexe, Shu Wang, Arshed Al-Obeidi
Publikováno v:
Cancer Genet
Genetic modification of human leukemic cell lines using CRISPR-Cas9 has become a staple of gene-function studies. Single-cell cloning of modified cells is frequently used to facilitate studies of gene function. Inherent in this approach is an assumpt
Autor:
Mathilde Gavillet, Shu Wang, Ben A. Croker, David A. Williams, Michelle A. Kelliher, Meghan Bliss-Moreau, Sylvia Dietrich, Manolis Pasparakis, Alyce A. Chen, Akshay A. D’Cruz, Mary Speir, James E Vince, Arshed Al-Obeidi, Kate E. Lawlor, Maria Ericsson, Razq Hakem
Publikováno v:
Science signaling. 11(546)
Neutrophil extracellular trap (NET) formation can generate short-term, functional anucleate cytoplasts and trigger loss of cell viability. We demonstrated that the necroptotic cell death effector mixed lineage kinase domain–like (MLKL) translocated
Autor:
Arshed Al-Obeidi, Yesim Ozguler, Matthias C. Kugler, Johannes Nowatzky, Olivier Manches, Beatrix Ueberheide, H. Zhong, Gulen Hatemi, Berna Yurttas, A. Cavers
Publikováno v:
Annals of the Rheumatic Diseases. 79:22.2-23
Background:HLA-B51 is a definite risk factor for Behçet’s disease (BD). A coding variant of ERAP1, Hap10 – with low peptide-trimming activity – vastly potentiates this risk, but is mechanistically unclear1,2).Objectives:To test the hypothesis
Publikováno v:
Apoptosis and Beyond
An understanding of the mechanism of apoptosis has received a substantial amount of interest over the past decades. It is evident from many works that mitochondria play a pivotal role in keeping cell homeostasis in both physiological and pathological
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dfd882ad23893057c344bf178a12c29d
https://doi.org/10.1002/9781119432463.ch2
https://doi.org/10.1002/9781119432463.ch2