Zobrazeno 1 - 10
of 128
pro vyhledávání: '"Arnfinn Ilebekk"'
Publikováno v:
Blood Coagulation & Fibrinolysis. 23:714-722
Myocardial ischemia induces cardiac tissue plasminogen activator (tPA) release, declining by repeated periods of ischemia. However, the mechanisms and cellular sources are unknown. Sympathetic nerve stimulation (SS) and bradykinin (BK), an endogenous
Autor:
Haakon K. Grøgaard, Ketil Lunde, Harald Arnesen, Ingebjørg Seljeflot, Arnfinn Ilebekk, Svein Solheim, Kolbjørn Forfang, Svend Aakhus
Publikováno v:
Scandinavian Cardiovascular Journal. 44:267-272
To examine the influence of intracoronary autologous bone marrow cell transplantation after acute myocardial infarction on circulating growth factors and their relationship to left ventricular function.Circulating insulin-like growth factor-1 (IGF-1)
Publikováno v:
Thrombosis Research. 125:e106-e109
Background There is a substantial local release of tissue-type plasminogen activator (t-PA) in the splanchnic vascular bed, and this release is increased at high sympathetic tone. Coronary t-PA release is also significant, and this release increases
Autor:
Trude Aspelin, Morten Eriksen, Jan-Arne Björkman, Arnfinn Ilebekk, Margareta Nordlander, Knut Sevre, Torstein Lyberg
Publikováno v:
Journal of Cardiovascular Pharmacology and Therapeutics. 11:177-183
Neuropeptide Y is released together with norepinephrine from sympathetic nerve terminals during conditions of increased sympathetic activity. Neuropeptide Y is known to inhibit vagal activity, and accordingly, it might increase the risk for ventricul
Publikováno v:
Journal of Thrombosis and Haemostasis. 3:1947-1954
Summary. Background: Fibrinolysis in blood is mainly reflected by the activities of tissue plasminogen activator (tPA) and of plasminogen activator inhibitor-1 (PAI-1). The effect of myocardial ischemia on their activities in the coronary circulation
Publikováno v:
Scandinavian Cardiovascular Journal. 39:60-66
To examine if the decline in post-ischemic hyperemic flow after repeated brief periods of myocardial ischemia is accompanied by augmented cardiac release of the vasoconstrictors endothelin-1 (ET-1) and norepinephrine (NE).Mid-LAD (left anterior desce
Publikováno v:
Acta Physiologica Scandinavica. 175:261-269
During ischaemia and reperfusion the intracellular Na + concentration is elevated in the cardiomyocytes and the cells are depolarized, both favouring reverse mode Na,Ca-exchange loading of the cell with Ca 2+ . We examined whether cardiomyocytes from
Autor:
Torstein Lyberg, Alessandro Cataliotti, Trude Aspelin, Morten Eriksen, Cathrine R. Carlson, Arnfinn Ilebekk
Publikováno v:
Thrombosis and haemostasis. 112(5)
SummaryBradykinin (BK) receptor-2 (B2R) and β2-adrenergic receptor (β2AR) have been shown to form heterodimers in vitro. However, in vivo proofs of the functional effects of B2R-β2AR heterodimerisation are missing. Both BK and adrenergic stimulati
Autor:
Georgios K. Kanellopoulos, Aud Svindland, Ingeborg Løstegaard Goverud, Arnfinn Ilebekk, Knut Kvernebo
Publikováno v:
European Journal of Cardio-Thoracic Surgery. 16:135-143
Objective: Creation of non-transmural myocardial channels by lasers transmitted through endovascular fiberoptics is a novel therapeutic option in the management of patients with coronary artery disease. The acute effect of transventricular laser trea
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 277:H533-H542
Coronary microembolization has been reported to increase coronary blood flow (CBF) through adenosine release. Because adenosine may increase ischemic tolerance against infarction, we tested the hypothesis that myocardial microembolization, a common f