Zobrazeno 1 - 10
of 15
pro vyhledávání: '"Ariana Ern, Schmitz"'
Autor:
Gudrian Ricardo Lopes de Almeida, Jozimar Carlos Szczepanik, Alcir Luiz Dafre, Gabriela Cristina de Paula, Isabella A. Heinrich, Ariana Ern Schmitz, Mauricio P. Cunha, Barbara dos Santos, Rodrigo B. Leal, Ingrid Selhorst, Andreza Fabro de Bem
Publikováno v:
Molecular Neurobiology. 58:735-749
Methylglyoxal (MGO) is an endogenous toxin, mainly produced as a by-product of glycolysis that has been associated to aging, Alzheimer’s disease, and inflammation. Cell culture studies reported that MGO could impair the glyoxalase, thioredoxin, and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d6a4d530a564d45c5bea140927a4036c
https://doi.org/10.1007/s12035-020-02146-3
https://doi.org/10.1007/s12035-020-02146-3
Autor:
Ariana Ern Schmitz, Josiel Mileno Mack, Everson Araújo Nunes, Jozimar Carlos Szczepanik, Mauricio P. Cunha, Gudrian Ricardo Lopes de Almeida, Henver Simionato Brunetta, Luiz Felipe de Souza, Barbara dos Santos, Rui Daniel Prediger, Alcir Luiz Dafre
Publikováno v:
Neurochemical Research. 45:2868-2883
Recent evidence suggests that young rodents submitted to high fructose (FRU) diet develop metabolic, and cognitive dysfunctions. However, it remains unclear whether these detrimental effects of FRU intake can also be observed in middle-aged mice. Nin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::df74d3c2c698b14ffe6b1fff2913d302
https://doi.org/10.1007/s11064-020-03135-7
https://doi.org/10.1007/s11064-020-03135-7
Autor:
Roger Walz, Cláudia P. Figueiredo, Daniel Rial, Rui Daniel Prediger, Alcir Luiz Dafre, João Carlos Xikota, Ariana Ern Schmitz, Filipe S. Duarte
Publikováno v:
Neuroscience. 480:248-249
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::813d6fa3e91550541f8e76a7122b1b37
https://doi.org/10.1016/j.neuroscience.2021.10.028
https://doi.org/10.1016/j.neuroscience.2021.10.028
Autor:
Gudrian Ricardo Lopes, de Almeida, Jozimar Carlos, Szczepanik, Ingrid, Selhorst, Ariana Ern, Schmitz, Bárbara, Dos Santos, Maurício Peña, Cunha, Isabella Aparecida, Heinrich, Gabriela Cristina, de Paula, Andreza Fabro, De Bem, Rodrigo Bainy, Leal, Alcir Luiz, Dafre
Publikováno v:
Molecular neurobiology. 58(2)
Methylglyoxal (MGO) is an endogenous toxin, mainly produced as a by-product of glycolysis that has been associated to aging, Alzheimer's disease, and inflammation. Cell culture studies reported that MGO could impair the glyoxalase, thioredoxin, and g
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::ac2426e3851eba950b60571511383aa3
https://pubmed.ncbi.nlm.nih.gov/33011857
https://pubmed.ncbi.nlm.nih.gov/33011857
Autor:
Andreza Fabro de Bem, Luiz Felipe de Souza, Alcir Luiz Dafre, Ariana Ern Schmitz, Carla I. Tasca, Marcelo Farina, Karen Andrinéia de Oliveira, Cláudia Beatriz Nedel, Luana Caroline da Silva
Publikováno v:
Toxicology in Vitro. 42:273-280
Thiol homeostasis has a critical role in the maintenance of proper cellular functions and survival, being coordinated by the action of several reductive enzymes, including glutathione (GSH)/glutathione reductase (GR) and thioredoxin (Trx)/thioredoxin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d3137e81823ddc99fa646077c85be76f
https://doi.org/10.1016/j.tiv.2017.04.028
https://doi.org/10.1016/j.tiv.2017.04.028
Publikováno v:
Free Radical Biology and Medicine. 108:270-279
Methylglyoxal (MGO) is a major glycating agent that reacts with basic residues of proteins and promotes the formation of advanced glycation end products which are believed to play key roles in a number of pathologies, such as diabetes, Alzheimer’s
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ed4946e0e0fdefe2b1a243fce36b1984
https://doi.org/10.1016/j.freeradbiomed.2017.03.028
https://doi.org/10.1016/j.freeradbiomed.2017.03.028
Autor:
Ana Lúcia S. Rodrigues, Mauricio P. Cunha, Bettina Moritz, Alcir Luiz Dafre, Ariana Ern Schmitz
Publikováno v:
The Journal of nutritional biochemistry. 85
Stress-related disorders, such as depression and anxiety, present marked deficits in behavioral and cognitive functions related to reward. These are highly prevalent disabling conditions with high social and economic costs. Furthermore, a significant
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::13d8887bb1b674caeafb7a5ee6b9182d
https://pubmed.ncbi.nlm.nih.gov/32745879
https://pubmed.ncbi.nlm.nih.gov/32745879
Publikováno v:
Neurochemistry international. 132
Thioredoxin interacting protein (TXNIP) binds to thioredoxin thereby limiting its activity, but it also promotes internalization of glucose transporters, participates in inflammasome activation, and controls autophagy. Published data and this work de
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e4a5a9e9eae0cef0a676347e993a52cc
https://pubmed.ncbi.nlm.nih.gov/31678323
https://pubmed.ncbi.nlm.nih.gov/31678323
Hypothalamic Agrp neurons are critical regulators of food intake in adult mice. In addition to food intake, these neurons have been involved in other cognitive processes, such as the manifestation of stereotyped behaviors. Here, we evaluated the exte
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0826eb6771f0508c47460920f9f07c8e
https://europepmc.org/articles/PMC6400640/
https://europepmc.org/articles/PMC6400640/
Publikováno v:
Oxidative Medicine and Cellular Longevity, Vol 2019 (2019)
Oxidative Medicine and Cellular Longevity
Oxidative Medicine and Cellular Longevity
Background. Hyperosmotic stress is an important pathophysiologic condition in diabetes, severe trauma, dehydration, infection, and ischemia. Furthermore, brain neuronal cells face hyperosmotic stress in ageing and Alzheimer’s disease. Despite the e
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cf1eac6d020849e407e2430e0398405c
https://pubmed.ncbi.nlm.nih.gov/31049129
https://pubmed.ncbi.nlm.nih.gov/31049129