Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Aramati, Bindu Madhava Reddy"'
Publikováno v:
Cell Death and Disease, Vol 14, Iss 12, Pp 1-19 (2023)
Abstract The E3 ubiquitin ligase WWP1 (WW Domain-containing E3 Ubiquitin Protein Ligase 1) is a member of the HECT (Homologous to the E6-associated protein Carboxyl Terminus) E3 ligase family. It is conserved across several species and plays crucial
Externí odkaz:
https://doaj.org/article/5c7b1f454df043da80631b80b9faf407
Autor:
Meher Bolisetti Gayatri, Rama Krishna Kancha, Abhayananda Behera, Dorababu Patchva, Nagaraj Velugonda, Sadasivudu Gundeti, Aramati Bindu Madhava Reddy
Publikováno v:
Cell Death Discovery, Vol 9, Iss 1, Pp 1-12 (2023)
Abstract Imatinib resistance remains an unresolved problem in CML disease. Activation of JAK2/STAT3 pathway and increased expression of RUNX1 have become one reason for development of imatinib resistance in CML subjects. Metformin has gained attentio
Externí odkaz:
https://doaj.org/article/3d3b4f3397d24ba483df1239aecf2893
Autor:
Shaik, Jeelan Basha, Kandrakonda, Yelamanda Rao, Kallubai, Monika, Gajula, Navya Naidu, Dubey, Shreya, Aramati, Bindu Madhava Reddy, Subramanyam, Rajagopal, Amooru, Gangaiah Damu
Publikováno v:
In International Journal of Biological Macromolecules 15 December 2021 193 Part B:1409-1420
Akademický článek
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Autor:
Yelamanda Rao, Kandrakonda, Jeelan Basha, Shaik, Monika, Kallubai, Naidu Gajula, Navya, Sivakumar, Irla, Kumar, Sandeep, Vadde, Ramakrishna, Aramati, Bindu Madhava Reddy, Subramanyam, Rajagopal, Damu, Amooru Gangaiah
Publikováno v:
Journal of Biomolecular Structure and Dynamics; December 2023, Vol. 41 Issue: 20 p11148-11165, 18p
Autor:
Sarfraz Ahmad, Meghavarnam Anil Kumar, Varimadugu Aruna, Richa Bajpai, Obul Reddy Bandapalli, Abhayananda Behera, Lambodar Behera, L.V.K.S. Bhaskar, Gopamma Daka, Begum Dariya, Kalyani Dasari, Ravindra Donde, Debayan Ganguli, Bala Prabhakar Girish, Gayatri Gouda, Cheemachanahalli Muninanjappa Mohan Gowda, Nirmala Gollarahalli Sannappa Gowda, Manoj Kumar Gupta, D. Sai Harshitha, Mohammad Imran, Rajeswari Jinka, Deepika Divya Kadiri, Dowluru S.V.G.K. Kaladhar, Sirisha Kalam, Swarnalatha Kodidela, Seema Kumari, Dhatri Madduru, Rama Rao Malla, Jyothi Priya Mandala, Arundhati Mehta, Neha Merchant, Vidya Murugesan, Ganji Purnachandra Nagaraju, Srinivas Namuduri, Vijaya Nirmala Pangi, Pranathi Pappu, Sujatha Peela, Srinivas Podeti, Mahendar Porika, Senthilkumar Rajagopal, Samrat Rakshit, Jagadeeswara Rao Kakarla, Mamoon Ur. Rashid, Yashwant Kumar Ratre, Aramati Bindu Madhava Reddy, Nisha Sahu, Prasanna Kumar Santhekadur, Deepika Sarvepalli, Rajesh Sharma, Varsha Dilip Shiragannavar, Dhananjay Shukla, A. Sneha, Vivek Kumar Soni, Syamala Soumyakrishnan, Meenakshisundaram Sreepriya, Tantravahi Srinivasan, Gowru Srivani, L.S.S. Srivani Nagam, Ngalah Bidii Stephen, Prashanth Suravajhala, Chintapanti Swetha, Radhika Tippani, Vibhay Nath Tripathi, Shivanna Uma, Ramakrishna Vadde, Urvashi Vijay, Naveen Kumar Vishvakarma
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0f5c930251055994e9b40ea0d5a1bedf
https://doi.org/10.1016/b978-0-323-98806-3.00029-5
https://doi.org/10.1016/b978-0-323-98806-3.00029-5
Autor:
Aramati Bindu Madhava, Reddy, Kiranpreet, Kaur, Anil Kumar, Mandal, Shirly George, Panicker, Ravi, Thomas, Seyed Ehtesham, Hasnain, Dorairajan, Balasubramanian, Subhabrata, Chakrabarti
Publikováno v:
Molecular vision. 10
The human Cytochrome P450 gene CYP1B1 has been implicated in primary congenital glaucoma worldwide. The aim of this study was to understand the role of CYP1B1 mutations in causing primary congenital glaucoma in Indian populations.The study included 6
Autor:
Sreelatha, Komatireddy, Subhabrata, Chakrabarti, Anil Kumar, Mandal, Aramati Bindu Madhava, Reddy, Srirangan, Sampath, Shirly George, Panicker, Dorairajan, Balasubramanian
Publikováno v:
Molecular vision. 9
Axenfeld-Rieger anomaly (ARA) is a form of anterior segment dysgenesis of the eye, mainly caused by mutations in the FOXC1 gene. We had earlier reported a novel mutation in the wing region of FOXC1 in an autosomal dominant family. The present study w