Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Anwen S, Williams"'
Autor:
Fraser L. Collins, Michael D. Stone, Jane Turton, Laura R. McCabe, Eddie C. Y. Wang, Anwen S. Williams
Publikováno v:
BMC Musculoskeletal Disorders, Vol 20, Iss 1, Pp 1-9 (2019)
Abstract Background Oestrogen-deficiency induced by menopause is associated with reduced bone density and primary osteoporosis, resulting in an increased risk of fracture. While the exact etiology of menopause-induced primary osteoporotic bone loss i
Externí odkaz:
https://doaj.org/article/8dd198f2a3ae4bde95b064e87491e36b
Autor:
Fraser L. Collins, Jessica O. Williams, Anja C. Bloom, Michael D. Stone, Ernest Choy, Eddie C. Y. Wang, Anwen S. Williams
Publikováno v:
Journal of Immunology Research, Vol 2015 (2015)
Objectives. Genome wide association studies identified TNFSF member TNF-like protein 1A (TL1A, TNFSF15) as a potential modulator of ankylosing spondylitis (AS). TL1A is the only confirmed TNFSF ligand of death receptor 3 (DR3, TNFRSF25); however, its
Externí odkaz:
https://doaj.org/article/c00bfc7402a942d6b9aadd0101fa6b22
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 1725
In vivo mouse models of inflammatory arthritis are extensively used to investigate pathogenic mechanisms governing inflammation-driven joint damage. Two commonly utilized models include collagen-induced arthritis (CIA) and methylated bovine serum alb
Autor:
Fraser L, Collins, Jessica O, Williams, Anja C, Bloom, Michael D, Stone, Ernest, Choy, Eddie C Y, Wang, Anwen S, Williams
Publikováno v:
Journal of Immunology Research
Objectives. Genome wide association studies identified TNFSF member TNF-like protein 1A (TL1A, TNFSF15) as a potential modulator of ankylosing spondylitis (AS). TL1A is the only confirmed TNFSF ligand of death receptor 3 (DR3, TNFRSF25); however, its
Autor:
Eddie C Y, Wang, Zarabeth, Newton, Olivia A, Hayward, Stephen R, Clark, Fraser, Collins, William V, Perks, Ravinder K, Singh, Jason P, Twohig, Anwen S, Williams
Publikováno v:
Arthritis & Rheumatology (Hoboken, N.j.)
Objective To investigate the role of death receptor 3 (DR-3) and its ligand tumor necrosis factor–like molecule 1A (TL1A) in the early stages of inflammatory arthritis. Methods Antigen-induced arthritis (AIA) was generated in C57BL/6 mice deficient
Autor:
Anwen S. Williams, Richard A.G. Smith, Ian Dodd, Stuart M. Linton, Bryan D. Williams, B. Paul Morgan
Publikováno v:
Arthritis & Rheumatism. 43:2590-2597
Objective. Complement system activation is strongly implicated as a factor in the pathogenesis of chronic synovitis in human rheumatoid arthritis. The objective of this study was to explore the therapeutic potential and local retention of a novel mem
Autor:
Sarah N, Lauder, Emma, Jones, Kathryn, Smart, Anja, Bloom, Anwen S, Williams, James P, Hindley, Beatrice, Ondondo, Philip R, Taylor, Mathew, Clement, Ceri, Fielding, Andrew J, Godkin, Simon A, Jones, Awen M, Gallimore
Publikováno v:
European journal of immunology. 43(10)
Balancing the generation of immune responses capable of controlling virus replication with those causing immunopathology is critical for the survival of the host and resolution of influenza-induced inflammation. Based on the capacity of interleukin-6
Autor:
Deborah A, Fraser, Claire L, Harris, Anwen S, Williams, Masashi, Mizuno, Sean, Gallagher, Richard A G, Smith, B Paul, Morgan
Publikováno v:
The Journal of biological chemistry. 278(49)
Inappropriate activation of complement contributes to pathology in diverse inflammatory diseases. Soluble recombinant forms of the natural cell membrane regulators of complement are effective in animal models and some human diseases. However, their u
Autor:
Mari A. Nowell, Peter J. Richards, Ceri A. Fielding, Simona Ognjanovic, Nick Topley, Anwen S. Williams, Gillian Bryant‐Greenwood, Simon A. Jones
Publikováno v:
Arthritis & Rheumatism; Jul2006, Vol. 54 Issue 7, p2084-2095, 12p
Autor:
Peter J. Richards, Mari A. Nowell, Sankichi Horiuchi, Rachel M. McLoughlin, Ceri A. Fielding, Sandra Grau, Naoki Yamamoto, Michael Ehrmann, Stefan Rose‐John, Anwen S. Williams, Nicholas Topley, Simon A. Jones
Publikováno v:
Arthritis & Rheumatism; May2006, Vol. 54 Issue 5, p1662-1672, 11p