Zobrazeno 1 - 10
of 24
pro vyhledávání: '"Anuli C, Anyanwu"'
Autor:
Paul D Bozyk, J Kelley Bentley, Antonia P Popova, Anuli C Anyanwu, Marisa D Linn, Adam M Goldsmith, Gloria S Pryhuber, Bethany B Moore, Marc B Hershenson
Publikováno v:
PLoS ONE, Vol 10, Iss 6, p e0130369 (2015)
Externí odkaz:
https://doaj.org/article/17feeb61e58d4832a7fc5bf40a143ec8
Autor:
Michael Holinstat, Anuli C. Anyanwu, Raymond Zhao, J. Michelle Kahlenberg, Sharon Koonse, Hui Liao, Yogendra Kanthi, Benjamin E. Tourdot, Benjamin N. Jacobs, Scott H. Visovatti, Srilakshmi Yalavarthi, Alison L. Banka, Liguo Chi, Vinita Yadav, David J. Pinsky, Jason S. Knight
Publikováno v:
Journal of Clinical Investigation. 129:2872-2877
Deep vein thrombosis (DVT), caused by alterations in venous homeostasis is the third most common cause of cardiovascular mortality; however, key molecular determinants in venous thrombosis have not been fully elucidated. Several lines of evidence ind
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8bcbeee03e13e4f5227c3ef48e4d2090
https://doi.org/10.1172/jci124804
https://doi.org/10.1172/jci124804
Autor:
Paul D Bozyk, J Kelley Bentley, Antonia P Popova, Anuli C Anyanwu, Marisa D Linn, Adam M Goldsmith, Gloria S Pryhuber, Bethany B Moore, Marc B Hershenson
Publikováno v:
PLoS ONE, Vol 7, Iss 2, p e31336 (2012)
In bronchopulmonary dysplasia (BPD), alveolar septae are thickened with collagen and α-smooth muscle actin, transforming growth factor (TGF)-β-positive myofibroblasts. Periostin, a secreted extracellular matrix protein, is involved in TGF-β-mediat
Externí odkaz:
https://doaj.org/article/03574b837b9f402e8b9a4a0327bc01c0
Autor:
Vinita, Yadav, Liguo, Chi, Raymond, Zhao, Benjamin E, Tourdot, Srilakshmi, Yalavarthi, Benjamin N, Jacobs, Alison, Banka, Hui, Liao, Sharon, Koonse, Anuli C, Anyanwu, Scott H, Visovatti, Michael A, Holinstat, J Michelle, Kahlenberg, Jason S, Knight, David J, Pinsky, Yogendra, Kanthi
Publikováno v:
The Journal of clinical investigation. 129(7)
Deep vein thrombosis (DVT), caused by alterations in venous homeostasis, is the third most common cause of cardiovascular mortality, however, key molecular determinants in venous thrombosis have not been fully elucidated. Several lines of evidence in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::ae70b8aa66d7eb283a91c4e459d3190a
https://pubmed.ncbi.nlm.nih.gov/30990798
https://pubmed.ncbi.nlm.nih.gov/30990798
Autor:
Yogendra Kanthi, Tekashi Mimura, Raymond Zhao, Keigo Fukase, Matthew C. Hyman, Vinita Yadav, David J. Pinsky, Hui Liao, Saabir Kaskar, Karl C. Desch, Liguo Chi, Martin M. Gruca, Thomas W. Wakefield, Anuli C. Anyanwu, Hussein Sheikh-Aden
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 39
Objective— Leukocyte flux contributes to thrombus formation in deep veins under pathological conditions, but mechanisms that inhibit venous thrombosis are incompletely understood. Ectonucleotide di(tri)phosphohydrolase 1 ( ENTPD1 or Cd39 ), an ecto
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2f7f0c1a9c86f88f4ed0e9564840f031
https://doi.org/10.1161/atvbaha.119.312407
https://doi.org/10.1161/atvbaha.119.312407
Autor:
Takanori Hayasaki, Yogendra Kanthi, David J. Pinsky, Jessica J. Ray, Scott H. Visovatti, Matthew C. Hyman, Amy E. Baek, Hui Liao, Danica Petrovic-Djergovic, Sascha N. Goonewardena, Anuli C. Anyanwu, N.M. Walker, Ellen L. Horste, Nadia R. Sutton, Keigo Fukase, Linda Badri
Mechanical complications of myocardial infarction (MI) are often fatal. Little is known about endogenous factors that predispose to myocardial rupture after MI. Ectonucleoside triphosphate diphosphohydrolase (CD39) could be a critical mediator of pro
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::469f8f16bfb2ac0af3a67f09d0ea6626
https://europepmc.org/articles/PMC5213916/
https://europepmc.org/articles/PMC5213916/
Autor:
David J. Pinsky, J. Kelley Bentley, Antonia P. Popova, Adam M. Goldsmith, Marc B. Hershenson, Anuli C. Anyanwu, Omar Malas, Husam Alghanem
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology. 306:L749-L763
Bronchopulmonary dysplasia (BPD), a lung disease of prematurely born infants, is characterized in part by arrested development of pulmonary alveolae. We hypothesized that heme oxygenase (HO-1) and its byproduct carbon monoxide (CO), which are thought
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9dc98fece183685d8a024a3399569d0b
https://doi.org/10.1152/ajplung.00236.2013
https://doi.org/10.1152/ajplung.00236.2013
Autor:
Danica Petrovic-Djergovic, Anuli C. Anyanwu, Yogendra Kanthi, Patrick Robichaud, Scott H. Visovatti, David J. Pinsky, Rahul Rattan, Matthew C. Hyman, Jintao Wang, Sascha N. Goonewardena, Charles F. Burant
Publikováno v:
American journal of physiology. Heart and circulatory physiology. 311(1)
Despite the fact that nucleotides and adenosine help regulate vascular tone through purinergic signaling pathways, little is known regarding their contributions to the pathobiology of pulmonary arterial hypertension, a condition characterized by elev
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ad61ab9597363d639dda939c1f157b8d
https://pubmed.ncbi.nlm.nih.gov/27208163
https://pubmed.ncbi.nlm.nih.gov/27208163
Autor:
Martin M. Gruca, Keigo Fukase, Anuli C. Anyanwu, David J. Pinsky, Hui Liao, Marc B. Hershenson, Kelley Bentley, Takeshi Mimura
Publikováno v:
C69. CELLULAR SIGNALING OF OXIDATIVE STRESS.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8cd99f7b59eeafb482dc6f056e4f049e
https://doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4956
https://doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4956
Autor:
Jing Lei, J. K. Bentley, Marisa J. Linn, Anuli C. Anyanwu, Antonia P. Popova, Marc B. Hershenson
Publikováno v:
A28. LUNG DEVELOPMENT: BENCH TO BEDSIDE.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2c468fda5c82619438568f39526152a2
https://doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a1213
https://doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a1213