Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Antoine Leboucher"'
Autor:
Antoine Leboucher, Tariq Ahmed, Emilie Caron, Anne Tailleux, Sylvie Raison, Aurélie Joly-Amado, Elodie Marciniak, Kevin Carvalho, Malika Hamdane, Kadiombo Bantubungi, Steve Lancel, Sabiha Eddarkaoui, Raphaelle Caillierez, Emmanuelle Vallez, Bart Staels, Didier Vieau, Detlef Balschun, Luc Buee, David Blum
Publikováno v:
Neurobiology of Disease, Vol 125, Iss , Pp 14-22 (2019)
Accumulation of hyper-phosphorylated and aggregated Tau proteins is a neuropathological hallmark of Alzheimer's Disease (AD) and Tauopathies. AD patient brains also exhibit insulin resistance. Whereas, under normal physiological conditions insulin si
Externí odkaz:
https://doaj.org/article/1e5dc71246064ca998b489ffffebbd64
Autor:
Antoine Leboucher, Didier F. Pisani, Laura Martinez-Gili, Julien Chilloux, Patricia Bermudez-Martin, Anke Van Dijck, Tariq Ganief, Boris Macek, Jérôme A.J. Becker, Julie Le Merrer, R. Frank Kooy, Ez-Zoubir Amri, Edouard W. Khandjian, Marc-Emmanuel Dumas, Laetitia Davidovic
Publikováno v:
Molecular Metabolism, Vol 21, Iss , Pp 22-35 (2019)
Objectives: The Fragile X Mental Retardation Protein (FMRP) is a widely expressed RNA-binding protein involved in translation regulation. Since the absence of FMRP leads to Fragile X Syndrome (FXS) and autism, FMRP has been extensively studied in bra
Externí odkaz:
https://doaj.org/article/bf35ddf6316a40d5be9efe3bf42b1cc8
Autor:
Antoine Leboucher, Patricia Bermudez-Martin, Xavier Mouska, Ez-Zoubir Amri, Didier F. Pisani, Laetitia Davidovic
Publikováno v:
Frontiers in Endocrinology, Vol 10 (2019)
Fragile X syndrome (FXS) is a neurodevelopmental disorder associated with intellectual disability, hyperactivity, and autism. FXS is due to the silencing of the X-linked FMR1 gene. Murine models of FXS, knock-out (KO) for the murine homolog Fmr1, hav
Externí odkaz:
https://doaj.org/article/df11223abb414054b5593b9a4548c9b3
Autor:
Mareike Schell, Chantal Chudoba, Antoine Leboucher, Eugenia Alfine, Tanina Flore, Katrin Ritter, Katharina Weiper, Andreas Wernitz, Janin Henkel, André Kleinridders
Publikováno v:
Nutrients, Vol 12, Iss 5, p 1518 (2020)
Overconsumption of high-fat and cholesterol-containing diets is detrimental for metabolism and mitochondrial function, causes inflammatory responses and impairs insulin action in peripheral tissues. Dietary fatty acids can enter the brain to mediate
Externí odkaz:
https://doaj.org/article/e6b96d0596f74683b775fcda5e3dc680
Beneficial effects of exercise in a transgenic mouse model of Alzheimer's disease-like Tau pathology
Autor:
Karim Belarbi, Sylvie Burnouf, Francisco-Jose Fernandez-Gomez, Cyril Laurent, Sophie Lestavel, Martin Figeac, Audrey Sultan, Laetitia Troquier, Antoine Leboucher, Raphaëlle Caillierez, Marie-Eve Grosjean, Dominique Demeyer, Hélène Obriot, Ingrid Brion, Bérangère Barbot, Marie-Christine Galas, Bart Staels, Sandrine Humez, Nicolas Sergeant, Susanna Schraen-Maschke, Anne Muhr-Tailleux, Malika Hamdane, Luc Buée, David Blum
Publikováno v:
Neurobiology of Disease, Vol 43, Iss 2, Pp 486-494 (2011)
Tau pathology is encountered in many neurodegenerative disorders known as tauopathies, including Alzheimer's disease. Physical activity is a lifestyle factor affecting processes crucial for memory and synaptic plasticity. Whether long-term voluntary
Externí odkaz:
https://doaj.org/article/474624de82d042e7851d6576a260bf6a
Publikováno v:
Annales des Mines - Réalités industrielles. :60-63
Autor:
Ruan Carlos Macêdo de, Moraes, Gabriely Cristina Alves, Lima, Camila Aparecida Erreiras Fernandes, Cardinali, Alisson Carvalho, Gonçalves, Guilherme Vannucchi, Portari, Elvira Maria, Guerra-Shinohara, Antoine, Leboucher, José, Donato, André, Kleinridders, Andréa da Silva, Torrão
Publikováno v:
Repositório Institucional da USP (Biblioteca Digital da Produção Intelectual)
Universidade de São Paulo (USP)
instacron:USP
Universidade de São Paulo (USP)
instacron:USP
The neurodegeneration of Alzheimer's disease (AD) affects not only brain structures associate with cognition early in the progression of the disease, but other areas such as the hypothalamus, a region involved in the control of metabolism and appetit
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cb350bb4ca5b71f54eff020d20b2c7de
Autor:
Xavier Mouska, Ez-Zoubir Amri, Patricia Bermudez-Martin, Laetitia Davidovic, Didier F. Pisani, Antoine Leboucher
Publikováno v:
Frontiers in Endocrinology
Frontiers in Endocrinology, Frontiers, 2019, 10, ⟨10.3389/fendo.2019.00678⟩
Frontiers in Endocrinology, Frontiers, 2019, 10, pp.678. ⟨10.3389/fendo.2019.00678⟩
Frontiers in Endocrinology, 2019, 10, pp.678. ⟨10.3389/fendo.2019.00678⟩
Frontiers in Endocrinology, Vol 10 (2019)
Frontiers in Endocrinology, Frontiers, 2019, 10, ⟨10.3389/fendo.2019.00678⟩
Frontiers in Endocrinology, Frontiers, 2019, 10, pp.678. ⟨10.3389/fendo.2019.00678⟩
Frontiers in Endocrinology, 2019, 10, pp.678. ⟨10.3389/fendo.2019.00678⟩
Frontiers in Endocrinology, Vol 10 (2019)
International audience; Fragile X syndrome (FXS) is a neurodevelopmental disorder associated with intellectual disability, hyperactivity, and autism. FXS is due to the silencing of the X-linked FMR1 gene. Murine models of FXS, knock-out (KO) for the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b342224864fff004891ba3f599c1c151
https://hal.archives-ouvertes.fr/hal-02323472
https://hal.archives-ouvertes.fr/hal-02323472
Autor:
Kadiombo Bantubungi, Luc Buée, Anne Tailleux, David Blum, Emmanuelle Vallez, Sylvie Raison, Elodie Marciniak, Didier Vieau, Tariq Ahmed, Raphaëlle Caillierez, Steve Lancel, Detlef Balschun, Antoine Leboucher, Aurelie Joly-Amado, Emilie Caron, Sabiha Eddarkaoui, Kevin Carvalho, Bart Staels, Malika Hamdane
Publikováno v:
Neurobiology of Disease
Neurobiology of Disease, 2019, 125, pp.14-22. ⟨10.1016/j.nbd.2019.01.008⟩
Neurobiology of Disease, Elsevier, 2019, 125, pp.14-22. ⟨10.1016/j.nbd.2019.01.008⟩
Neurobiology of Disease, Vol 125, Iss, Pp 14-22 (2019)
Neurobiology of Disease, Elsevier, 2019, 125, pp.14-22. 〈10.1016/j.nbd.2019.01.008〉
Neurobiology of Disease, 2019, 125, pp.14-22. ⟨10.1016/j.nbd.2019.01.008⟩
Neurobiology of Disease, Elsevier, 2019, 125, pp.14-22. ⟨10.1016/j.nbd.2019.01.008⟩
Neurobiology of Disease, Vol 125, Iss, Pp 14-22 (2019)
Neurobiology of Disease, Elsevier, 2019, 125, pp.14-22. 〈10.1016/j.nbd.2019.01.008〉
International audience; Accumulation of hyper-phosphorylated and aggregated Tau proteins is a neuropathological hallmark of Alzheimer's Disease (AD) and Tauopathies. AD patient brains also exhibit insulin resistance. Whereas, under normal physiologic
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::06908d9b26ee8bf45b6b42dced23ef06
https://www.hal.inserm.fr/inserm-02350072
https://www.hal.inserm.fr/inserm-02350072
Autor:
Dominique Demeyer, David Blum, Christophe Breton, Pascal Pigny, Raphaëlle Caillierez, Laetitia Troquier, Francisco-Jose Fernandez-Gomez, Malika Hamdane, Kadiombo Bantubungi, Luc Buée, Sabiha Eddarkaoui, Nadège Zommer, Anne Tailleux, Emmanuelle Vallez, Bart Staels, Antoine Leboucher, Sylvie Burnouf, Cyril Laurent, Valérie Buée-Scherrer
Publikováno v:
Diabetes. 62:1681-1688
The τ pathology found in Alzheimer disease (AD) is crucial in cognitive decline. Midlife development of obesity, a major risk factor of insulin resistance and type 2 diabetes, increases the risk of dementia and AD later in life. The impact of obesit