Zobrazeno 1 - 2
of 2
pro vyhledávání: '"Antoine De Zelicourt"'
Autor:
Antoine de Zélicourt, Abdallah Fayssoil, Mbarka Dakouane‐Giudicelli, Isley De Jesus, Ahmed Karoui, Faouzi Zarrouki, Florence Lefebvre, Arnaud Mansart, Jean‐Marie Launay, Jerome Piquereau, Mariana G Tarragó, Marcel Bonay, Anne Forand, Sophie Moog, France Piétri‐Rouxel, Elise Brisebard, Claudia C S Chini, Sonu Kashyap, Matthew J Fogarty, Gary C Sieck, Mathias Mericskay, Eduardo N Chini, Ana Maria Gomez, José‐Manuel Cancela, Sabine de la Porte
Publikováno v:
EMBO Molecular Medicine, Vol 14, Iss 5, Pp 1-23 (2022)
Abstract Duchenne muscular dystrophy (DMD) is characterized by progressive muscle degeneration. Two important deleterious features are a Ca2+ dysregulation linked to Ca2+ influxes associated with ryanodine receptor hyperactivation, and a muscular nic
Externí odkaz:
https://doaj.org/article/ff8aa5831114408f802cc62d7a45fce4
Autor:
José-Manuel Cancela, Oscar Bauer, Jean-Marc Edeline, Gabriel Benet, Antoine De Zelicourt, Catherine Sebrié, Cyrille Vaillend, Muriel Amar, Sabine De La Porte, Antony Galione, Philippe Fossier, Lora L Martucci, Anne Nosjean, Jacques Callebert, Rémi Chaussenot, Sylvie Granon, Jean-Marie Launay
Publikováno v:
FASEB Journal
FASEB Journal, 2019, 54, pp.fj.201800489R. ⟨10.1096/fj.201800489R⟩
FASEB Journal, Federation of American Society of Experimental Biology, 2019, 54, pp.fj.201800489R. ⟨10.1096/fj.201800489R⟩
FASEB Journal, Federation of American Society of Experimental Biology, 2019, 33 (5), pp.5823-5835. ⟨10.1096/fj.201800489R⟩
FASEB Journal, 2019, 54, pp.fj.201800489R. ⟨10.1096/fj.201800489R⟩
FASEB Journal, Federation of American Society of Experimental Biology, 2019, 54, pp.fj.201800489R. ⟨10.1096/fj.201800489R⟩
FASEB Journal, Federation of American Society of Experimental Biology, 2019, 33 (5), pp.5823-5835. ⟨10.1096/fj.201800489R⟩
International audience; Autism spectrum disorder (ASD) is characterized by early onset of behavioral and cognitive alterations. Low plasma levels of oxytocin (OT) have also been found in ASD patients; recently, a critical role for the enzyme CD38 in