Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Annika Forsingdal"'
Autor:
Anne Krogh Nøhr, Annika Forsingdal, Ida Moltke, Oliver D. Howes, Morana Vitezic, Anders Albrechtsen, Maria Dalby
Publikováno v:
Translational Psychiatry, Vol 12, Iss 1, Pp 1-8 (2022)
Abstract The genetic architecture of antidepressant response is poorly understood. Polygenic risk scores (PRS), exploration of placebo response and the use of sub-scales might provide insights. Here, we investigate the association between PRSs for re
Externí odkaz:
https://doaj.org/article/1ef5143e614e451f83ea81e64b2cbff3
Autor:
Anne Krogh Nøhr, Samuel Demharter, Morana Vitezic, Ida Moltke, Raimund Buller, Anders Albrechtsen, Annika Forsingdal, Morten Lindow, Troels Nielsen
Publikováno v:
Neuropsychopharmacology
Nøhr, A K, Lindow, M, Forsingdal, A, Demharter, S, Nielsen, T, Buller, R, Moltke, I, Vitezic, M & Albrechtsen, A 2021, ' A large-scale genome-wide gene expression analysis in peripheral blood identifies very few differentially expressed genes related to antidepressant treatment and response in patients with major depressive disorder ', Neuropsychopharmacology, vol. 46, no. 7, pp. 1324-1332 . https://doi.org/10.1038/s41386-021-01002-9
Nøhr, A K, Lindow, M, Forsingdal, A, Demharter, S, Nielsen, T, Buller, R, Moltke, I, Vitezic, M & Albrechtsen, A 2021, ' A large-scale genome-wide gene expression analysis in peripheral blood identifies very few differentially expressed genes related to antidepressant treatment and response in patients with major depressive disorder ', Neuropsychopharmacology, vol. 46, no. 7, pp. 1324-1332 . https://doi.org/10.1038/s41386-021-01002-9
A better understanding of the biological factors underlying antidepressant treatment in patients with major depressive disorder (MDD) is needed. We perform gene expression analyses and explore sources of variability in peripheral blood related to ant
Autor:
Trine Nygaard Jørgensen, Michael Didriksen, Line Olsen, Annika Forsingdal, Jacob Nielsen, Thomas Werge
Publikováno v:
Biological Psychiatry
Background Elevated striatal dopamine synthesis capacity has been implicated in the etiology and antipsychotic response in psychotic illness. The effects of antipsychotic medication on dopamine synthesis capacity are poorly understood, and no prospec
Autor:
Aaron, Gordon, Annika, Forsingdal, Ib Vestergaard, Klewe, Jacob, Nielsen, Michael, Didriksen, Thomas, Werge, Daniel H, Geschwind
Publikováno v:
Molecular psychiatry. 26(5)
Genetic risk for psychiatric illness is complex, so identification of shared molecular pathways where distinct forms of genetic risk might coincide is of substantial interest. A growing body of genetic and genomic studies suggest that such shared mol
Publikováno v:
European Neuropsychopharmacology. 29:S855
Background The 15q13.3 microdeletion syndrome is caused by a 1.5 MB hemizygous microdeletion at chromosome 15q13.3 affecting seven genes: FAN1, MTMR10, TRPM1, MIR211, KLF13, OTUD7A, and CHRNA7. The 15q13.3 microdeletion increases the risk of intellec
Autor:
Denise Haslinger, Zuzanna Misiewicz, Luca Pagliaroli, Giorgia Quadri, Jose Estrada, Jie Song, Lynn E. DeLisi, Suhas Ganesham, Joanna Martin, Monique van der Voet, Alex D. Shaw, Lynsey Hall, Shing Wan Choi, Antonio F. Pardiñas, Samuel J.R.A. Chawner, Siri Ranlund, Maximilian Friedrich, Claudia Pisanu, Maria Tropeano, Monica Aas, Laura M. Huckins, Erik K. Loken, Marcos L. Santoro, Kate Wolfe, Annika Forsingdal, Stefanie Malan-Müller, Martin Tesli, Freida K. Cormack, Gabriëlla A.M. Blokland
Publikováno v:
Psychiatric Genetics, 26, 1, pp. 1-47
Repositório Institucional da UNIFESP
Universidade Federal de São Paulo (UNIFESP)
instacron:UNIFESP
Psychiatric Genetics, 26, 1-47
Repositório Institucional da UNIFESP
Universidade Federal de São Paulo (UNIFESP)
instacron:UNIFESP
Psychiatric Genetics, 26, 1-47
NIH The XXII World Congress of Psychiatric Genetics, sponsored by the International Society of Psychiatric Genetics, took place in Copenhagen, Denmark, on 12-16 October 2014. A total of 883 participants gathered to discuss the latest findings in the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::060cdd5f7394034489d2dba9eba5b5f9
https://hdl.handle.net/2066/165788
https://hdl.handle.net/2066/165788
Publikováno v:
Forsingdal, A, Fejgin, K, Nielsen, V, Werge, T & Nielsen, J 2016, ' 15q13.3 homozygous knockout mouse model display epilepsy-, autism-and schizophrenia-related phenotypes ', Translational Psychiatry, vol. 6, no. 7, e860, pp. 1-9 . https://doi.org/10.1038/tp.2016.125
Translational Psychiatry
Translational Psychiatry
The 15q13.3 microdeletion syndrome is caused by a 1.5-MB hemizygous microdeletion located on 15q13.3 affecting seven genes: FAN1; MTMR10; TRPM1; miR-211; KLF13; OTUD7A; and CHRNA7. The 15q13.3 microdeletion increases the risk of intellectual disabili
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::95cd32cdca6106e8007067ef6bc4d7ce
https://curis.ku.dk/ws/files/179630224/tp2016125a.pdf
https://curis.ku.dk/ws/files/179630224/tp2016125a.pdf