Zobrazeno 1 - 10
of 26
pro vyhledávání: '"Annica Rönnbäck"'
Autor:
Sonia George, Annica Rönnbäck, Gunnar K. Gouras, Géraldine H. Petit, Fiona Grueninger, Bengt Winblad, Caroline Graff, Patrik Brundin
Publikováno v:
Acta Neuropathologica Communications, Vol 12, Iss 1, Pp 1-5 (2024)
Externí odkaz:
https://doaj.org/article/58b662a6554f4f979652818fc4b7bcad
Autor:
Håkan Thonberg, Huei-Hsin Chiang, Lena Lilius, Charlotte Forsell, Anna-Karin Lindström, Charlotte Johansson, Jenny Björkström, Steinunn Thordardottir, Kristel Sleegers, Christine Van Broeckhoven, Annica Rönnbäck, Caroline Graff
Publikováno v:
Acta Neuropathologica Communications, Vol 5, Iss 1, Pp 1-14 (2017)
Abstract Alzheimer disease (AD) is a progressive neurodegenerative disorder and the most common form of dementia. The majority of AD cases are sporadic, while up to 5% are families with an early onset AD (EOAD). Mutations in one of the three genes: a
Externí odkaz:
https://doaj.org/article/c59c108dc0284f3baaa06cab13de6805
Autor:
Sonia George, Annica Rönnbäck, Gunnar K Gouras, Géraldine H Petit, Fiona Grueninger, Bengt Winblad, Caroline Graff, Patrik Brundin
Publikováno v:
Acta Neuropathologica Communications, Vol 2, Iss 1, Pp 1-8 (2014)
Abstract Background The progressive development of Alzheimer’s disease (AD) pathology follows a spatiotemporal pattern in the human brain. In a transgenic (Tg) mouse model of AD expressing amyloid precursor protein (APP) with the arctic (E693G) mut
Externí odkaz:
https://doaj.org/article/6ffc29574ae24c519248f66dfe289338
Autor:
Lisa, Dolfe, Simone, Tambaro, Helene, Tigro, Marta, Del Campo, Jeroen J M, Hoozemans, Birgitta, Wiehager, Caroline, Graff, Bengt, Winblad, Maria, Ankarcrona, Margit, Kaldmäe, Charlotte E, Teunissen, Annica, Rönnbäck, Jan, Johansson, Jenny, Presto
Publikováno v:
Journal of Alzheimer's Disease Reports
Alzheimer’s disease (AD) is the most common form of dementia and there is no successful treatment available. Evidence suggests that fibril formation of the amyloid β-peptide (Aβ) is a major underlying cause of AD, and treatment strategies that re
Autor:
Caroline Graff, Prisca Gonze, Mansorah Mansory, Annica Rönnbäck, Bengt Winblad, Homira Behbahani, Pavel F. Pavlov, Nicolas Marlière
Publikováno v:
Journal of Neurochemistry. 136:497-502
Accumulation of amyloid β-peptide (Aβ) in the brain is an important event in the pathogenesis of Alzheimer disease. We have used a transgenic mouse model expressing human amyloid precursor protein (APP) with the Arctic mutation to investigate wheth
Autor:
Annica Rönnbäck, Charlotte E. Teunissen, Bengt Winblad, Birgitta Wiehager, Helene Tigro, Maria Ankarcrona, Jenny Presto, Marta Del Campo, Simone Tambaro, Margit Kaldmäe, Caroline Graff, Lisa Dolfe, Jan Johansson, Jeroen J.M. Hoozemans
Publikováno v:
Alzheimers & Dementia, 2(1), 27-39. Elsevier
Dolfe, L, Tambaro, S, Tigro, H, Del Campo, M, Hoozemans, J J M, Wiehager, B, Graff, C, Winblad, B, Ankarcrona, M, Kaldmäe, M, Teunissen, C E, Rönnbäck, A, Johansson, J & Presto, J 2018, ' The Bri2 and Bri3 BRICHOS Domains Interact Differently with Aβ42 and Alzheimer Amyloid Plaques ', Alzheimers & Dementia, vol. 2, no. 1, pp. 27-39 . https://doi.org/10.3233/ADR-170051
Dolfe, L, Tambaro, S, Tigro, H, Del Campo, M, Hoozemans, J J M, Wiehager, B, Graff, C, Winblad, B, Ankarcrona, M, Kaldmäe, M, Teunissen, C E, Rönnbäck, A, Johansson, J & Presto, J 2018, ' The Bri2 and Bri3 BRICHOS Domains Interact Differently with Aβ42 and Alzheimer Amyloid Plaques ', Alzheimers & Dementia, vol. 2, no. 1, pp. 27-39 . https://doi.org/10.3233/ADR-170051
Alzheimer's disease (AD) is the most common form of dementia and there is no successful treatment available. Evidence suggests that fibril formation of the amyloid β-peptide (Aβ) is a major underlying cause of AD, and treatment strategies that redu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fd3e0e2c427a72aff7ee76c864c21d14
https://research.vumc.nl/en/publications/28380d64-35dd-408f-b061-b51de1a951ff
https://research.vumc.nl/en/publications/28380d64-35dd-408f-b061-b51de1a951ff
Autor:
Swapnali Barde, Tomas G. M. Hökfelt, Tibor Harkany, Mathias Uhlén, Annica Rönnbäck, Miklós Palkovits, Stefan Schulz, Laura Glück, Nicholas Mitsios, Gabor G. Kovacs, János Szolcsányi, László Havas, Thue W. Schwartz, Jens F. Rehfeld, Kata Bölcskei, Jan Kehr, Takashi Yoshitake, Csaba Adori, Zsófia Maglóczky, Jan Mulder
Publikováno v:
Acta Neuropathologica. 129:541-563
Alzheimer's disease and other age-related neurodegenerative disorders are associated with deterioration of the noradrenergic locus coeruleus (LC), a probable trigger for mood and memory dysfunction. LC noradrenergic neurons exhibit particularly high
Autor:
Charlotte Johansson, Annica Rönnbäck, Anna-Karin Lindström, Huei-Hsin Chiang, Kristel Sleegers, Håkan Thonberg, Steinunn Thordardottir, Lena Lilius, Christine Van Broeckhoven, Caroline Graff, Charlotte Forsell, Jenny Björkström
Publikováno v:
Acta Neuropathologica Communications
Acta Neuropathologica Communications, Vol 5, Iss 1, Pp 1-14 (2017)
Acta neuropathologica communications
Acta Neuropathologica Communications, Vol 5, Iss 1, Pp 1-14 (2017)
Acta neuropathologica communications
Alzheimer disease (AD) is a progressive neurodegenerative disorder and the most common form of dementia. The majority of AD cases are sporadic, while up to 5% are families with an early onset AD (EOAD). Mutations in one of the three genes: amyloid be
Autor:
Shunwei Zhu, Annica Rönnbäck, Caroline Graff, Bengt Winblad, Mikio Aoki, Lena Lilius, Jan Näslund, Karin Dillner
Publikováno v:
Neurobiology of Aging. 32:280-292
The Arctic APP mutation (E693G) leads to dementia with clinical features similar to Alzheimer disease (AD), but little is known about the pathogenic mechanism of this mutation. To address this question, we have generated a transgenic mouse model, TgA
Publikováno v:
Neuroscience Letters. 476:79-83
Housing rodents in an enriched environment (EE) induces structural and functional plasticity in the adult brain, including increased dendritic sprouting and number of dendritic spines. However, the molecular mechanisms behind EE-induced brain plastic