Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Anna V Vaaben"'
Autor:
Sebastian Rask Hamm, Sunil Kumar Saini, Annemette Hald, Anna V. Vaaben, Natasja Wulff Pedersen, Moises Alberto Suarez-Zdunek, Zitta Barrella Harboe, Helle Bruunsgaard, Isik Somuncu Johansen, Carsten Schade Larsen, Claus Bistrup, Henrik Birn, Søren Schwartz Sørensen, Sine Reker Hadrup, Susanne Dam Nielsen
Publikováno v:
BMC Infectious Diseases, Vol 23, Iss 1, Pp 1-7 (2023)
Abstract Background Kidney transplant recipients receive maintenance immunosuppressive therapy to avoid allograft rejection resulting in increased risk of infections and infection-related morbidity and mortality. Approximately 98% of adults are infec
Externí odkaz:
https://doaj.org/article/83e12673459a4efbae5077b556f816c3
Autor:
Anna V Vaaben, Justine Levan, Catherine B T Nguyen, Perri C Callaway, Mary Prahl, Lakshmi Warrier, Felistas Nankya, Kenneth Musinguzi, Abel Kakuru, Mary K Muhindo, Grant Dorsey, Moses R Kamya, Margaret E Feeney
Publikováno v:
The Journal of infectious diseases, vol 226, iss 4
BackgroundCongenital cytomegalovirus (CMV) infection is the most common infectious cause of birth defects and neurological damage in newborns. Despite a well-established role for natural killer (NK) cells in control of CMV infection in older children
Autor:
Anna V Vaaben, Justine Levan, Catherine B T Nguyen, Perri C Callaway, Mary Prahl, Lakshmi Warrier, Felistas Nankya, Kenneth Musinguzi, Abel Kakuru, Mary K Muhindo, Grant Dorsey, Moses R. Kamya, Margaret E. Feeney
BackgroundCongenital cytomegalovirus (CMV) infection is the most common infectious cause of birth defects and neurological damage in newborns. Despite a well-established role for NK cells in control of CMV infection in older children and adults, it r
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f7fbd76590ee93c11fbbba45dc56e5a8
https://doi.org/10.1101/2022.04.04.487059
https://doi.org/10.1101/2022.04.04.487059
Autor:
Steven B. Mwakalinga, Christian W. Wang, Jens E.V. Petersen, John Lusingu, Sixbert I. Mkumbaye, Eric Lyimo, Alphaxard Manjurano, Thor G. Theander, Jacklin F. Mosha, Anna V. Vaaben, Thomas Lavstsen, Daniel T. R. Minja, Reginald A. Kavishe
Publikováno v:
Scientific Reports
Petersen, J E V, Mkumbaye, S I, Vaaben, A V, Manjurano, A, Lyimo, E, Kavishe, R A, Mwakalinga, S B, Mosha, J, Minja, D T R, Lusingu, J P A, Theander, T G, Lavstsen, T & Wang, C W 2016, ' Plasma Ang2 and ADAM17 levels are elevated during clinical malaria; Ang2 level correlates with severity and expression of EPCR-binding PfEMP1 ', Scientific Reports, vol. 6, 35950 . https://doi.org/10.1038/srep35950
Petersen, J E V, Mkumbaye, S I, Vaaben, A V, Manjurano, A, Lyimo, E, Kavishe, R A, Mwakalinga, S B, Mosha, J, Minja, D T R, Lusingu, J P A, Theander, T G, Lavstsen, T & Wang, C W 2016, ' Plasma Ang2 and ADAM17 levels are elevated during clinical malaria; Ang2 level correlates with severity and expression of EPCR-binding PfEMP1 ', Scientific Reports, vol. 6, 35950 . https://doi.org/10.1038/srep35950
The pathogenesis of Plasmodium falciparum malaria involves a complex interplay between parasite adhesion and inflammatory response that includes release of cytokines and activation of the endothelium with accompanying release of Angiopoitin 2 (Ang2)