Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Anna Smok-Pieniazek"'
Autor:
Peter Grešner, Maciej Stepnik, Magdalena Beata Król, Radoslaw Swiercz, Anna Smok-Pieniazek, Ewa Twardowska, Jolanta Gromadzińska, Wojciech Wasowicz
Publikováno v:
Scandinavian Journal of Work, Environment & Health, Vol 41, Iss 6, Pp 579-593 (2015)
OBJECTIVE: The study aimed to compare levels of selected biomarkers of oxidative stress and DNA damage and their correlation with occupational exposure to volatile organic compounds (VOC) among female nail technicians and a group of unexposed volunte
Externí odkaz:
https://doaj.org/article/6d31aa572d8a4411bc717c13e780de71
Autor:
Edyta Reszka, Ewa Jabłońska, Jolanta Gromadzinska, Katarzyna Socha, Wojciech Wasowicz, M. Nocun, Maciej Stępnik, Anna Smok-Pieniazek, Sara Raimondi, Maria H. Borawska, Edyta Wieczorek, Magdalena Beata Krol
Publikováno v:
European Journal of Nutrition
Purpose Selenium, both essential and toxic element, is considered to protect against cancer, though human supplementation trials have generated many inconsistent data. Genetic background may partially explain a great variability of the studies relate
Autor:
Wojciech Wasowicz, Jolanta Gromadzinska, Magdalena Beata Krol, Anna Smok-Pieniazek, Radoslaw Swiercz, Peter Gresner, Ewa Twardowska, Maciej Stępnik
Publikováno v:
Scandinavian Journal of Work, Environment & Health, Vol 41, Iss 6, Pp 579-593 (2015)
Objective The study aimed to compare levels of selected biomarkers of oxidative stress and DNA damage and their correlation with occupational exposure to volatile organic compounds (VOC) among female nail technicians and a group of unexposed voluntee
Publikováno v:
Clinical Cancer Research. 16:B16-B16
Arsenic trioxide (ATO) is a well-known carcinogen but also an effective cancer therapeutic drug for acute promyelocytic leukemia. Despite intensive research efforts, the molecular mechanisms underlying antileukemic effect of ATO as well as resistance
Publikováno v:
Clinical Cancer Research. 16:A28-A28
Arsenic trioxide (ATO) induces growth inhibition and apoptosis in a wide variety of cancer cell lines. Some reports indicate that this effect can be enhanced by inhibiting the activity of transcription factor NF-κB. The aim of our study was to evalu