Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Anna, Sirico"'
Autor:
Laura Brandolini, Michele d’Angelo, Rubina Novelli, Vanessa Castelli, Cristina Giorgio, Anna Sirico, Pasquale Cocchiaro, Francesco D’Egidio, Elisabetta Benedetti, Claudia Cristiano, Antonella Bugatti, Anna Ruocco, Pier Giorgio Amendola, Carmine Talarico, Candida Manelfi, Daniela Iaconis, Andrea Beccari, Andreza U. Quadros, Thiago M. Cunha, Arnaldo Caruso, Roberto Russo, Annamaria Cimini, Andrea Aramini, Marcello Allegretti
Publikováno v:
Cell Death and Disease, Vol 13, Iss 5, Pp 1-15 (2022)
Abstract Chemotherapy-induced peripheral neuropathy (CIPN) and hypersensitivity reactions (HSRs) are among the most frequent and impairing side effects of the antineoplastic agent paclitaxel. Here, we demonstrated that paclitaxel can bind and activat
Externí odkaz:
https://doaj.org/article/482c74a958854186b399d4cb5817aa32
Autor:
Anna Ruocco, Anna Sirico, Rubina Novelli, Silvia Iannelli, Shane Vontelin Van Breda, Diego Kyburz, Paul Hasler, Andrea Aramini, Pier Giorgio Amendola
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 10 (2022)
Bone remodeling is a physiological, dynamic process that mainly depends on the functions of 2 cell types: osteoblasts and osteoclasts. Emerging evidence suggests that complement system is crucially involved in the regulation of functions of these cel
Externí odkaz:
https://doaj.org/article/f23cf6c0bb754aaebb1c01628cc98e8e
Autor:
Gianluca Bianchini, Cecilia Nigro, Anna Sirico, Rubina Novelli, Immacolata Prevenzano, Claudia Miele, Francesco Beguinot, Andrea Aramini
Publikováno v:
Biomedicine & Pharmacotherapy, Vol 139, Iss , Pp 111613- (2021)
G-protein coupled receptors 40 and 120 (GPR40 and GPR120) are increasingly emerging as potential therapeutic targets for the treatment of altered glucose homeostasis, and their agonists are under evaluation for their glucagon-like peptide-1 (GLP-1)-m
Externí odkaz:
https://doaj.org/article/ff02de5f55984e2eaf598240e9f57907
Autor:
Andrea Aramini, Silvia Bovolenta, Chiara Liberati, Lorena Za, Gianluca Bianchini, Mara Tomassetti, Samuele Lillini, Rubina Novelli, Anna Sirico
Publikováno v:
Journal of Medicinal Chemistry. 64:16820-16837
Transient receptor potential melastatin 8 (TRPM8) is crucially involved in pain modulation and perception, and TRPM8 antagonists have been proposed as potential therapeutic approaches for pain treatment. Previously, we developed two TRPM8 antagonists
Autor:
Gianluca, Bianchini, Mara, Tomassetti, Samuele, Lillini, Anna, Sirico, Silvia, Bovolenta, Lorena, Za, Chiara, Liberati, Rubina, Novelli, Andrea, Aramini
Publikováno v:
Journal of medicinal chemistry. 64(22)
Transient receptor potential melastatin 8 (TRPM8) is crucially involved in pain modulation and perception, and TRPM8 antagonists have been proposed as potential therapeutic approaches for pain treatment. Previously, we developed two TRPM8 antagonists
Autor:
Rubina Novelli, Gianluca Bianchini, Immacolata Prevenzano, Claudia Miele, Andrea Aramini, Francesco Beguinot, Anna Sirico, Cecilia Nigro
Publikováno v:
Biomedicine & Pharmacotherapy, Vol 139, Iss, Pp 111613-(2021)
G-protein coupled receptors 40 and 120 (GPR40 and GPR120) are increasingly emerging as potential therapeutic targets for the treatment of altered glucose homeostasis, and their agonists are under evaluation for their glucagon-like peptide-1 (GLP-1)-m
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b658fb44b89a243d65d03f715de7e828
http://hdl.handle.net/11588/867668
http://hdl.handle.net/11588/867668
Autor:
Caterina Missero, Satrajit Sinha, Christian Osterburg, Marco Ferniani, Anna Sirico, Claudia Russo, Sebastian Kehrloesser, Dario Antonini, Birgit Schäfer, Raffaele Ambrosio, Jörg Rinnenthal, Peter Güntert, Julia M. Würz, Volker Dötsch
Publikováno v:
Proceedings of the National Academy of Sciences of the United States of America, 115 (5)
Proceedings of the National Academy of Sciences of the United States of America
Proceedings of the National Academy of Sciences of the United States of America
The p63 gene encodes a master regulator of epidermal commitment, development, and differentiation. Heterozygous mutations in the C-terminal domain of the p63 gene can cause ankyloblepharon-ectodermal defects-cleft lip/palate (AEC) syndrome, a life-th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4e3d0c7af049e664ab6a9939e41007b0
https://www.repository.cam.ac.uk/handle/1810/279044
https://www.repository.cam.ac.uk/handle/1810/279044
Autor:
Edith Aberdam, Janice L. Brissette, Sharmila Fagoonee, Dario Antonini, Fiorella Altruda, Daniel Aberdam, Anna Sirico, Raffaele Ambrosio, Caterina Missero, Carmen Campanile
Publikováno v:
Nucleic acids research (Online) 43 (2015): 862–874. doi:10.1093/nar/gku1396
info:cnr-pdr/source/autori:Antonini, Dario; Sirico, Anna; Aberdam, Edith; Ambrosio, Raffaele; Campanile, Carmen; Fagoonee, Sharmila; Altruda, Fiorella; Aberdam, Daniel; Brissette, Janice L; Missero, Caterina/titolo:A composite enhancer regulates p63 gene expression in epidermal morphogenesis and in keratinocyte differentiation by multiple mechanisms./doi:10.1093%2Fnar%2Fgku1396/rivista:Nucleic acids research (Online)/anno:2015/pagina_da:862/pagina_a:874/intervallo_pagine:862–874/volume:43
Nucleic Acids Research
info:cnr-pdr/source/autori:Antonini, Dario; Sirico, Anna; Aberdam, Edith; Ambrosio, Raffaele; Campanile, Carmen; Fagoonee, Sharmila; Altruda, Fiorella; Aberdam, Daniel; Brissette, Janice L; Missero, Caterina/titolo:A composite enhancer regulates p63 gene expression in epidermal morphogenesis and in keratinocyte differentiation by multiple mechanisms./doi:10.1093%2Fnar%2Fgku1396/rivista:Nucleic acids research (Online)/anno:2015/pagina_da:862/pagina_a:874/intervallo_pagine:862–874/volume:43
Nucleic Acids Research
p63 is a crucial regulator of epidermal development, but its transcriptional control has remained elusive. Here, we report the identification of a long-range enhancer (p63LRE) that is composed of two evolutionary conserved modules (C38 and C40), acti
Autor:
Ada T. Agidi, Kathleen J. Green, Jennifer L. Koetsier, Anna Sirico, Jodi L. Johnson, Dario Antonini, Caterina Missero
Publikováno v:
The Journal of investigative dermatology
Epidermal structure is damaged by exposure to ultraviolet (UV) light but the molecular mechanisms governing structural repair are largely unknown. UVB (290-320 nm wavelengths) exposure prior to induction of differentiation reduced expression of diffe