Zobrazeno 1 - 10
of 15
pro vyhledávání: '"Anna, Kuret"'
Autor:
Nadja I. Bork, Anna Kuret, Melanie Cruz Santos, Cristina E. Molina, Beate Reiter, Hermann Reichenspurner, Andreas Friebe, Boris V. Skryabin, Timofey S. Rozhdestvensky, Michaela Kuhn, Robert Lukowski, Viacheslav O. Nikolaev
Publikováno v:
Redox Biology, Vol 48, Iss , Pp 102179- (2021)
3′,5′-cyclic guanosine monophosphate (cGMP) is a druggable second messenger regulating cell growth and survival in a plethora of cells and disease states, many of which are associated with hypoxia. For example, in myocardial infarction and heart
Externí odkaz:
https://doaj.org/article/06f06ef82ac1467b9416c06fe88290d6
Autor:
Natalie Längst, Julia Adler, Anna Kuret, Andreas Peter, Peter Ruth, Karsten Boldt, Robert Lukowski
Publikováno v:
Cells, Vol 11, Iss 8, p 1364 (2022)
Vascular smooth muscle cells (VSMCs) can switch from their contractile state to a synthetic phenotype resulting in high migratory and proliferative capacity and driving atherosclerotic lesion formation. The cysteine-rich LIM-only protein 4 (CRP4) rep
Externí odkaz:
https://doaj.org/article/c7e2b37d986b419982e697d6908c2bd0
Publikováno v:
British Journal of Pharmacology. 179:2344-2360
The 3',5'-cGMP pathway triggers cytoprotective responses and improves cardiomyocyte survival during myocardial ischaemia and reperfusion (I/R) injury. These beneficial effects were attributed to NO-sensitive GC induced cGMP production leading to acti
Autor:
Lukowski, Natalie Längst, Julia Adler, Anna Kuret, Andreas Peter, Peter Ruth, Karsten Boldt, Robert
Publikováno v:
Cells; Volume 11; Issue 8; Pages: 1364
Vascular smooth muscle cells (VSMCs) can switch from their contractile state to a synthetic phenotype resulting in high migratory and proliferative capacity and driving atherosclerotic lesion formation. The cysteine-rich LIM-only protein 4 (CRP4) rep
Publikováno v:
Journal of Cardiovascular Pharmacology. 75:494-507
The 3',5'-cyclic guanosine monophosphate (cGMP)-dependent protein kinase type I (cGKI aka PKGI) is a major cardiac effector acting downstream of nitric oxide (NO)-sensitive soluble guanylyl cyclase and natriuretic peptides (NPs), which signal through
Autor:
Andreas Friebe, Viacheslav O. Nikolaev, B Reiter, Boris V. Skryabin, Nadja I. Bork, Robert Lukowski, Melanie Cruz Santos, Timofey S. Rozhdestvensky, Cristina E. Molina, Anna Kuret, Hermann Reichenspurner, Michaela Kuhn
Publikováno v:
Redox Biology, Vol 48, Iss, Pp 102179-(2021)
Redox Biology
Redox Biology
3′,5′-cyclic guanosine monophosphate (cGMP) is a druggable second messenger regulating cell growth and survival in a plethora of cells and disease states, many of which are associated with hypoxia. For example, in myocardial infarction and heart
Publikováno v:
British journal of pharmacologyREFERENCES. 179(11)
The 3',5'-cGMP pathway triggers cytoprotective responses and improves cardiomyocyte survival during myocardial ischaemia and reperfusion (I/R) injury. These beneficial effects were attributed to NO-sensitive GC induced cGMP production leading to acti
Autor:
Clement Kabagema-Bilan, Nadine Frank, Katharina Wild, Anne E. Bausch, Roland Malli, Peter Ruth, Helmut Bischof, Robert Lukowski, Anna Kuret, Rebekka Ehinger, Lucas Matt
Publikováno v:
The FASEB Journal. 35
The neuronal Na+ -activated K+ channel Slack (aka Slo2.2, KNa 1.1, or Kcnt1) has been implicated in setting and maintaining the resting membrane potential and defining excitability and firing patterns, as well as in the generation of the slow afterhy
Autor:
Rebekka, Ehinger, Anna, Kuret, Lucas, Matt, Nadine, Frank, Katharina, Wild, Clement, Kabagema-Bilan, Helmut, Bischof, Roland, Malli, Peter, Ruth, Anne E, Bausch, Robert, Lukowski
Publikováno v:
FASEB journal : official publication of the Federation of American Societies for Experimental BiologyREFERENCES. 35(5)
The neuronal Na
Autor:
Peter Ruth, Rebekka Ehinger, Xiaobo Zhou, Roland Malli, A Luczak, Helmut Bischof, Anna Kuret, Robert Lukowski, Anne E. Bausch
Publikováno v:
European Heart Journal. 41
Background Sodium-activated potassium channels (KNa) Slack (sequence like a calcium-activated potassium channel, aka Slo 2.2, KNa1.1 or KCNT1) are widely expressed throughout neuronal tissue, whereas their presence and function in the cardiovascular