Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Anita Olofsson"'
Autor:
Bengt Nilsson, Anita Olofsson, Helene Sjögren, Nihal Chebbo, Fredrik Persson, Göran Stenman, Pierre Åman
Publikováno v:
Cancer Letters. 260:37-47
The cytogenetic hallmark of well-differentiated liposarcoma (WDLS) is a giant marker chromosomes containing amplified genes from chromosome 12q13-q15. Here, we have employed SKY and high-resolution 244K oligonucleotide array CGH to characterize rearr
Autor:
Christina Kåbjörn-Gustafsson, Marita Olsson, Helena Willén, Anita Olofsson, Melker Göransson, Sofia Järnum, Pierre Åman, Carola Andersson, Katarina Engström, Elisabeth Warnhammar
Publikováno v:
The American Journal of Pathology. 168:1642-1653
Myxoid/round cell liposarcoma (MLS/RCLS) is the most common subtype of liposarcoma. Most MLS/RCLS carry a t(12;16) translocation, resulting in a FUS-DDIT3 fusion gene. We investigated the role of the FUS-DDIT3 fusion in the development of MLS/RCLS in
Autor:
Anita Olofsson, Erik Elias, Pierre Åman, Melker Göransson, Carola Andersson, Anders Ståhlberg
Publikováno v:
International Journal of Cancer. 115:556-560
The myxoid/round cell liposarcoma oncogene FUS-DDIT3 is the result of a translocation derived gene fusion between the splicing factor FUS and DDIT3. In order to investigate the downstream targets of DDIT3, and the transforming effects of the FUS-DDIT
Autor:
Alondra Schweizer Burguete, Melker Göransson, Pierre Åman, Anita Olofsson, Sofia Thelin-Järnum
Publikováno v:
International Journal of Cancer. 97:446-450
CHOP in 12q13, also called GADD153 or DDIT3, encodes a transcription factor of the C/EBP type. As a result of t(12;16) translocations, CHOP is rearranged and fused to TLS in 16p11 in about 90% of myxoid liposarcomas/round cell liposarcomas (MLS/RCLS)
Publikováno v:
The American Journal of Pathology. 153:149-157
Mucosal mast cells (MCs) are normally found in the connective tissue stroma but are redistributed into the epithelium in conditions associated with immunoglobulin E responses, such as allergic inflammation and nematode infections, as well as in inter
Publikováno v:
BMC Cancer
BMC Cancer, Vol 10, Iss 1, p 249 (2010)
BMC Cancer, Vol 10, Iss 1, p 249 (2010)
Background The FUS-DDIT3 fusion oncogene encodes an abnormal transcription factor that has a causative role in the development of myxoid/round-cell liposarcomas (MLS/RCLS). We have previously identified FLT1 (VEGFR1) as a candidate downstream target
Autor:
Ola Nilsson, Henrik Semb, Göran Stenman, Pierre Åman, Anders Ståhlberg, Mattias K Andersson, Anita Olofsson, Yvonne Arvidsson
Publikováno v:
BMC Cell Biology, Vol 9, Iss 1, p 37 (2008)
BMC Cell Biology
BMC Cell Biology
Background FUS, EWS and TAF15 are structurally similar multifunctional proteins that were first discovered upon characterization of fusion oncogenes in human sarcomas and leukemias. The proteins belong to the FET (previously TET) family of RNA-bindin
Autor:
Lars-Gunnar Kindblom, Göran Stenman, Katarina Engström, Jeanne M. Meis-Kindblom, Pierre Åman, Helena Willén, Melker Göransson, Anita Olofsson
Publikováno v:
International journal of oncology. 25(5)
Myxoid/round cell liposarcomas (MLS/RCLS) are characterized by chromosome translocations that result in formation of FUS-CHOP or EWSR1-CHOP fusion oncogenes. More than 95% of the tumors carry one of these fusion genes. FUS-CHOP transforms 3T3 cells a
Autor:
Sofia, Thelin-Järnum, Melker, Göransson, Alondra Schweizer, Burguete, Anita, Olofsson, Pierre, Aman
Publikováno v:
International journal of cancer. 97(4)
CHOP in 12q13, also called GADD153 or DDIT3, encodes a transcription factor of the C/EBP type. As a result of t(12;16) translocations, CHOP is rearranged and fused to TLS in 16p11 in about 90% of myxoid liposarcomas/round cell liposarcomas (MLS/RCLS)
Autor:
Sadamu Nagai, S. Mor, Douglas J. Ormrod, Tor Lea, Lynn O’Neill-Davis, Richard P. Carlson, B.A. Spicer, D.C. Henderson, Makoto Owhashi, Daniel Czarny, Nancy Cruz, A.W. Thomson, I.J. Chopra, A.W. Wheeler, Ulf Pipkorn, S.C. Agarwal, H. Smith, Naohiro Watanabe, G.N. Beall, Alan J. Lewis, Anita Olofsson, S.M. Laycock, V. Mutt, I.A. Khan, L.-Å. Nilsson, K. Nordlind, G. Bhardwaj, H. Carlsten, Clara Ramirez, T. Gal, G. Sandberg, Morten Harboe, B. Rapoport, D.M. Moran, Mark A. Vanderwee, Laura M. Webster, Konrad Muller, N. Malla, C. Wattal, H. Vik, Sally R. Kruger, Å. Ljungdahl, A. Tarkowski, Y.H. Thong, Lennart Enerbäck, S. Elsayed, Julian Cornish, D.J. Radford, Beate M. Czarnetzki, Thomas E. Miller, Jo Douglass, S. Ben-Efraim, G.B. West, R. Lachman, Janet C. Ruby, Joseph Chang, Yukifumi Nawa, S. Gitter, Eberhard Zimmermann, Harald G. Wiker, N. Whittall, A. Ben-David, Manuel E. Patarroyo
Publikováno v:
International Archives of Allergy and Immunology. 81:I-V