Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Anita M. Klous"'
Autor:
Anita M. Klous, J. Wouter Jukema, Edward J. Bradley, Robbert J. de Winter, Claudia M. van Tiel, Frank Baas, Douwe Pons, Hans Pannekoek, Carlie J. M. de Vries, Marcel A.M. Beijk, M. Lourdes Sampietro, Saskia Z.H. Rittersma, Karel T. Koch, Peter I. Bonta
Publikováno v:
Circulation, 120(8), 669-676. Lippincott Williams and Wilkins Ltd.
Circulation, 120(8), 669-676. Lippincott Williams and Wilkins
Circulation, 120(8), 669-676. Lippincott Williams and Wilkins
Background— The cyclin-dependent kinase inhibitor p27 kip1 is a key regulator of smooth muscle cell and leukocyte proliferation in vascular disease, including in-stent restenosis. We therefore hypothesized that common genetic variations or single n
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::96e11be95a081a9d8055dc6fb7f590c2
https://doi.org/10.1161/CIRCULATIONAHA.108.842179
https://doi.org/10.1161/CIRCULATIONAHA.108.842179
Autor:
Hans G. Dauwerse, Nikos Tripodis, Anita M. Klous, Lucie Boerrigter, Wolter J. Mooi, Claudia A. L. Ruivenkamp, Václav Pačes, Nico van Zandwijk, Gert-Jan B. van Ommen, Gerrit A. Meijjer, Jan H.N. Lindeman, Gert Scholten, Tamás Csikós, Anastassis Perrakis, Peter Demant, Carlo Zanon, Čestmír Vlček, Alphons P. M. Stassen, Peter C. Groot, Tom van Wezel
Publikováno v:
Nature Genetics. 31:295-300
Only a small proportion of cancers result from familial cancer syndromes with Mendelian inheritance. Nonfamilial, 'sporadic' cancers, which represent most cancer cases, also have a significant hereditary component1,2, but the genes involved have low
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2fa7471fa50c6eaeb7f9fa1cd0eddc73
https://doi.org/10.1038/ng903
https://doi.org/10.1038/ng903
Autor:
Mario A J A Hermsen, Peter Demant, Jan Baak, Claudia A. L. Ruivenkamp, Cindy Postma, Gerrit A. Meijer, Anita M. Klous
Publikováno v:
Oncogene, 22(22), 3472-4. Nature Publishing Group
Ruivenkamp, C, Hermsen, M A J A, Postma, C, Klous, A, Baak, J, Meijer, G A & Demant, P 2003, ' LOH of PTPRJ occurs early in colorectal cancer and is associated with chromosomal loss of 18q12-21. ', Oncogene, vol. 22, no. 22, pp. 3472-4 . https://doi.org/10.1038/sj.onc.1206246
Ruivenkamp, C, Hermsen, M A J A, Postma, C, Klous, A, Baak, J, Meijer, G A & Demant, P 2003, ' LOH of PTPRJ occurs early in colorectal cancer and is associated with chromosomal loss of 18q12-21. ', Oncogene, vol. 22, no. 22, pp. 3472-4 . https://doi.org/10.1038/sj.onc.1206246
Recently, the gene PTPRJ (protein tyrosine phosphatase receptor type J) was identified as the candidate gene for the mouse colon cancer susceptibility locus Scc1. Its human homologue PTPRJ is frequently deleted in several cancer types, including colo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c5cc1629ffd80b986568dfeb37a41186
https://doi.org/10.1038/sj.onc.1206246
https://doi.org/10.1038/sj.onc.1206246
Autor:
Maarten L. Janmaat, Jasper Heerkens, Vivian de Waard, Anita M. Klous, Carlie J.M. de Vries, Alexander M. de Bruin
Publikováno v:
Blood, 115(7), 1453-1460. American Society of Hematology
In this study, the effect of human erythropoietin Δ (Epo) on smooth muscle cell (SMC)–rich lesions was evaluated. Mice, of which the left carotid artery was ligated, were treated with suberythropoietic as well as erythropoietic doses of Epo and bo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::23199a49b7462505e0fcbba8aa6830a7
https://pubmed.ncbi.nlm.nih.gov/20008786
https://pubmed.ncbi.nlm.nih.gov/20008786
Publikováno v:
Oncogene. 22(46)
Although several genes causing familial cancer syndromes have been identified, susceptibility to sporadic cancer remains unsolved. Animal experiments have demonstrated a large number of quantitative trait loci affecting cancer susceptibility. Previou
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::33acab7682db8d7ddb94b8e5af4e2a47
https://pubmed.ncbi.nlm.nih.gov/14562056
https://pubmed.ncbi.nlm.nih.gov/14562056