Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Anil Kachroo"'
Autor:
César Quiroz, Rafael Luján, Motokazu Uchigashima, Ana Patrícia Simoes, Talia N. Lerner, Janusz Borycz, Anil Kachroo, Paula M. Canas, Marco Orru, Michael A. Schwarzschild, Diane L. Rosin, Anatol C. Kreitzer, Rodrigo A. Cunha, Masahiko Watanabe, Sergi Ferré
Publikováno v:
The Scientific World Journal, Vol 9, Pp 1321-1344 (2009)
Basal ganglia processing results from a balanced activation of direct and indirect striatal efferent pathways, which are controlled by dopamine D1 and D2 receptors, respectively. Adenosine A2A receptors are considered novel antiparkinsonian targets,
Externí odkaz:
https://doaj.org/article/ad275f79176c452d8034ffe263f2ceba
Autor:
Anil Kachroo, Michael A. Schwarzschild
Publikováno v:
Brain Research. 1563:103-109
Robust epidemiological data link higher levels of the antioxidant urate to a reduced risk of developing Parkinson’s disease (PD) and to a slower rate of its progression. Allopurinol, an inhibitor of xanthine oxidoreductase (XOR), blocks the oxidati
Autor:
Michael A. Schwarzschild, Anil Kachroo
Publikováno v:
Annals of Neurology. 71:278-282
To investigate the putative interaction between chronic exposure to adenosine receptor antagonist caffeine and genetic influences on Parkinson's disease (PD), we determined whether deletion of the adenosine A(2A) receptor in knockout (KO) mice protec
Autor:
Danqing Xiao, Daniela Pellegrino, Anil Kachroo, Anna-Liisa Brownell, Yolanda D. Black, Michael A. Schwarzschild
Publikováno v:
Neuroscience Letters. 486:161-165
Pharmacologic or genetic blockade of metabotropic glutamate mGlu5 receptors (mGluR5) has been shown to attenuate parkinsonian motor deficits and protect nigrostriatal neurons from damage in the acute MPTP model of Parkinson's disease (PD), suggesting
Publikováno v:
Experimental Neurology. 223:657-661
Environmental exposures suspected of contributing to the pathophysiology of Parkinson's disease (PD) include potentially neurotoxic pesticides, which have been linked to an increased risk of PD. Conversely, possible protective factors such as the ade
Autor:
Anil Kachroo, Micaela Morelli, Nicoletta Schintu, Michael A. Schwarzschild, Jadwiga Wardas, Kui Xu, Anna R. Carta
Publikováno v:
Journal of Neurochemistry. 111:1478-1489
Adenosine A(2A) receptors antagonists produce neuroprotective effects in animal models of Parkinson's disease (PD). As neuroinflammation is involved in PD pathogenesis, both neuronal and glial A(2A) receptors might participate to neuroprotection. We
Autor:
Anil Kachroo, John A. Guarisco, Godwin C. Durunna, Edward C. Meek, Terrilyn A. Couch, Russell L. Carr, Jason R. Richardson, Janice E. Chambers
Publikováno v:
Toxicological Sciences. 67:311-321
Exposure to polychlorinated biphenyls (PCBs) can alter the metabolism of organophosphorus (OP) insecticides. Female rats were fed vanilla wafers containing either 4 mg/kg/day of Aroclor 1254 (PCB-treated) or safflower oil (oil-treated) for 50 days. R
Autor:
Bhat, Anil & Kachroo, Jyoti
The book “Economics of Production and Marketing of Citrus” provides an overview of the citrus production and marketing in the world in general and India in particular. The book is designed to throw some new light on the various aspects of citrus
The motor symptoms of Parkinson's disease (PD) are primarily due to the degeneration of the dopaminergic neurons in the nigrostriatal pathway. However, several other brain areas and neurotransmitters other than dopamine such as noradrenaline, 5-hydro
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::bcd8b617097bc05d548bc2b177c1e991
https://doi.org/10.1016/s0079-6123(10)83010-9
https://doi.org/10.1016/s0079-6123(10)83010-9
Autor:
Anna R, Carta, Anil, Kachroo, Nicoletta, Schintu, Kui, Xu, Michael A, Schwarzschild, Jadwiga, Wardas, Micaela, Morelli
Publikováno v:
Journal of neurochemistry. 111(6)
Adenosine A2A receptors antagonists produce neuroprotective effects in animal models of Parkinson’s disease (PD). Since neuroinflammation is involved in PD pathogenesis, both neuronal and glial A2A receptors might participate to neuroprotection. We