Zobrazeno 1 - 10
of 21
pro vyhledávání: '"Anika Götz"'
Autor:
Fakher Assaad, Martin Bercx, Florian Goth, Anika Götz, Johannes Hofmann, Emilie Huffman, Zihong Liu, Francesco Parisen Toldin, Jefferson Portela, Jonas Schwab
Publikováno v:
SciPost Physics Codebases.
The Algorithms for Lattice Fermions package provides a general code for the finite-temperature and projective auxiliary-field quantum Monte Carlo algorithm. The code is engineered to be able to simulate any model that can be written in terms of sums
Autor:
Fakher Assaad, Martin Bercx, Florian Goth, Anika Götz, Johannes Hofmann, Emilie Huffman, Zihong Liu, Francesco Parisen Toldin, Jefferson Portela, Jonas Schwab
Publikováno v:
SciPost Physics Codebases.
The Algorithms for Lattice Fermions package provides a general code for the finite-temperature and projective auxiliary-field quantum Monte Carlo algorithm. The code is engineered to be able to simulate any model that can be written in terms of sums
Autor:
Patrick Linsel-Nitschke, Anika Götz, Jeanette Erdmann, Ingrid Braenne, Peter Braund, Christian Hengstenberg, Klaus Stark, Marcus Fischer, Stefan Schreiber, Nour Eddine El Mokhtari, Arne Schaefer, Jürgen Schrezenmeir, Diana Rubin, Anke Hinney, Thomas Reinehr, Christian Roth, Jan Ortlepp, Peter Hanrath, Alistair S Hall, Massimo Mangino, Wolfgang Lieb, Claudia Lamina, Iris M Heid, Angela Doering, Christian Gieger, Annette Peters, Thomas Meitinger, H-Erich Wichmann, Inke R König, Andreas Ziegler, Florian Kronenberg, Nilesh J Samani, Heribert Schunkert, Wellcome Trust Case Control Consortium (WTCCC), Cardiogenics Consortium
Publikováno v:
PLoS ONE, Vol 3, Iss 8, p e2986 (2008)
Rare mutations of the low-density lipoprotein receptor gene (LDLR) cause familial hypercholesterolemia, which increases the risk for coronary artery disease (CAD). Less is known about the implications of common genetic variation in the LDLR gene rega
Externí odkaz:
https://doaj.org/article/5721ef7b49874c65bb1475a6eb9de291
Autor:
Patrick Linsel-Nitschke, Anika Götz, Jeanette Erdmann, Ingrid Braenne, Peter Braund, Christian Hengstenberg, Klaus Stark, Marcus Fischer, Stefan Schreiber, Nour Eddine El Mokhtari, Arne Schaefer, Jürgen Schrezenmeir, Diana Rubin, Anke Hinney, Thomas Reinehr, Christian Roth, Jan Ortlepp, Peter Hanrath, Alistair S. Hall, Massimo Mangino, Wolfgang Lieb, Claudia Lamina, Iris M. Heid, Angela Doering, Christian Gieger, Annette Peters, Thomas Meitinger, H.-Erich Wichmann, Inke R. König, Andreas Ziegler, Florian Kronenberg, Nilesh J. Samani, Heribert Schunkert
Publikováno v:
PLoS ONE, Vol 3, Iss 9 (2008)
Externí odkaz:
https://doaj.org/article/f3ca093aff2e471481e40480b42087f0
Autor:
Massimo Mangino, Jeanette Erdmann, Mark J. Caulfield, Andreas Ziegler, Peter S. Braund, Paul Burton, Anika Götz, Patrick Linsel-Nitschke, Klaus Stark, Cother Hajat, Maciej Tomaszewski, Nilesh J. Samani, Martin D. Tobin, Heribert Schunkert, Christian Hengstenberg
Publikováno v:
Journal of Molecular Medicine. 86:1233-1241
Through genome-wide association studies, we have recently identified seven novel loci that confer a substantial increase in risk for coronary artery disease (CAD). Elucidating the mechanisms by which these loci affect CAD risk could have important cl
Autor:
Stefan Blankenberg, Alistair S. Hall, Ralph McGinnis, Peter S. Braund, Alison H. Goodall, Richard J. Dixon, Nilesh J. Samani, Inke R. König, Daniel F Schwarz, Christian Hengstenberg, Andrew Keniry, John R. Thompson, Jeanette Erdmann, François Cambien, Nour Eddine El Mokhtari, Stefan Schreiber, Massimo Mangino, Panos Deloukas, Willem H. Ouwehand, Klaus Stark, Pierre Ducimetière, Marcus Fischer, Laurence Tiret, Andreas Ziegler, Christa Meisinger, Henrike Liptau, H.-Erich Wichmann, Anika Götz, Helen Pollard, David-Alexandre Trégouët, Mohammed J. R. Ghori, Patrick Linsel-Nitschke, Heribert Schunkert, Ludwig A. Hothorn
Publikováno v:
Circulation. 117:1675-1684
Background— Recently, genome-wide association studies identified variants on chromosome 9p21.3 as affecting the risk of coronary artery disease (CAD). We investigated the association of this locus with CAD in 7 case-control studies and undertook a
Autor:
Christian Hengstenberg, Wolfgang Lieb, Björn Mayer, Iris Borwitzky, Silke Kain, H.-Erich Wichmann, Andreas Ziegler, Inke R. König, Thomas Meitinger, Heribert Schunkert, Jeanette Erdmann, Marcus Fischer, Angela Döring, Anika Götz, Reinhold Kreutz, Patrick Linsel-Nitschke
Publikováno v:
Circulation 117, 185-191 (2008)
Background— Coronary artery disease (CAD) and myocardial infarction (MI) are caused in part by genetic factors. Recently, the MEF2A gene was linked to MI/CAD in a single pedigree with autosomal-dominant pattern of inheritance. In addition, genetic
Autor:
Peter W. Radke, Hans-Werner Hense, Henrike Liptau, Björn Mayer, Jeanette Erdmann, Andrea Bässler, Heribert Schunkert, Marcus Fischer, Lars C. Doehring, Anika Götz, Stephan R. Holmer, Zouhair Aherrahrou, Wolfgang Lieb, Christian Hengstenberg
Publikováno v:
Journal of Hypertension. 25:1731-1738
OBJECTIVES Coronary artery calcification (CAC) determined by electron beam computed tomography is a predictor of future cardiovascular events. This study investigates conditions affecting CAC severity in patients with coronary artery disease (CAD) un
Autor:
Eva-Maria Ehlers, Sandra Wrobel, Piotr M. Kaczmarek, Andrea Pomarino, Anika Götz, Jeanette Erdmann, Lars C. Doehring, Heribert Schunkert, Zouhair Aherrahrou, Henrike Liptau, Bjoern Mayer
Publikováno v:
Physiological Genomics. 28:203-212
In mice, dystrophic cardiovascular calcification (DCC) is controlled by a major locus on proximal mouse chromosome 7 named Dyscalc1. Here we present a strategy that combines in silico analysis, expression analysis, and extensive sequencing for ultraf
Autor:
Björn Mayer, Anika Götz, Jan Stritzke, Jochen Graf, Stephan R. Holmer, Marcus Fischer, Wolfgang Lieb, Christian Hengstenberg, Angela Döring, Heribert Schunkert, Hannelore Löwel, Jeanette Erdmann, Inke R. König
Publikováno v:
J. Mol. Med. 84, 88-96 (2006)
Angiotensin-converting enzyme (ACE) activity is considered to be of major importance for the conversion of angiotensin (Ang) I to Ang II. Recently, a second ACE, named ACE2, has been identified. Experimental data provide evidence that ACE2 might be i