Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Anica Scholz"'
Autor:
Benjamin Koch, Mahmoud Shehata, Christin Müller-Ruttloff, Shady A. Gouda, Nils Wetzstein, Sammy Patyna, Anica Scholz, Tobias Schmid, Ursula Dietrich, Christian Münch, John Ziebuhr, Helmut Geiger, Luis Martinez-Sobrido, Patrick C. Baer, Ahmed Mostafa, Stephan Pleschka
Publikováno v:
Frontiers in Cellular and Infection Microbiology, Vol 14 (2024)
IntroductionInfluenza A virus (IAV) infection can cause the often-lethal acute respiratory distress syndrome (ARDS) of the lung. Concomitantly, acute kidney injury (AKI) is frequently noticed during IAV infection, correlating with an increased mortal
Externí odkaz:
https://doaj.org/article/7ab24c6b07b54d538909b968359b9e77
Autor:
Andrea Hildebrandt, Mirko Brüggemann, Cornelia Rücklé, Susan Boerner, Jan B. Heidelberger, Anke Busch, Heike Hänel, Andrea Voigt, Martin M. Möckel, Stefanie Ebersberger, Anica Scholz, Annabelle Dold, Tobias Schmid, Ingo Ebersberger, Jean-Yves Roignant, Kathi Zarnack, Julian König, Petra Beli
Publikováno v:
Genome Biology, Vol 20, Iss 1, Pp 1-20 (2019)
Abstract Background Cells have evolved quality control mechanisms to ensure protein homeostasis by detecting and degrading aberrant mRNAs and proteins. A common source of aberrant mRNAs is premature polyadenylation, which can result in non-functional
Externí odkaz:
https://doaj.org/article/184af80dfd314f3694eb2622520459ab
Autor:
Christina A. Ortmann, Lena Dorsheimer, Khalil Abou-El-Ardat, Jennifer Hoffrichter, Birgit Assmus, Halvard Bonig, Anica Scholz, Heike Pfeifer, Hans Martin, Tobias Schmid, Bernhard Brüne, Sebastian Scheich, Björn Steffen, Julia Riemann, Stella Hermann, Alexandra Dukat, Gesine Bug, Christian H. Brandts, Sebastian Wagner, Hubert Serve, Michael A. Rieger
Publikováno v:
Cell Reports, Vol 27, Iss 7, Pp 2022-2028.e3 (2019)
Summary: Clonal hematopoiesis of indeterminate potential (CHIP) is caused by recurrent somatic mutations leading to clonal blood cell expansion. However, direct evidence of the fitness of CHIP-mutated human hematopoietic stem cells (HSCs) in blood re
Externí odkaz:
https://doaj.org/article/c59b55a4abea4891a6ce2eedd4dfb75b
Autor:
Yingying Han, Javier Mora, Arnaud Huard, Priscila da Silva, Svenja Wiechmann, Mateusz Putyrski, Christian Schuster, Eiman Elwakeel, Guangping Lang, Anica Scholz, Tatjana Scholz, Tobias Schmid, Natasja de Bruin, Pierre Billuart, Carlo Sala, Harald Burkhardt, Michael J. Parnham, Andreas Ernst, Bernhard Brüne, Andreas Weigert
Publikováno v:
Cell Reports, Vol 27, Iss 3, Pp 835-846.e5 (2019)
Summary: Interleukin-38 (IL-38) is a cytokine of the IL-1 family with a role in chronic inflammation. However, its main cellular targets and receptors remain obscure. IL-38 is highly expressed in the skin and downregulated in psoriasis patients. We r
Externí odkaz:
https://doaj.org/article/6b0ea48022dd45428022b5ac6a3fd6d3
Autor:
Anica Scholz, Florian Eggenhofer, Rick Gelhausen, Björn Grüning, Kathi Zarnack, Bernhard Brüne, Rolf Backofen, Tobias Schmid
Publikováno v:
PLoS ONE, Vol 14, Iss 9, p e0222459 (2019)
Ribosome profiling (ribo-seq) provides a means to analyze active translation by determining ribosome occupancy in a transcriptome-wide manner. The vast majority of ribosome protected fragments (RPFs) resides within the protein-coding sequence of mRNA
Externí odkaz:
https://doaj.org/article/49a2a1380fb540198a48dc14691e8913
Autor:
Sofia Winslow, Anica Scholz, Peter Rappl, Thilo F Brauß, Christina Mertens, Michaela Jung, Andreas Weigert, Bernhard Brüne, Tobias Schmid
Publikováno v:
PLoS ONE, Vol 14, Iss 1, p e0209694 (2019)
While aberrant cells are routinely recognized and removed by immune cells, tumors eventually escape innate immune responses. Infiltrating immune cells are even corrupted by the tumor to acquire a tumor-supporting phenotype. In line, tumor-associated
Externí odkaz:
https://doaj.org/article/c171fb79eb7740a9b8daa89a0fef07c7
Autor:
Anica Scholz, Nicola Böffinger, Bernhard Brüne, Ana Carolina Mota, Tobias Schmid, Peter Rappl
Translation is a highly regulated process, both at the global as well as on a transcript-specific level. Regulatory upstream open reading frames (uORFs) represent a mode to alter cap-dependent translation efficiency in a transcript-specific manner an
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::65cb9772fffbeaaf5752d6d5276167cf
https://publica.fraunhofer.de/handle/publica/267035
https://publica.fraunhofer.de/handle/publica/267035
Autor:
Stefan Simm, Michael M. Kunze, Sofia Winslow, Tobias Schmid, Mario Keller, Anica Scholz, Thilo F. Brauß, Sebastian Lampe, Juliana Heidler, Ilka Wittig, Bernhard Brüne
Publikováno v:
Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms. 1861:147-157
Translation is a tightly regulated process and is predominantly controlled at the level of its initiation. Translation initiation mostly occurs in a cap-dependent manner. Under stress conditions when cap-dependent translation is hampered, internal ri
Autor:
Kristoffer von Stedingk, Nathalie Dehne, Bernhard Brüne, Anica Scholz, Tobias Schmid, Sofia Winslow, Thilo F. Brauß, Andreas Weigert, Sebastian Lampe
Publikováno v:
Molecular Carcinogenesis. 56:2620-2629
The RNA-binding protein HuR promotes tumor growth by affecting proliferation, metastasis, apoptosis, and angiogenesis. Although immune cells, especially tumor-associated macrophages, are critical components of the tumor stroma, the influence of HuR i
Autor:
Heike Hänel, Stefanie Ebersberger, Cornelia Rücklé, Jan B. Heidelberger, Julian König, Jean-Yves Roignant, Ingo Ebersberger, Tobias Schmid, Anica Scholz, Mirko Brüggemann, Andrea Hildebrandt, Anke Busch, Andrea Voigt, Petra Beli, Kathi Zarnack, Susan Boerner, Martin Möckel, Annabelle Dold
Publikováno v:
Genome Biology
Genome biology, vol. 20, no. 1, pp. 216
Genome Biology, Vol 20, Iss 1, Pp 1-20 (2019)
Genome biology, vol. 20, no. 1, pp. 216
Genome Biology, Vol 20, Iss 1, Pp 1-20 (2019)
Background Cells have evolved quality control mechanisms to ensure protein homeostasis by detecting and degrading aberrant mRNAs and proteins. A common source of aberrant mRNAs is premature polyadenylation, which can result in non-functional protein