Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Angy J. Kallarackal"'
Benefits of Using the Invertebrate Model Organism C. Elegans in an Inquiry-Based Laboratory Activity
Autor:
Angy J. Kallarackal
Publikováno v:
Teaching of Psychology. 50:26-31
Background: The goals of laboratory experiences include developing knowledge base, research skills, and scientific communication abilities. Objective: The aim was to assess an inquiry-based laboratory activity using the model organism Caenorhabditis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c8062d65ad915a9cac6dacd44687aaf6
https://doi.org/10.1177/00986283211029649
https://doi.org/10.1177/00986283211029649
Autor:
Rui Wang, Frédéric J. Hoerndli, David M. Madsen, Colin Thacker, Penelope J. Brockie, Andres V. Maricq, Jerry E. Mellem, Angy J. Kallarackal
Publikováno v:
Neuron. 86:457-474
Summary Excitatory glutamatergic synaptic transmission is critically dependent on maintaining an optimal number of postsynaptic AMPA receptors (AMPARs) at each synapse of a given neuron. Here, we show that presynaptic activity, postsynaptic potential
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::10c13fab1f8cbcf7bd7d669c820cb6ad
https://doi.org/10.1016/j.neuron.2015.03.011
https://doi.org/10.1016/j.neuron.2015.03.011
Autor:
Scott M. Thompson, Kaitlin Gaylor, Angy J. Kallarackal, Aileen M. Bailey, Xiang Cai, Mark D. Kvarta, Richard L. Huganir, Sasha Goluskin, Hey Kyoung Lee
Publikováno v:
Nature neuroscience
The causes of major depression remain unknown. Antidepressants elevate concentrations of monoamines, particularly serotonin, but it remains uncertain which downstream events are critical to their therapeutic effects. We found that endogenous serotoni
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0a9b67451253e1b4508478de7de47ab4
https://doi.org/10.1038/nn.3355
https://doi.org/10.1038/nn.3355
Publikováno v:
Channels (Austin, Tex.). 9(5)
Excitatory glutamatergic synaptic transmission is critically dependent on maintaining an optimal number of postsynaptic AMPA receptors (AMPARs) at each synapse of a given neuron. Here, we show that presynaptic activity, postsynaptic potential, voltag
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d2f4f6ce10c8216e67e15431ad061230
https://pubmed.ncbi.nlm.nih.gov/25843407
https://pubmed.ncbi.nlm.nih.gov/25843407
Autor:
Mark D. Kvarta, Angy J. Kallarackal, Xiang Cai, Adam M. Van Dyke, Tara A. LeGates, Scott M. Thompson
Depression is a common cause of mortality and morbidity, but the biological bases of the deficits in emotional and cognitive processing remain incompletely understood. Current antidepressant therapies are effective in only some patients and act slowl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2f27b31e1505f0d02f1f1c6cc8b74b44
https://europepmc.org/articles/PMC4417609/
https://europepmc.org/articles/PMC4417609/
Autor:
Erin Cammarata, Mark D. Kvarta, Xiang Cai, Angy J. Kallarackal, Leelah Jaberi, Scott M. Thompson, Aileen M. Bailey
Publikováno v:
The Journal of neuroscience : the official journal of the Society for Neuroscience. 33(40)
Chronic stress promotes depression, but how it disrupts cognition and mood remains unknown. Chronic stress causes atrophy of pyramidal cell dendrites in the hippocampus and cortex in human and animal models, and a depressive-like behavioral state. We
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b6f4616a8b72cf249156918be9790a89
https://pubmed.ncbi.nlm.nih.gov/24089474
https://pubmed.ncbi.nlm.nih.gov/24089474
Publikováno v:
Behavioural brain research. 237
Neurofibromatosis 1 (NF1) is a common genetic disorder known to cause a variety of physiological symptoms such as the formation of both benign and malignant tumors, and is also known to cause visuospatial learning deficits. Mouse models of NF1 show i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4d81b89c6fba3d763156bf0d15724e0c
https://pubmed.ncbi.nlm.nih.gov/22983217
https://pubmed.ncbi.nlm.nih.gov/22983217